ZPR1 is essential for survival and is required for localization of the survival motor neurons (SMN) protein to Cajal bodies.
about
The zinc finger protein ZPR1 is a potential modifier of spinal muscular atrophy.Deficiency of the zinc finger protein ZPR1 causes defects in transcription and cell cycle progressionMolecular Mechanisms of Neurodegeneration in Spinal Muscular AtrophyStructural insights into the interaction of the evolutionarily conserved ZPR1 domain tandem with eukaryotic EF1A, receptors, and SMN complexesA short antisense oligonucleotide masking a unique intronic motif prevents skipping of a critical exon in spinal muscular atrophyDeficiency of the zinc finger protein ZPR1 causes neurodegenerationc-Jun NH2-terminal kinase is required for lineage-specific differentiation but not stem cell self-renewal.Reduced viability, fertility and fecundity in mice lacking the cajal body marker protein, coilin.The role of nuclear bodies in gene expression and disease.Drosophila Zpr1 (Zinc finger protein 1) is required downstream of both EGFR and FGFR signaling in tracheal subcellular lumen formationJNK regulates FoxO-dependent autophagy in neuronsGenetic inhibition of JNK3 ameliorates spinal muscular atrophy.ARS2 is a conserved eukaryotic gene essential for early mammalian development.SMN complex localizes to the sarcomeric Z-disc and is a proteolytic target of calpainPhosphorylation and the Cajal body: modification in search of function.Fam118B, a newly identified component of Cajal bodies, is required for Cajal body formation, snRNP biogenesis and cell viability.Spinal muscular atrophy and the antiapoptotic role of survival of motor neuron (SMN) protein.SMN - A chaperone for nuclear RNP social occasions?Solanum tuberosum ZPR1 encodes a light-regulated nuclear DNA-binding protein adjusting the circadian expression of StBBX24 to light cycle.UV-induced fragmentation of Cajal bodies.Deregulation of ZPR1 causes respiratory failure in spinal muscular atrophy.ZNF259 inhibits non-small cell lung cancer cells proliferation and invasion by FAK-AKT signaling.THEME 9IN VIVOEXPERIMENTAL MODELSZNF259 promotes breast cancer cells invasion and migration via ERK/GSK3β/snail signaling
P2860
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P2860
ZPR1 is essential for survival and is required for localization of the survival motor neurons (SMN) protein to Cajal bodies.
description
2005 nî lūn-bûn
@nan
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies
@nl
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@ast
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en-gb
type
label
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies
@nl
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@ast
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en-gb
prefLabel
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies
@nl
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@ast
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en-gb
P2093
P2860
P921
P3181
P1476
ZPR1 is essential for survival ...... (SMN) protein to Cajal bodies.
@en
P2093
Laxman Gangwani
Richard A Flavell
Roger J Davis
P2860
P304
P3181
P356
10.1128/MCB.25.7.2744-2756.2005
P407
P577
2005-04-01T00:00:00Z