about
ATG16L1 phosphorylation is oppositely regulated by CSNK2/casein kinase 2 and PPP1/protein phosphatase 1 which determines the fate of cardiomyocytes during hypoxia/reoxygenation.Increasing gap junction coupling suppresses ibutilide-induced torsades de pointes.Suppression of ryanodine receptor function prolongs Ca2+ release refractoriness and promotes cardiac alternans in intact hearts.Antiarrhythmic peptide AAP10 prevents arrhythmias induced by protein kinase C activation in rabbit left ventricular wedges.Reduced Expression of Cardiac Ryanodine Receptor Protects against Stress-induced Ventricular Tachyarrhythmia, but Increases the Susceptibility to Cardiac Alternans.
P50
description
researcher
@en
wetenschapper
@nl
name
Mingke Ni
@en
Mingke Ni
@nl
type
label
Mingke Ni
@en
Mingke Ni
@nl
prefLabel
Mingke Ni
@en
Mingke Ni
@nl
P31
P496
0000-0002-1100-8265