Lethal influenza virus infection in macaques is associated with early dysregulation of inflammatory related genes - Test2 - elhamkhani - TriplyDB

Lethal influenza virus infection in macaques is associated with early dysregulation of inflammatory related genes

Lethal influenza virus infection in macaques is associated with early dysregulation of inflammatory related genes

http://www.wikidata.org/entity/Q21559413

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Recently emerged swine influenza A virus (H2N3) causes severe pneumonia in Cynomolgus macaques Experimental primates and non-human primate (NHP) models of human diseases in China: current status and progress Systems approaches to influenza-virus host interactions and the pathogenesis of highly virulent and pandemic viruses Human H7N9 avian influenza virus infection: a review and pandemic risk assessment Pathogenicity of pandemic H1N1 influenza A virus in immunocompromised cynomolgus macaques Evidence for a Novel Mechanism of Influenza Virus-Induced Type I Interferon Expression by a Defective RNA-Encoded Protein An Ultrasensitive Mechanism Regulates Influenza Virus-Induced Inflammation Cross-reactive T cells are involved in rapid clearance of 2009 pandemic H1N1 influenza virus in nonhuman primates Central roles of NLRs and inflammasomes in viral infection Allele-specific induction of IL-1β expression by C/EBPβ and PU.1 contributes to increased tuberculosis susceptibility Mesenchymal stromal cell treatment prevents H9N2 avian influenza virus-induced acute lung injury in mice Innate immune response of human alveolar macrophages during influenza A infection Avian influenza A H5N1 virus: a continuous threat to humans.Disease severity is associated with differential gene expression at the early and late phases of infection in nonhuman primates infected with different H5N1 highly pathogenic avian influenza viruses The use of nonhuman primates in research on seasonal, pandemic and avian influenza, 1893-2014.Pandemic Swine-Origin H1N1 Influenza Virus Replicates to Higher Levels and Induces More Fever and Acute Inflammatory Cytokines in Cynomolgus versus Rhesus Monkeys and Can Replicate in Common Marmosets.microRNAs Regulate Host Immune Response and Pathogenesis During Influenza Infection in Rhesus Macaques.Lethal dissemination of H5N1 influenza virus is associated with dysregulation of inflammation and lipoxin signaling in a mouse model of infection.Genomic profiling of tumor necrosis factor alpha (TNF-alpha) receptor and interleukin-1 receptor knockout mice reveals a link between TNF-alpha signaling and increased severity of 1918 pandemic influenza virus infection.Viral replication rate regulates clinical outcome and CD8 T cell responses during highly pathogenic H5N1 influenza virus infection in mice Sensitization with vaccinia virus encoding H5N1 hemagglutinin restores immune potential against H5N1 influenza virus.Pandemic swine-origin H1N1 influenza A virus isolates show heterogeneous virulence in macaques Pandemic (H1N1) 2009 influenza virus induces weaker host immune responses in vitro: a possible mechanism of high transmissibility.Suppression of cytokine storm with a sphingosine analog provides protection against pathogenic influenza virus.Influenza A viruses: new research developments.Temporal dynamics of host molecular responses differentiate symptomatic and asymptomatic influenza a infection.Viral infection of the lung: host response and sequelae Molecular signatures associated with Mx1-mediated resistance to highly pathogenic influenza virus infection: mechanisms of survival Case report: Increased viral receptor expression associated with high viral load and severe pneumonia in a young patient infected with 2009 H1N1 influenza a with no pre-existing conditions.A computationally optimized hemagglutinin virus-like particle vaccine elicits broadly reactive antibodies that protect nonhuman primates from H5N1 infection.Increased viral loads and exacerbated innate host responses in aged macaques infected with the 2009 pandemic H1N1 influenza A virus.Integrated network analysis reveals a novel role for the cell cycle in 2009 pandemic influenza virus-induced inflammation in macaque lungs H5N1 pathogenesis studies in mammalian models.Systems virology: host-directed approaches to viral pathogenesis and drug targeting.Massive mobilization of dendritic cells during influenza A virus subtype H5N1 infection of nonhuman primates Mixed Lineage Kinase 3 deficiency delays viral clearance in the lung and is associated with diminished influenza-induced cytopathic effect in infected cells Inhibition of the inflammatory cytokine tumor necrosis factor-alpha with etanercept provides protection against lethal H1N1 influenza infection in mice.High interferon type I responses in the lung, plasma and spleen during highly pathogenic H5N1 infection of chicken.Type I interferon signaling regulates Ly6C(hi) monocytes and neutrophils during acute viral pneumonia in mice Influenza-Omics and the Host Response: Recent Advances and Future Prospects.

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Lethal influenza virus infection in macaques is associated with early dysregulation of inflammatory related genes

http://www.wikidata.org/entity/Q21559413

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2009 nî lūn-bûn

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2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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Cristian Cillóniz

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Friedericke Feldmann

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James E Strong

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Jean H Chang

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Kyoko Shinya

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Lauri D Aicher

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Marcus J Korth

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Sean C Proll

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Victoria S Carter

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Xinxia Peng

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P2860

Pandemic potential of a strain of influenza A (H1N1): early findings

Interferon-induced ISG15 conjugation inhibits influenza A virus gene expression and replication in human cells

ISG15 inhibits Ebola VP40 VLP budding in an L-domain-dependent manner by blocking Nedd4 ligase activity

Human ISG15 conjugation targets both IFN-induced and constitutively expressed proteins functioning in diverse cellular pathways

Identification of interferon-stimulated gene 15 as an antiviral molecule during Sindbis virus infection in vivo.

ISG15 inhibits Nedd4 ubiquitin E3 activity and enhances the innate antiviral response

The intracellular sensor NLRP3 mediates key innate and healing responses to influenza A virus via the regulation of caspase-1

Genomic analysis of increased host immune and cell death responses induced by 1918 influenza virus

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

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10

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Yoshihiro Kawaoka

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2009-10-01T00:00:00Z

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26864796

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