The lipid mediator protectin D1 inhibits influenza virus replication and improves severe influenza
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Making Bunyaviruses Talk: Interrogation Tactics to Identify Host Factors Required for InfectionOmega-3 Fatty Acids in Modern Parenteral Nutrition: A Review of the Current EvidenceQuantification of viral infection dynamics in animal experimentsLipidomic profiling of bioactive lipids by mass spectrometry during microbial infectionsEicosanoid storm in infection and inflammationThe role of pro-resolution lipid mediators in infectious diseaseAdvances in Our Understanding of Oxylipins Derived from Dietary PUFAsCompounds with anti-influenza activity: present and future of strategies for the optimal treatment and management of influenza. Part II: Future compounds against influenza virusEmerging targets in neuroinflammation-driven chronic painResolvin D1 attenuates polyinosinic-polycytidylic acid-induced inflammatory signaling in human airway epithelial cells via TAK1Lipid mediators in the resolution of inflammationThe specialized proresolving mediator 17-HDHA enhances the antibody-mediated immune response against influenza virus: a new class of adjuvant?The resolution code of acute inflammation: Novel pro-resolving lipid mediators in resolutionIdentification of resolvin D2 receptor mediating resolution of infections and organ protectionDHA- and EPA-derived resolvins, protectins, and maresins in airway inflammationSpecialized pro-resolving mediators: endogenous regulators of infection and inflammationThe effects of diet-induced obesity on B cell functionProtectins and maresins: New pro-resolving families of mediators in acute inflammation and resolution bioactive metabolomeRole for phospholipid acyl chains and cholesterol in pulmonary infections and inflammationAvian influenza A H7N9 virus induces severe pneumonia in mice without prior adaptation and responds to a combination of zanamivir and COX-2 inhibitorInflammation-a Critical Appreciation of the Role of Myeloid CellsTranscriptomic characterization of the novel avian-origin influenza A (H7N9) virus: specific host response and responses intermediate between avian (H5N1 and H7N7) and human (H3N2) viruses and implications for treatment optionsLack of group X secreted phospholipase A₂ increases survival following pandemic H1N1 influenza infection.H7N9 and other pathogenic avian influenza viruses elicit a three-pronged transcriptomic signature that is reminiscent of 1918 influenza virus and is associated with lethal outcome in miceA novel genetic locus linked to pro-inflammatory cytokines after virulent H5N1 virus infection in mice.Obesity Increases Mortality and Modulates the Lung Metabolome during Pandemic H1N1 Influenza Virus Infection in Mice.Integrated Omics Analysis of Pathogenic Host Responses during Pandemic H1N1 Influenza Virus Infection: The Crucial Role of Lipid Metabolism.Improving the estimation of the death rate of infected cells from time course data during the acute phase of virus infections: application to acute HIV-1 infection in a humanized mouse model.Pro-resolving lipid mediators are leads for resolution physiology.Seeking a unique lipid signature predicting cardiovascular disease risk.Deciphering the role of n-3 polyunsaturated fatty acid-derived lipid mediators in health and disease.Protectin DX increases survival in a mouse model of sepsis by ameliorating inflammation and modulating macrophage phenotype.Biofluid lipidome: a source for potential diagnostic biomarkers.Lipidomics identifies a requirement for peroxisomal function during influenza virus replicationIdentification and signature profiles for pro-resolving and inflammatory lipid mediators in human tissue.Systems-level analysis of innate immunity.Innate immunity to influenza virus infection.Comparative analysis of seven viral nuclear export signals (NESs) reveals the crucial role of nuclear export mediated by the third NES consensus sequence of nucleoprotein (NP) in influenza A virus replication.Phospholipase D facilitates efficient entry of influenza virus, allowing escape from innate immune inhibitionAging delays resolution of acute inflammation in mice: reprogramming the host response with novel nano-proresolving medicines
P2860
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P2860
The lipid mediator protectin D1 inhibits influenza virus replication and improves severe influenza
description
2013 nî lūn-bûn
@nan
2013 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մարտին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
The lipid mediator protectin D ...... and improves severe influenza
@ast
The lipid mediator protectin D ...... and improves severe influenza
@en
The lipid mediator protectin D ...... and improves severe influenza
@en-gb
The lipid mediator protectin D ...... and improves severe influenza
@nl
type
label
The lipid mediator protectin D ...... and improves severe influenza
@ast
The lipid mediator protectin D ...... and improves severe influenza
@en
The lipid mediator protectin D ...... and improves severe influenza
@en-gb
The lipid mediator protectin D ...... and improves severe influenza
@nl
prefLabel
The lipid mediator protectin D ...... and improves severe influenza
@ast
The lipid mediator protectin D ...... and improves severe influenza
@en
The lipid mediator protectin D ...... and improves severe influenza
@en-gb
The lipid mediator protectin D ...... and improves severe influenza
@nl
P2093
P50
P3181
P1433
P1476
The lipid mediator protectin D ...... and improves severe influenza
@en
P2093
Akihiko Ichikawa
Ayumi Kadowaki
Hiroki Nakanishi
Hiroyuki Arai
Jun Katahira
Keiji Kuba
Makoto Arita
Makoto Murakami
Masayuki Morita
Mizuho Nakayama
P304
P3181
P356
10.1016/J.CELL.2013.02.027
P407
P577
2013-03-28T00:00:00Z