Entry of parainfluenza virus into cells as a target for interrupting childhood respiratory disease
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Illumination of parainfluenza virus infection and transmission in living animals reveals a tissue-specific dichotomyInteraction between the Hemagglutinin-Neuraminidase and Fusion Glycoproteins of Human Parainfluenza Virus Type III Regulates Viral Growth In VivoThe catalytic mechanism of human parainfluenza virus type 3 haemagglutinin-neuraminidase revealedA general strategy to endow natural fusion-protein-derived peptides with potent antiviral activityInhibition of human parainfluenza virus type 3 infection by novel small moleculesHuman parainfluenza virus type 2 (HPIV2) induced host ADAM8 expression in human salivary adenocarcinoma cell line (HSY) during cell fusion.Residues in the heptad repeat a region of the fusion protein modulate the virulence of Sendai virus in miceThe impact of the butterfly effect on human parainfluenza virus haemagglutinin-neuraminidase inhibitor design.A recombinant sialidase fusion protein effectively inhibits human parainfluenza viral infection in vitro and in vivo.Paramyxovirus receptor-binding molecules: engagement of one site on the hemagglutinin-neuraminidase protein modulates activity at the second site.Circulating clinical strains of human parainfluenza virus reveal viral entry requirements for in vivo infection.Parainfluenza virus type 3 Ab in allogeneic hematopoietic cell transplant recipients: factors influencing post-transplant Ab titers and associated outcomesInhibition of hendra virus fusion.Premature activation of the paramyxovirus fusion protein before target cell attachment with corruption of the viral fusion machineryMechanism of fusion triggering by human parainfluenza virus type III: communication between viral glycoproteins during entryDAS181 treatment of hematopoietic stem cell transplant patients with parainfluenza virus lung disease requiring mechanical ventilationA second receptor binding site on human parainfluenza virus type 3 hemagglutinin-neuraminidase contributes to activation of the fusion mechanism.Detailed genetic analysis of hemagglutinin-neuraminidase glycoprotein gene in human parainfluenza virus type 1 isolates from patients with acute respiratory infection between 2002 and 2009 in Yamagata prefecture, JapanFusion promotion by a paramyxovirus hemagglutinin-neuraminidase protein: pH modulation of receptor avidity of binding sites I and IIAdaptation of human parainfluenza virus to airway epithelium reveals fusion properties required for growth in host tissue.Molecular determinants of antiviral potency of paramyxovirus entry inhibitors.Isolation & molecular characterization of human parainfluenza virus in Chennai, IndiaFeatures of Circulating Parainfluenza Virus Required for Growth in Human Airway.A dual drug regimen synergistically blocks human parainfluenza virus infection.Integrin alphavbeta1 promotes infection by human metapneumovirus.Human parainfluenza virus infection of the airway epithelium: viral hemagglutinin-neuraminidase regulates fusion protein activation and modulates infectivity.Identification of a region in the stalk domain of the nipah virus receptor binding protein that is critical for fusion activation.Development of oligomannose-coated liposome-based nasal vaccine against human parainfluenza virus type 3.Current management of parainfluenza pneumonitis in immunocompromised patients: a review.Structure-guided discovery of potent and dual-acting human parainfluenza virus haemagglutinin-neuraminidase inhibitors.Measles virus fusion machinery activated by sialic acid binding globular domain.Characterization of a key residue for hyperfusogenic phenotype in human parainfluenza virus type 2 (hPIV-2) fusion glycoprotein.'a'-Position-mutated and G4-mutated hemagglutinin-neuraminidase proteins of Newcastle disease virus impair fusion and hemagglutinin-neuraminidase-fusion interaction by different mechanisms.Regulation of paramyxovirus fusion activation: the hemagglutinin-neuraminidase protein stabilizes the fusion protein in a pretriggered state.Molecular characterization and phylogenetic analysis of human parainfluenza virus type 3 isolated from Saudi Arabia.The second receptor binding site of the globular head of the Newcastle disease virus hemagglutinin-neuraminidase activates the stalk of multiple paramyxovirus receptor binding proteins to trigger fusion.Spring-loaded model revisited: paramyxovirus fusion requires engagement of a receptor binding protein beyond initial triggering of the fusion protein.Glycoprotein interactions in paramyxovirus fusion.Characterizing the Cellular Immune Response to Parainfluenza Virus 3.Human parainfluenza virus-3 can be targeted by rapidly ex vivo expanded T lymphocytes.
P2860
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P2860
Entry of parainfluenza virus into cells as a target for interrupting childhood respiratory disease
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
name
Entry of parainfluenza virus i ...... childhood respiratory disease
@ast
Entry of parainfluenza virus i ...... childhood respiratory disease
@en
Entry of parainfluenza virus i ...... childhood respiratory disease
@nl
type
label
Entry of parainfluenza virus i ...... childhood respiratory disease
@ast
Entry of parainfluenza virus i ...... childhood respiratory disease
@en
Entry of parainfluenza virus i ...... childhood respiratory disease
@nl
prefLabel
Entry of parainfluenza virus i ...... childhood respiratory disease
@ast
Entry of parainfluenza virus i ...... childhood respiratory disease
@en
Entry of parainfluenza virus i ...... childhood respiratory disease
@nl
P2860
P3181
P356
P1476
Entry of parainfluenza virus i ...... childhood respiratory disease
@en
P2093
Anne Moscona
P2860
P304
P3181
P356
10.1172/JCI25669
P407
P577
2005-07-01T00:00:00Z