about
Rapalogs in cancer prevention: anti-aging or anticancer?GSK-3 as potential target for therapeutic intervention in cancerAdvances in targeting signal transduction pathwaysDiscovery and SAR of Novel 2,3-Dihydroimidazo[1,2-c]quinazoline PI3K Inhibitors: Identification of Copanlisib (BAY 80-6946)Selective anti-cancer agents as anti-aging drugsLentiviral vector-based insertional mutagenesis identifies genes involved in the resistance to targeted anticancer therapies.Elevated PI3K signaling drives multiple breast cancer subtypes.Molecular damage in cancer: an argument for mTOR-driven agingRecent progress in targeting cancer.NCI's provocative questions on cancer: some answers to ignite discussion.Ectopic NGAL expression can alter sensitivity of breast cancer cells to EGFR, Bcl-2, CaM-K inhibitors and the plant natural product berberineMutations and deregulation of Ras/Raf/MEK/ERK and PI3K/PTEN/Akt/mTOR cascades which alter therapy response.Upregulated WDR26 serves as a scaffold to coordinate PI3K/ AKT pathway-driven breast cancer cell growth, migration, and invasion.Signaling via class IA Phosphoinositide 3-kinases (PI3K) in human, breast-derived cell linesProgress in the preclinical discovery and clinical development of class I and dual class I/IV phosphoinositide 3-kinase (PI3K) inhibitors.Abi1, a critical molecule coordinating actin cytoskeleton reorganization with PI-3 kinase and growth signaling.PI-3 kinase p110β: a therapeutic target in advanced prostate cancers.PI3King the right partner: unique interactions and signaling by p110β.ErbB2-positive mammary tumors can escape PI3K-p110α loss through downregulation of the Pten tumor suppressor.Feedback suppression of PI3Kα signaling in PTEN-mutated tumors is relieved by selective inhibition of PI3Kβ.Decatenation checkpoint-defective melanomas are dependent on PI3K for survival.Prodrug strategy for PSMA-targeted delivery of TGX-221 to prostate cancer cells.[3a,4]-Dihydropyrazolo[1,5a]pyrimidines: Novel, Potent, and Selective Phosphatidylinositol-3-kinase β Inhibitors.The Role of Class IA Phosphatidylinositol-4,5-Bisphosphate 3-Kinase Catalytic Subunits in Glioblastoma.miR-101-2, miR-125b-2 and miR-451a act as potential tumor suppressors in gastric cancer through regulation of the PI3K/AKT/mTOR pathway.
P2860
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P2860
description
2010 nî lūn-bûn
@nan
2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
A beta version of life: p110β takes center stage
@ast
A beta version of life: p110β takes center stage
@en
A beta version of life: p110β takes center stage
@en-gb
A beta version of life: p110β takes center stage
@nl
type
label
A beta version of life: p110β takes center stage
@ast
A beta version of life: p110β takes center stage
@en
A beta version of life: p110β takes center stage
@en-gb
A beta version of life: p110β takes center stage
@nl
prefLabel
A beta version of life: p110β takes center stage
@ast
A beta version of life: p110β takes center stage
@en
A beta version of life: p110β takes center stage
@en-gb
A beta version of life: p110β takes center stage
@nl
P2860
P921
P356
P1433
P1476
A beta version of life: p110β takes center stage
@en
P2093
Hashem A Dbouk
Jonathan M Backer
P2860
P304
P356
10.18632/ONCOTARGET.207
P407
P5008
P577
2010-12-01T00:00:00Z