about
Activation of microglial P2Y12 receptor is required for outward potassium currents in response to neuronal injury.Microglial Hv1 proton channel promotes cuprizone-induced demyelination through oxidative damage.Microglial P2Y12 receptors regulate microglial activation and surveillance during neuropathic pain.Microglia and monocytes synergistically promote the transition from acute to chronic pain after nerve injuryInterleukin-1β overproduction is a common cause for neuropathic pain, memory deficit, and depression following peripheral nerve injury in rodentsRegulation of Physical Microglia-Neuron Interactions by Fractalkine Signaling after Status Epilepticus.TNF-α Differentially Regulates Synaptic Plasticity in the Hippocampus and Spinal Cord by Microglia-Dependent Mechanisms after Peripheral Nerve Injury.Glutamate receptors in microglia.Microglia-Neuron Communication in Epilepsy.Fingolimod Protects Against Ischemic White Matter Damage by Modulating Microglia Toward M2 Polarization via STAT3 Pathway.The GluN2A Subunit Regulates Neuronal NMDA receptor-Induced Microglia-Neuron Physical Interactions.Extracellular ATP enhances radiation-induced brain injury through microglial activation and paracrine signaling via P2X7 receptor.Chemokine CCL2-CCR2 Signaling Induces Neuronal Cell Death via STAT3 Activation and IL-1β Production after Status Epilepticus.P2Y12R-Dependent Translocation Mechanisms Gate the Changing Microglial Landscape.
P50
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P50
description
researcher
@en
wetenschapper
@nl
name
Madhuvika Murugan
@en
Madhuvika Murugan
@nl
type
label
Madhuvika Murugan
@en
Madhuvika Murugan
@nl
prefLabel
Madhuvika Murugan
@en
Madhuvika Murugan
@nl
P31
P496
0000-0002-0706-0730