Chemotactic activity of S100A7 (Psoriasin) is mediated by the receptor for advanced glycation end products and potentiates inflammation with highly homologous but functionally distinct S100A15
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The secreted protein S100A7 (psoriasin) is induced by telomere dysfunction in human keratinocytes independently of a DNA damage response and cell cycle regulatorsGene from a psoriasis susceptibility locus primes the skin for inflammationS100A7 enhances mammary tumorigenesis through upregulation of inflammatory pathwaysStructural characterization of S100A15 reveals a novel zinc coordination site among S100 proteins and altered surface chemistry with functional implications for receptor bindingHighly homologous hS100A15 and hS100A7 proteins are distinctly expressed in normal breast tissue and breast cancerLeukocyte integrins: role in leukocyte recruitment and as therapeutic targets in inflammatory diseaseThe new insight into the role of antimicrobial proteins-alarmins in the immunopathogenesis of psoriasisStructural and functional characterization of a triple mutant form of S100A7 defective for Jab1 bindingBinding of transition metals to S100 proteinsNovel S100A7 (psoriasin)/S100A15 (koebnerisin) subfamily: highly homologous but distinct in regulation and functionPeripheral immune cell gene expression changes in advanced non-small cell lung cancer patients treated with first line combination chemotherapyNovel sulfated polysaccharides disrupt cathelicidins, inhibit RAGE and reduce cutaneous inflammation in a mouse model of rosaceaInteraction with adipocyte stromal cells induces breast cancer malignancy via S100A7 upregulation in breast cancer microenvironment.ANTI-INFECTIVE PROTECTIVE PROPERTIES OF S100 CALGRANULINS.Psoriasin (S100A7) increases the expression of ROS and VEGF and acts through RAGE to promote endothelial cell proliferation.Inflammation-mediated skin tumorigenesis induced by epidermal c-Fos.RAGE mediates S100A7-induced breast cancer growth and metastasis by modulating the tumor microenvironmentPost-operative recurrent trachomatous trichiasis is associated with increased conjunctival expression of S100A7 (psoriasin).The expression of Psoriasin (S100A7) and CD24 is linked and related to the differentiation of mammary epithelial cells.S100A9 induced inflammatory responses are mediated by distinct damage associated molecular patterns (DAMP) receptors in vitro and in vivo.Overexpression of S100A7 protects LPS-induced mitochondrial dysfunction and stimulates IL-6 and IL-8 in HaCaT cells.Diminished levels of nasal S100A7 (psoriasin) in seasonal allergic rhinitis: an effect mediated by Th2 cytokinesNutritional Immunity: S100 Proteins at the Host-Pathogen Interface.Glycation and insulin resistance: novel mechanisms and unique targets?Multiple Evolutionary Origins of Ubiquitous Cu2+ and Zn2+ Binding in the S100 Protein Family.Human S100A7 Induces Mature Interleukin1α Expression by RAGE-p38 MAPK-Calpain1 Pathway in Psoriasis.Th17 micro-milieu regulates NLRP1-dependent caspase-5 activity in skin autoinflammation.Chronic exposure to chewing tobacco selects for overexpression of stearoyl-CoA desaturase in normal oral keratinocytes.Gene profiling of narrowband UVB-induced skin injury defines cellular and molecular innate immune responsesFunctions of S100 proteins.The IL-23/T17 pathogenic axis in psoriasis is amplified by keratinocyte responses.RAGE (Receptor for Advanced Glycation Endproducts), RAGE ligands, and their role in cancer and inflammation.Endogenous damage-associated molecular pattern molecules at the crossroads of inflammation and cancer.The pattern recognition receptor CD36 is a chondrocyte hypertrophy marker associated with suppression of catabolic responses and promotion of repair responses to inflammatory stimuliA multidisciplinary approach to study a couple of monozygotic twins discordant for the chronic fatigue syndrome: a focus on potential salivary biomarkers.S100A8 and S100A9: DAMPs at the crossroads between innate immunity, traditional risk factors, and cardiovascular disease.Suppression of skin tumorigenesis in CD109-deficient mice.Inflammation-associated S100 proteins: new mechanisms that regulate function.The role of zinc in the S100 proteins: insights from the X-ray structures.Joining S100 proteins and migration: for better or for worse, in sickness and in health.
P2860
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P2860
Chemotactic activity of S100A7 (Psoriasin) is mediated by the receptor for advanced glycation end products and potentiates inflammation with highly homologous but functionally distinct S100A15
description
2008 nî lūn-bûn
@nan
2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@ast
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en-gb
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@nl
type
label
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@ast
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en-gb
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@nl
prefLabel
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@ast
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en-gb
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@nl
P2093
P2860
P921
P3181
P1476
Chemotactic activity of S100A7 ...... functionally distinct S100A15
@en
P2093
Bijan Ahvazi
Christopher Voscopoulos
Hui-Fang Dong
Jason Winston
Joost J Oppenheim
Karen Boeshans
Michele Gunsior
O M Zack Howard
Paul Goldsmith
P2860
P304
P3181
P356
10.4049/JIMMUNOL.181.2.1499
P407
P577
2008-07-15T00:00:00Z