Rotavirus NSP1 inhibits NFkappaB activation by inducing proteasome-dependent degradation of beta-TrCP: a novel mechanism of IFN antagonism - Test2 - elhamkhani - TriplyDB

Rotavirus NSP1 inhibits NFkappaB activation by inducing proteasome-dependent degradation of beta-TrCP: a novel mechanism of IFN antagonism

Rotavirus NSP1 inhibits NFkappaB activation by inducing proteasome-dependent degradation of beta-TrCP: a novel mechanism of IFN antagonism

http://www.wikidata.org/entity/Q21090517

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Rotavirus nonstructural protein 1 antagonizes innate immune response by interacting with retinoic acid inducible gene I MAVS protein is attenuated by rotavirus nonstructural protein 1 Rotaviruses: from pathogenesis to vaccination Rotavirus nonstructural protein 1 suppresses virus-induced cellular apoptosis to facilitate viral growth by activating the cell survival pathways during early stages of infection Inflammatory and oxidative stress in rotavirus infection Viral Mimicry to Usurp Ubiquitin and SUMO Host Pathways The role of ubiquitylation in immune defence and pathogen evasion The battle between rotavirus and its host for control of the interferon signaling pathway Rotavirus non-structural proteins: structure and function The Rotavirus Interferon Antagonist NSP1: Many Targets, Many Questions Comparative Proteomics Reveals Strain-Specific β-TrCP Degradation via Rotavirus NSP1 Hijacking a Host Cullin-3-Rbx1 Complex Modulation of NF-κB signalling by microbial pathogens Nlrp9b inflammasome restricts rotavirus infection in intestinal epithelial cells.Rotavirus structural proteins and dsRNA are required for the human primary plasmacytoid dendritic cell IFNalpha response Absence of genetic differences among G10P[11] rotaviruses associated with asymptomatic and symptomatic neonatal infections in Vellore, India.Viral hijacking of the host ubiquitin system to evade interferon responses.Negative regulation of NF-κB activity by brain-specific TRIpartite Motif protein 9.Identification of cellular calcium binding protein calmodulin as a regulator of rotavirus A infection during comparative proteomic study.Poxvirus targeting of E3 ligase β-TrCP by molecular mimicry: a mechanism to inhibit NF-κB activation and promote immune evasion and virulence.Diversity of interferon antagonist activities mediated by NSP1 proteins of different rotavirus strains.Cullin E3 ligases and their rewiring by viral factors Characterization of rotavirus RNAs that activate innate immune signaling through the RIG-I-like receptors.Putative E3 ubiquitin ligase of human rotavirus inhibits NF-κB activation by using molecular mimicry to target β-TrCP.The early interferon response to rotavirus is regulated by PKR and depends on MAVS/IPS-1, RIG-I, MDA-5, and IRF3.Microbial strategies for antagonizing Toll-like-receptor signal transduction.Molecular epidemiology and genetic evolution of the whole genome of G3P[8] human rotavirus in Wuhan, China, from 2000 through 2013 Rotavirus antagonism of the innate immune response Replication of the rotavirus genome requires an active ubiquitin-proteasome system.Type I interferon response is delayed in human astrovirus infections.The ORF61 Protein Encoded by Simian Varicella Virus and Varicella-Zoster Virus Inhibits NF-κB Signaling by Interfering with IκBα Degradation.The role of type I interferons in intestinal infection, homeostasis, and inflammation.Rotavirus immune responses and correlates of protection.Multiple UBXN family members inhibit retrovirus and lentivirus production and canonical NFκΒ signaling by stabilizing IκBα.Innate immune response to homologous rotavirus infection in the small intestinal villous epithelium at single-cell resolution.Anti-rotaviral effects of Glycyrrhiza uralensis extract in piglets with rotavirus diarrhea.The Role of F-Box Proteins during Viral Infection.Viral infection. Prevention and cure of rotavirus infection via TLR5/NLRC4-mediated production of IL-22 and IL-18 β-TrCP-mediated ubiquitination and degradation of liver-enriched transcription factor CREB-H.Rotavirus NSP1 Associates with Components of the Cullin RING Ligase Family of E3 Ubiquitin Ligases.PPARγ Agonists as an Anti-Inflammatory Treatment Inhibiting Rotavirus Infection of Small Intestinal Villi.

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Rotavirus NSP1 inhibits NFkappaB activation by inducing proteasome-dependent degradation of beta-TrCP: a novel mechanism of IFN antagonism

http://www.wikidata.org/entity/Q21090517

description

2009 nî lūn-bûn

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2009 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2009年の論文

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Rotavirus NSP1 inhibits NFkapp ...... el mechanism of IFN antagonism

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Joel W Graff

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Khalil Ettayebi

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Michele E Hardy

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Virus-dependent phosphorylation of the IRF-3 transcription factor regulates nuclear translocation, transactivation potential, and proteasome-mediated degradation

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