Outer surface protein B is critical for Borrelia burgdorferi adherence and survival within Ixodes ticks
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Biology of infection with Borrelia burgdorferiRole of outer surface protein D in the Borrelia burgdorferi life cycleReviewing molecular adaptations of Lyme borreliosis spirochetes in the context of reproductive fitness in natural transmission cyclesIncreasing the interaction of Borrelia burgdorferi with decorin significantly reduces the 50 percent infectious dose and severely impairs dissemination.Borrelia burgdorferi complement regulator-acquiring surface protein 2 does not contribute to complement resistance or host infectivity.The chitobiose transporter, chbC, is required for chitin utilization in Borrelia burgdorferi.Passage through Ixodes scapularis ticks enhances the virulence of a weakly pathogenic isolate of Borrelia burgdorferi.Analysis of the dbpBA upstream regulatory region controlled by RpoS in Borrelia burgdorferi.The abundance of the Lyme disease pathogen Borrelia afzelii declines over time in the tick vector Ixodes ricinus.Toll-like receptors 1 and 2 heterodimers alter Borrelia burgdorferi gene expression in mice and ticks.The bba64 gene of Borrelia burgdorferi, the Lyme disease agent, is critical for mammalian infection via tick bite transmission.Functional analysis of the Borrelia burgdorferi bba64 gene product in murine infection via tick infestation.Cyclic di-GMP is essential for the survival of the lyme disease spirochete in ticksAnaplasma phagocytophilum induces actin phosphorylation to selectively regulate gene transcription in Ixodes scapularis ticks.Ecology of Borrelia burgdorferi sensu lato in Europe: transmission dynamics in multi-host systems, influence of molecular processes and effects of climate change.Anaplasma phagocytophilum induces Ixodes scapularis ticks to express an antifreeze glycoprotein gene that enhances their survival in the cold.Lipoproteins of bacterial pathogens.Expression of Ixodes scapularis antifreeze glycoprotein enhances cold tolerance in Drosophila melanogaster.Genome stability of Lyme disease spirochetes: comparative genomics of Borrelia burgdorferi plasmids.Two regulatory elements required for enhancing ospA expression in Borrelia burgdorferi grown in vitro but repressing its expression during mammalian infection.Borrelia burgdorferi BBA52 is a potential target for transmission blocking Lyme disease vaccine.The role of Borrelia burgdorferi outer surface proteinsFlagellin and outer surface proteins from Borrelia burgdorferi are not glycosylated.Borrelia burgdorferi and tick proteins supporting pathogen persistence in the vector.A tightly regulated surface protein of Borrelia burgdorferi is not essential to the mouse-tick infectious cycleFunctional Equivalence of OspA and OspB, but Not OspC, in Tick Colonization by Borrelia burgdorferiMotility is crucial for the infectious life cycle of Borrelia burgdorferi.Outer surface protein A protects Lyme disease spirochetes from acquired host immunity in the tick vector.Essential protective role attributed to the surface lipoproteins of Borrelia burgdorferi against innate defencesBorrelia burgdorferi bba66 gene inactivation results in attenuated mouse infection by tick transmissionA manganese transporter, BB0219 (BmtA), is required for virulence by the Lyme disease spirochete, Borrelia burgdorferi.Borreliosis: recent research, diagnosis, and management.Evidence that BosR (BB0647) Is a Positive Autoregulator in Borrelia burgdorferiBorrelia burgdorferi small lipoprotein Lp6.6 is a member of multiple protein complexes in the outer membrane and facilitates pathogen transmission from ticks to mice.Gut microbiota of the tick vector Ixodes scapularis modulate colonization of the Lyme disease spirochete.The lipid raft proteome of Borrelia burgdorferi.Physiologic and Genetic Factors Influencing the Zoonotic Cycle of Borrelia burgdorferi.Sequences essential for transmission of Spiroplasma citri by its leafhopper vector, Circulifer haematoceps, revealed by plasmid curing and replacement based on incompatibility.Global Tn-seq analysis of carbohydrate utilization and vertebrate infectivity of Borrelia burgdorferi.Verification and dissection of the ospC operator by using flaB promoter as a reporter in Borrelia burgdorferi.
P2860
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P2860
Outer surface protein B is critical for Borrelia burgdorferi adherence and survival within Ixodes ticks
description
2007 nî lūn-bûn
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2007 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մարտին հրատարակված գիտական հոդված
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2007年の論文
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2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
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2007年论文
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name
Outer surface protein B is cri ...... d survival within Ixodes ticks
@ast
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en-gb
Outer surface protein B is cri ...... d survival within Ixodes ticks
@nl
type
label
Outer surface protein B is cri ...... d survival within Ixodes ticks
@ast
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en-gb
Outer surface protein B is cri ...... d survival within Ixodes ticks
@nl
altLabel
Outer Surface Protein B Is Cri ...... d Survival within Ixodes Ticks
@en
prefLabel
Outer surface protein B is cri ...... d survival within Ixodes ticks
@ast
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en-gb
Outer surface protein B is cri ...... d survival within Ixodes ticks
@nl
P2093
P2860
P1433
P1476
Outer surface protein B is cri ...... d survival within Ixodes ticks
@en
P2093
Deborah S Beck
Fred S Kantor
Girish Neelakanta
Kathleen DePonte
Xianzhong Liu
P2860
P356
10.1371/JOURNAL.PPAT.0030033
P407
P577
2007-03-01T00:00:00Z