Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss - Test2 - elhamkhani - TriplyDB

Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

http://www.wikidata.org/entity/Q21145275

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Comprehensive analysis of interactions between the Src-associated protein in mitosis of 68 kDa and the human Src-homology 3 proteome The STAR RNA binding proteins GLD-1, QKI, SAM68 and SLM-2 bind bipartite RNA motifs The RNA recognition motif protein RBM11 is a novel tissue-specific splicing regulator SAM68 regulates neuronal activity-dependent alternative splicing of neurexin-1 Sam68 is required for both NF-κB activation and apoptosis signaling by the TNF receptor Sam68 regulates translation of target mRNAs in male germ cells, necessary for mouse spermatogenesis Identification of a Sam68 ribonucleoprotein complex regulated by epidermal growth factor Sam68 regulates a set of alternatively spliced exons during neurogenesis SAM68: Signal Transduction and RNA Metabolism in Human Cancer The neurogenetics of alternative splicing Sam68 haploinsufficiency delays onset of mammary tumorigenesis and metastasis High expression level and nuclear localization of Sam68 are associated with progression and poor prognosis in colorectal cancer Esg1, expressed exclusively in preimplantation embryos, germline, and embryonic stem cells, is a putative RNA-binding protein with broad RNA targets An adaptor role for cytoplasmic Sam68 in modulating Src activity during cell polarization.A mouse chromosome 4 balancer ENU-mutagenesis screen isolates eleven lethal lines.Proteomic identification of heterogeneous nuclear ribonucleoprotein L as a novel component of SLM/Sam68 Nuclear Bodies.Global survey of protein expression during gonadal sex determination in mice Control of messenger RNA fate by RNA-binding proteins: an emphasis on mammalian spermatogenesis Transcription and post-transcriptional regulation of spermatogenesis.The RNA binding protein SAM68 transiently localizes in the chromatoid body of male germ cells and influences expression of select microRNAs.The tissue-specific RNA binding protein T-STAR controls regional splicing patterns of neurexin pre-mRNAs in the brain Sam68 relocalization into stress granules in response to oxidative stress through complexing with TIA-1 Sam68 marks the transcriptionally active stages of spermatogenesis and modulates alternative splicing in male germ cells.Inhibition of Sam68 triggers adipose tissue browning.Genetic analysis of low survival rate of pups in RR/Sgn inbred mice.SAM68 is a physiological regulator of SMN2 splicing in spinal muscular atrophy.Alternative splicing switching in stem cell lineages.Emerging roles for Sam68 in adipogenesis and neuronal development.RNA-binding protein Sam68 controls synapse number and local β-actin mRNA metabolism in dendrites.Stay lean without dieting: Lose Sam68 Targeting the RNA-binding protein Sam68 as a treatment for cancer?Selective translational repression of HIV-1 RNA by Sam68DeltaC occurs by altering PABP1 binding to unspliced viral RNA Sam68/KHDRBS1 is critical for colon tumorigenesis by regulating genotoxic stress-induced NF-κB activation.The epigenetics of adult (somatic) stem cells.RNA binding proteins in the regulation of heart development.Role of RNA-binding proteins in mammalian spermatogenesis.Role of Sam68 in post-transcriptional gene regulation.Sam68 promotes self-renewal and glycolytic metabolism in mouse neural progenitor cells by modulating Aldh1a3 pre-mRNA 3'-end processing.Transgenic mice with OIP-1/hSca overexpression targeted to the osteoclast lineage develop an osteopetrosis bone phenotype.The splicing landscape is globally reprogrammed during male meiosis.

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

http://www.wikidata.org/entity/Q21145275

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2005 nî lūn-bûn

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2005 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA Binding Protein Protects Mice from Age-Related Bone Loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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Ablation of the Sam68 RNA binding protein protects mice from age-related bone loss

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The novel zinc finger-containing transcription factor osterix is required for osteoblast differentiation and bone formation

Molecular cloning and nucleic acid binding properties of the GAP-associated tyrosine phosphoprotein p62

Association of p62, a multifunctional SH2- and SH3-domain-binding protein, with src family tyrosine kinases, Grb2, and phospholipase C gamma-1

Characterization of Sam68-like mammalian proteins SLM-1 and SLM-2: SLM-1 is a Src substrate during mitosis

The Dlx5 and Dlx6 homeobox genes are essential for craniofacial, axial, and appendicular skeletal development

Multilineage potential of adult human mesenchymal stem cells

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