Tau exon 10, whose missplicing causes frontotemporal dementia, is regulated by an intricate interplay of cis elements and trans factors
about
RBM4 interacts with an intronic element and stimulates tau exon 10 inclusionHeterogeneous nuclear ribonucleoprotein G regulates splice site selection by binding to CC(A/C)-rich regions in pre-mRNAMammalian splicing factor SF1 interacts with SURP domains of U2 snRNP-associated proteinsA microarray configuration to quantify expression levels and relative abundance of splice variants.Differential recruitment of pre-mRNA splicing factors to alternatively spliced transcripts in vivo.The importance of CELF control: molecular and biological roles of the CUG-BP, Elav-like family of RNA-binding proteinsCharacterization of the RNA recognition mode of hnRNP G extends its role in SMN2 splicing regulationSaitohin, which is nested in the tau locus and confers allele-specific susceptibility to several neurodegenerative diseases, interacts with peroxiredoxin 6Tau exons 2 and 10, which are misregulated in neurodegenerative diseases, are partly regulated by silencers which bind a SRp30c.SRp55 complex that either recruits or antagonizes htra2beta1Arginine/serine-rich protein interaction domain-dependent modulation of a tau exon 10 splicing enhancer: altered interactions and mechanisms for functionally antagonistic FTDP-17 mutations Delta280K AND N279KBioactive marine drugs and marine biomaterials for brain diseasesDifferential RNA-binding activity of the hnRNP G protein correlated with the sex genotype in the amphibian oocyte.Reprogramming of tau alternative splicing by spliceosome-mediated RNA trans-splicing: implications for tauopathies.The role of CELF proteins in neurological disorders.Cyclic AMP-dependent protein kinase regulates the alternative splicing of tau exon 10: a mechanism involved in tau pathology of Alzheimer disease.Functional Analysis of Mutations in Exon 9 of NF1 Reveals the Presence of Several Elements Regulating SplicingC6 pyridinium ceramide influences alternative pre-mRNA splicing by inhibiting protein phosphatase-1.MicroRNAs and the Regulation of Tau MetabolismStructural determinants for alternative splicing regulation of the MAPT pre-mRNAThe adipogenic transcriptional cofactor ZNF638 interacts with splicing regulators and influences alternative splicingTau exon 10 alternative splicing and tauopathiesCUG-BP, Elav-like family (CELF)-mediated alternative splicing regulation in the brain during health and disease.Genome-wide analysis of over 106 000 individuals identifies 9 neuroticism-associated loci.Alternative splicing and disease.PSF suppresses tau exon 10 inclusion by interacting with a stem-loop structure downstream of exon 10.Natural compounds from traditional medicinal herbs in the treatment of cerebral ischemia/reperfusion injury.Decoding the non-coding RNAs in Alzheimer's disease.Regulation of alternative splicing of tau exon 10.An SRp75/hnRNPG complex interacting with hnRNPE2 regulates the 5' splice site of tau exon 10, whose misregulation causes frontotemporal dementia.Heterogeneous nuclear ribonucleoprotein E3 modestly activates splicing of tau exon 10 via its proximal downstream intron, a hotspot for frontotemporal dementia mutations.Multiple and specific mRNA processing targets for the major human hnRNP proteins.Neuroprotective effects of cyclooxygenase-2 inhibitor celecoxib against toxicity of LPS-stimulated macrophages toward motor neurons.The Role of MicroRNAs in Aβ Deposition and Tau Phosphorylation in Alzheimer's Disease.MAPT Genetic Variation and Neuronal Maturity Alter Isoform Expression Affecting Axonal Transport in iPSC-Derived Dopamine Neurons.SR protein 9G8 modulates splicing of tau exon 10 via its proximal downstream intron, a clustering region for frontotemporal dementia mutationsMAP2 Splicing is Altered in Huntington's Disease.RBMX gene is essential for brain development in zebrafish.Cyclosporine decreases prostaglandin E2 production in mouse medullary thick ascending limb cultured cells.Challenges and opportunities in the investigation of unexplained intellectual disability using family-based whole-exome sequencing.Cyclic AMP-dependent protein kinase regulates 9G8-mediated alternative splicing of tau exon 10.
P2860
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P248
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P2860
Tau exon 10, whose missplicing causes frontotemporal dementia, is regulated by an intricate interplay of cis elements and trans factors
description
2004 nî lūn-bûn
@nan
2004 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի մարտին հրատարակված գիտական հոդված
@hy
2004年の論文
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2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
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2004年论文
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name
Tau exon 10, whose missplicing ...... cis elements and trans factors
@ast
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en-gb
Tau exon 10, whose missplicing ...... cis elements and trans factors
@nl
type
label
Tau exon 10, whose missplicing ...... cis elements and trans factors
@ast
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en-gb
Tau exon 10, whose missplicing ...... cis elements and trans factors
@nl
prefLabel
Tau exon 10, whose missplicing ...... cis elements and trans factors
@ast
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en-gb
Tau exon 10, whose missplicing ...... cis elements and trans factors
@nl
P2093
P921
P1476
Tau exon 10, whose missplicing ...... cis elements and trans factors
@en
P2093
Athena Andreadis
Junning Wang
Qing-Sheng Gao
Stefan Stamm
Yingzi Wang
P304
P356
10.1046/J.1471-4159.2003.02232.X
P407
P577
2004-03-01T00:00:00Z