about
Widespread Coronary Dysfunction in the Absence of HDL Receptor SR-B1 in an Ischemic Cardiomyopathy Mouse Model.High-density lipoprotein delivered after myocardial infarction increases cardiac glucose uptake and function in mice.Chronic intermittent hypoxia accelerates coronary microcirculatory dysfunction in insulin-resistant Goto-Kakizaki rats.Cooperative action of APJ and α1A-adrenergic receptor in vascular smooth muscle cells induces vasoconstrictionMitochondrial Fusion by M1 Promotes Embryoid Body Cardiac Differentiation of Human Pluripotent Stem CellsDiastolic dysfunction is initiated by cardiomyocyte impairment ahead of endothelial dysfunction due to increased oxidative stress and inflammation in an experimental prediabetes model
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description
researcher
@en
wetenschapper
@nl
name
Yi-Ching Chen
@en
Yi-Ching Chen
@nl
type
label
Yi-Ching Chen
@en
Yi-Ching Chen
@nl
prefLabel
Yi-Ching Chen
@en
Yi-Ching Chen
@nl
P31
P496
0000-0003-4683-8813