Human cytomegalovirus infection causes degradation of Sp100 proteins that suppress viral gene expression - Test2 - elhamkhani - TriplyDB

Human cytomegalovirus infection causes degradation of Sp100 proteins that suppress viral gene expression

Human cytomegalovirus infection causes degradation of Sp100 proteins that suppress viral gene expression

http://www.wikidata.org/entity/Q24337028

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Analysis of human cytomegalovirus-encoded SUMO targets and temporal regulation of SUMOylation of the immediate-early proteins IE1 and IE2 during infection Intrinsic host restriction factors of human cytomegalovirus replication and mechanisms of viral escape Proteomic analysis of detergent resistant membrane domains during early interaction of macrophages with rough and smooth Brucella melitensis Inhibition of human cytomegalovirus immediate-early gene expression by cyclin A2-dependent kinase activity Quantitative temporal viromics: an approach to investigate host-pathogen interaction Biphasic regulation of A20 gene expression during human cytomegalovirus infection.The 19S proteasome activator promotes human cytomegalovirus immediate early gene expression through proteolytic and nonproteolytic mechanisms.Positive role of promyelocytic leukemia protein in type I interferon response and its regulation by human cytomegalovirus.Protein-Protein Interactions of Viroporins in Coronaviruses and Paramyxoviruses: New Targets for Antivirals?Contribution of the Major ND10 Proteins PML, hDaxx and Sp100 to the Regulation of Human Cytomegalovirus Latency and Lytic Replication in the Monocytic Cell Line THP-1.Herpesvirus saimiri antagonizes nuclear domain 10-instituted intrinsic immunity via an ORF3-mediated selective degradation of cellular protein Sp100.Interferon Kappa Inhibits Human Papillomavirus 31 Transcription by Inducing Sp100 Proteins Characterization of Recombinant Human Cytomegaloviruses Encoding IE1 Mutants L174P and 1-382 Reveals that Viral Targeting of PML Bodies Perturbs both Intrinsic and Innate Immune Responses The replication defect of ICP0-null mutant herpes simplex virus 1 can be largely complemented by the combined activities of human cytomegalovirus proteins IE1 and pp71 Critical role of presenilin-dependent γ-secretase activity in DNA damage-induced promyelocytic leukemia protein expression and apoptosis.Sp100A promotes chromatin decondensation at a cytomegalovirus-promoter-regulated transcription site Sp100 provides intrinsic immunity against human papillomavirus infection.The use of fluorescence microscopy to study the association between herpesviruses and intrinsic resistance factors.Recent advances in understanding viral evasion of type I interferon.Herpesviruses: interfering innate immunity by targeting viral sensing and interferon pathways.Disruption of host antiviral resistances by gammaherpesvirus tegument proteins with homology to the FGARAT purine biosynthesis enzyme Stimulation of the Replication of ICP0-Null Mutant Herpes Simplex Virus 1 and pp71-Deficient Human Cytomegalovirus by Epstein-Barr Virus Tegument Protein BNRF1.Viral FGARAT Homolog ORF75 of Rhesus Monkey Rhadinovirus Effects Proteasomal Degradation of the ND10 Components SP100 and PML.Analysis of the functional interchange between the IE1 and pp71 proteins of human cytomegalovirus and ICP0 of herpes simplex virus 1.Manipulation of ubiquitin/SUMO pathways in human herpesviruses infection.Sp100 isoform-specific regulation of human adenovirus 5 gene expression.Components of promyelocytic leukemia nuclear bodies (ND10) act cooperatively to repress herpesvirus infection.Human cytomegalovirus IE1 downregulates Hes1 in neural progenitor cells as a potential E3 ubiquitin ligase.Alterations of Nuclear Architecture and Epigenetic Signatures during African Swine Fever Virus Infection.MORC3, a Component of PML Nuclear Bodies, Has a Role in Restricting Herpes Simplex Virus 1 and Human Cytomegalovirus.CTCF binding to the first intron of the major immediate early (MIE) gene of human cytomegalovirus (HCMV) negatively regulates MIE gene expression and HCMV replication.Sp100A is a tumor suppressor that activates p53-dependent transcription and counteracts E1A/E1B-55K-mediated transformation.WDR5 Facilitates Human Cytomegalovirus Replication by Promoting Capsid Nuclear Egress.The chromatin remodeling factor SPOC1 acts as a cellular restriction factor against human cytomegalovirus by repressing the major immediate-early promoter.High-Definition Analysis of Host Protein Stability during Human Cytomegalovirus Infection Reveals Antiviral Factors and Viral Evasion Mechanisms

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P2860

Human cytomegalovirus infection causes degradation of Sp100 proteins that suppress viral gene expression

http://www.wikidata.org/entity/Q24337028

description

2011 nî lūn-bûn

@nan

2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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type

label

Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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prefLabel

Human cytomegalovirus infectio ...... suppress viral gene expression

@ast

Human cytomegalovirus infectio ...... suppress viral gene expression

@en

Human cytomegalovirus infectio ...... suppress viral gene expression

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Human cytomegalovirus infectio ...... suppress viral gene expression

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Journal of Virology

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Human cytomegalovirus infectio ...... suppress viral gene expression

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P2093

Chan Hee Lee

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Eui Tae Kim

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Hyang Sook Seol

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Hye Jin Shin

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Jin-Hyoung Lee

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Jin-Hyun Ahn

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Paul D Ling

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Su Yeon Gu

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Young-Eui Kim

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P2860

Splice variants of the nuclear dot-associated Sp100 protein contain homologies to HMG-1 and a human nuclear phosphoprotein-box motif

Common properties of nuclear body protein SP100 and TIF1alpha chromatin factor: role of SUMO modification

Sp100 interacts with ETS-1 and stimulates its transcriptional activity

PML contributes to a cellular mechanism of repression of herpes simplex virus type 1 infection that is inactivated by ICP0

Evidence for a role of the cellular ND10 protein PML in mediating intrinsic immunity against human cytomegalovirus infections

Differential role of Sp100 isoforms in interferon-mediated repression of herpes simplex virus type 1 immediate-early protein expression

SP100 expression modulates ETS1 transcriptional activity and inhibits cell invasion

Interaction of SP100 with HP1 proteins: a link between the promyelocytic leukemia-associated nuclear bodies and the chromatin compartment

LYSP100-associated nuclear domains (LANDs): description of a new class of subnuclear structures and their relationship to PML nuclear bodies

The mechanisms of PML-nuclear body formation

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11928-37

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scholarly article

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4043475

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10.1128/JVI.00758-11

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22

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85

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2011-11-01T00:00:00Z

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21880768

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Cytomegalovirus

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3209270

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