Molecular mechanism of hTid-1, the human homolog of Drosophila tumor suppressor l(2)Tid, in the regulation of NF-kappaB activity and suppression of tumor growth
about
The tumor suppressor hTid1 inhibits STAT5b activity via functional interactionhTID-1 defines a novel regulator of c-Met Receptor signaling in renal cell carcinomasCellular stress response and innate immune signaling: integrating pathways in host defense and inflammation.HTLV-1 Tax deregulates autophagy by recruiting autophagic molecules into lipid raft microdomains.DNAJB6 induces degradation of beta-catenin and causes partial reversal of mesenchymal phenotype.Absence of a human DnaJ protein hTid-1S correlates with aberrant actin cytoskeleton organization in lesional psoriatic skin.Construction of a novel constitutively active chimeric EGFR to identify new targets for therapyTid1, the Mammalian Homologue of Drosophila Tumor Suppressor Tid56, Mediates Macroautophagy by Interacting with Beclin1-containing Autophagy Protein Complex.Quantitative Proteomics Reveals the Roles of Peroxisome-associated Proteins in Antiviral Innate Immune Responses.The Role of the Phylogenetically Conserved Cochaperone Protein Droj2/DNAJA3 in NF-κB Signaling.New insight into the oncogenic mechanism of the retroviral oncoprotein Tax.Sequestration of NF-kappaB signaling complexes in lipid rafts contributes to repression of NF-kappaB in T lymphocytes under hyperthermia stress.Transient IkappaB kinase activity mediates temporal NF-kappaB dynamics in response to a wide range of tumor necrosis factor-alpha doses.Tid1 is a Smad-binding protein that can modulate Smad7 activity in developing embryos.High nitric oxide production, secondary to inducible nitric oxide synthase expression, is essential for regulation of the tumour-initiating properties of colon cancer stem cells.Targeting Heat Shock Proteins in Cancer: A Promising Therapeutic ApproachHTLV-1 Tax is a critical lipid raft modulator that hijacks IkappaB kinases to the microdomains for persistent activation of NF-kappaB.Tazarotene-Induced Gene 1 Interacts with DNAJC8 and Regulates Glycolysis in Cervical Cancer Cells.
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P2860
Molecular mechanism of hTid-1, the human homolog of Drosophila tumor suppressor l(2)Tid, in the regulation of NF-kappaB activity and suppression of tumor growth
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@ast
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en-gb
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@nl
type
label
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@ast
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en-gb
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@nl
prefLabel
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@ast
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en-gb
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@nl
P2093
P2860
P1476
Molecular mechanism of hTid-1, ...... nd suppression of tumor growth
@en
P2093
Carlo Cenciarelli
Dominic Fan
Gina Nelkin
Isaiah J Fidler
Rachel Tsan
P2860
P356
10.1128/MCB.25.1.44-59.2005
P407
P577
2005-01-01T00:00:00Z