Herpes simplex virus 1 regulatory protein ICP22 interacts with a new cell cycle-regulated factor and accumulates in a cell cycle-dependent fashion in infected cells.
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Repressor element-1 silencing transcription factor/neuronal restrictive silencer factor (REST/NRSF) can regulate HSV-1 immediate-early transcription via histone modificationA novel cellular protein, p60, interacting with both herpes simplex virus 1 regulatory proteins ICP22 and ICP0 is modified in a cell-type-specific manner and Is recruited to the nucleus after infectionFunctional anatomy of herpes simplex virus 1 overlapping genes encoding infected-cell protein 22 and US1.5 proteinThe EICP22 protein of equine herpesvirus 1 physically interacts with the immediate-early protein and with itself to form dimers and higher-order complexesMCRS2 represses the transactivation activities of Nrf1.The disappearance of cyclins A and B and the increase in activity of the G(2)/M-phase cellular kinase cdc2 in herpes simplex virus 1-infected cells require expression of the alpha22/U(S)1.5 and U(L)13 viral genes.The U(L)3 protein of herpes simplex virus 1 is translated predominantly from the second in-frame methionine codon and is subject to at least two posttranslational modificationsPerturbation of cell cycle progression and cellular gene expression as a function of herpes simplex virus ICP0.Cellular transformation by the MSP58 oncogene is inhibited by its physical interaction with the PTEN tumor suppressor.HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.Herpes simplex virus 1 ICP22 inhibits the transcription of viral gene promoters by binding to and blocking the recruitment of P-TEFbICP22 is required for wild-type composition and infectivity of herpes simplex virus type 1 virions.Sequence variation in the herpes simplex virus U(S)1 ocular virulence determinant.Herpes simplex virus immediate-early protein ICP22 triggers loss of serine 2-phosphorylated RNA polymerase IIRoles of p53 in herpes simplex virus 1 replication.Characterization of the interaction between human respiratory syncytial virus and the cell cycle in continuous cell culture and primary human airway epithelial cells.Immediate-early expression of the herpes simplex virus type 1 ICP27 transcript is not critical for efficient replication in vitro or in vivoThe products of the herpes simplex virus type 1 immediate-early US1/US1.5 genes downregulate levels of S-phase-specific cyclins and facilitate virus replication in S-phase Vero cells.
P2860
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P2860
Herpes simplex virus 1 regulatory protein ICP22 interacts with a new cell cycle-regulated factor and accumulates in a cell cycle-dependent fashion in infected cells.
description
1998 nî lūn-bûn
@nan
1998 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Herpes simplex virus 1 regulat ...... dent fashion in infected cells
@nl
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@ast
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@en
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@en-gb
type
label
Herpes simplex virus 1 regulat ...... dent fashion in infected cells
@nl
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@ast
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@en
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@en-gb
prefLabel
Herpes simplex virus 1 regulat ...... dent fashion in infected cells
@nl
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@ast
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@en
Herpes simplex virus 1 regulat ...... ent fashion in infected cells.
@en-gb
P2860
P921
P1433
P1476
Herpes simplex virus 1 regulat ...... dent fashion in infected cells
@en
P2093
P2860
P304
P407
P577
1998-11-01T00:00:00Z