Solution structure of the CIDE-N domain of CIDE-B and a model for CIDE-N/CIDE-N interactions in the DNA fragmentation pathway of apoptosis
about
The hepatitis C virus NS2 protein is an inhibitor of CIDE-B-induced apoptosisComparative analysis of apoptosis and inflammation genes of mice and humansMolecular cloning and characterization of CIDE-3, a novel member of the cell-death-inducing DNA-fragmentation-factor (DFF45)-like effector familyIdentification of ICAD-derived peptides capable of inhibiting caspase-activated DNaseDual promoters control the cell-specific expression of the human cell death-inducing DFF45-like effector B geneSolution structure of DFF40 and DFF45 N-terminal domain complex and mutual chaperone activity of DFF40 and DFF45MiR-145, a new regulator of the DNA fragmentation factor-45 (DFF45)-mediated apoptotic networkAberrant caspase-activated DNase (CAD) transcripts in human hepatoma cellsCell-death-inducing DFFA-like Effector B Contributes to the Assembly of Hepatitis C Virus (HCV) Particles and Interacts with HCV NS5AMitochondria localization and dimerization are required for CIDE-B to induce apoptosisThe brown adipocyte protein CIDEA promotes lipid droplet fusion via a phosphatidic acid-binding amphipathic helixSalicylate-induced degeneration of cochlea spiral ganglion neurons-apoptosis signalingiSNO-AAPair: incorporating amino acid pairwise coupling into PseAAC for predicting cysteine S-nitrosylation sites in proteinsAutomated search of natively folded protein fragments for high-throughput structure determination in structural genomics.Cell death regulation in Drosophila: conservation of mechanism and unique insights.Molecular evolution of Cide family proteins: novel domain formation in early vertebrates and the subsequent divergenceProteases for cell suicide: functions and regulation of caspases.Polymorphic toxin systems: Comprehensive characterization of trafficking modes, processing, mechanisms of action, immunity and ecology using comparative genomicsCharacterization of splice variants of human caspase-activated DNase with CIDE-N structure and function.The domains of apoptosis: a genomics perspective.Gene flow and biological conflict systems in the origin and evolution of eukaryotes.Crystallization and preliminary X-ray crystallographic studies of the CIDE-domain complex between Drep2 and Drep3 from Drosophila melanogaster.Regulation of ATM/DNA-PKcs Phosphorylation by BRCA1-Associated BAAT1.The Complete Genome Sequence of a Second Distinct Betabaculovirus from the True Armyworm, Mythimna unipunctaCrystallization and preliminary X-ray crystallographic studies of Drep-3, a DFF-related protein from Drosophila melanogasterGeneration of aberrant forms of DFF40 concurrent with caspase-3 activation during acute and chronic liver injury in rats.Crystallization and preliminary X-ray crystallographic studies of the CIDE-N domain of CIDE-3.Which CIDE are you on? Apoptosis and energy metabolism.Structural insight into CIDE domains: the Janus face of CIDEs.Investigating the antiviral role of cell death-inducing DFF45-like effector B in HCV replication.Identification of functionally relevant histidine residues in the apoptotic nuclease CAD.Cell death-inducing DFF45-like effector b (Cideb) is present in pancreatic beta-cells and involved in palmitate induced beta-cell apoptosis.Involvement of conserved histidine, lysine and tyrosine residues in the mechanism of DNA cleavage by the caspase-3 activated DNase CAD.Analysis of an artificially selected GS-NS0 variant with increased resistance to apoptosis.Anti-proliferative and pro-apoptotic actions of a novel human and mouse ovarian tumor-associated gene OTAG-12: downregulation, alternative splicing and drug sensitization.The effect of ICAD-S on the formation and intracellular distribution of a nucleolytically active caspase-activated DNase.Functional analysis of FSP27 protein regions for lipid droplet localization, caspase-dependent apoptosis, and dimerization with CIDEA.Regulation of Cidea protein stability by the ubiquitin-mediated proteasomal degradation pathway.Oligomerization state of the DNA fragmentation factor in normal and apoptotic cells.Compound heterozygous BRAT1 mutations cause familial Ohtahara syndrome with hypertonia and microcephaly.
P2860
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P2860
Solution structure of the CIDE-N domain of CIDE-B and a model for CIDE-N/CIDE-N interactions in the DNA fragmentation pathway of apoptosis
description
1999 nî lūn-bûn
@nan
1999 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Solution structure of the CIDE ...... mentation pathway of apoptosis
@ast
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en-gb
Solution structure of the CIDE ...... mentation pathway of apoptosis
@nl
type
label
Solution structure of the CIDE ...... mentation pathway of apoptosis
@ast
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en-gb
Solution structure of the CIDE ...... mentation pathway of apoptosis
@nl
prefLabel
Solution structure of the CIDE ...... mentation pathway of apoptosis
@ast
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en-gb
Solution structure of the CIDE ...... mentation pathway of apoptosis
@nl
P2093
P921
P3181
P1433
P1476
Solution structure of the CIDE ...... mentation pathway of apoptosis
@en
P2093
P304
P3181
P356
10.1016/S0092-8674(00)81672-4
P407
P577
1999-12-23T00:00:00Z