Rhamnolipids are virulence factors that promote early infiltration of primary human airway epithelia by Pseudomonas aeruginosa
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Cif is negatively regulated by the TetR family repressor CifRThe Pseudomonas aeruginosa secreted protein PA2934 decreases apical membrane expression of the cystic fibrosis transmembrane conductance regulatorRole of defensins in corneal epithelial barrier function against Pseudomonas aeruginosa traversalA streptococcal lipid toxin induces membrane permeabilization and pyroptosis leading to fetal injuryMexT regulates the type III secretion system through MexS and PtrC in Pseudomonas aeruginosaEffect of Human Burn Wound Exudate on Pseudomonas aeruginosa VirulenceMicrobial, host and xenobiotic diversity in the cystic fibrosis sputum metabolomeComparative metabolic systems analysis of pathogenic BurkholderiaA molecular mechanism that stabilizes cooperative secretions in Pseudomonas aeruginosa.Burkholderia thailandensis harbors two identical rhl gene clusters responsible for the biosynthesis of rhamnolipids.Anti-Biofilm and Antivirulence Activities of Metabolites from Plectosphaerella cucumerina against Pseudomonas aeruginosaBreaking barriers. New insights into airway epithelial barrier function in health and disease.The chemical arsenal of Burkholderia pseudomallei is essential for pathogenicity.Mimicking the host and its microenvironment in vitro for studying mucosal infections by Pseudomonas aeruginosa.High-resolution time series of Pseudomonas aeruginosa gene expression and rhamnolipid secretion through growth curve synchronization.Disruption of the endothelial barrier by proteases from the bacterial pathogen Pseudomonas aeruginosa: implication of matrilysis and receptor cleavageElevated paracellular glucose flux across cystic fibrosis airway epithelial monolayers is an important factor for Pseudomonas aeruginosa growthSmall molecule disruption of quorum sensing cross-regulation in pseudomonas aeruginosa causes major and unexpected alterations to virulence phenotypes.Direct interactions between ENaC gamma subunit and ClCN2 in cystic fibrosis epithelial cells.Quorum Sensing Controls Swarming Motility of Burkholderia glumae through Regulation of Rhamnolipids.Phenotypic characterization of clonal and nonclonal Pseudomonas aeruginosa strains isolated from lungs of adults with cystic fibrosis.The influence of human respiratory epithelia on Pseudomonas aeruginosa gene expression.Pseudomonas aeruginosa lipopolysaccharide: a major virulence factor, initiator of inflammation and target for effective immunity.Natural products as mediators of disease.Ribosome protection prevents azithromycin-mediated quorum-sensing modulation and stationary-phase killing of Pseudomonas aeruginosa.Individual matrix metalloproteinases control distinct transcriptional responses in airway epithelial cells infected with Pseudomonas aeruginosa.Fatty acid cosubstrates provide β-oxidation precursors for rhamnolipid biosynthesis in Pseudomonas aeruginosa, as evidenced by isotope tracing and gene expression assaysAcyltransferases in bacteria.A Novel Glycolipid Biosurfactant Confers Grazing Resistance upon Pantoea ananatis BRT175 against the Social Amoeba Dictyostelium discoideum.In vitro effects of anthocyanidins on sinonasal epithelial nitric oxide production and bacterial physiology.Modulation of Human Airway Barrier Functions during Burkholderia thailandensis and Francisella tularensis Infection Running Title: Airway Barrier Functions during Bacterial Infections.Cystic fibrosis transmembrane conductance regulator and caveolin-1 regulate epithelial cell internalization of Pseudomonas aeruginosa.Intercellular communication in bacteria.Chemical shift assignments of domain 4 from the phosphohexomutase from Pseudomonas aeruginosa suggest that freeing perturbs its coevolved domain interface.Rhamnolipids: diversity of structures, microbial origins and roles.Quorum sensing: implications on rhamnolipid biosurfactant production.Rhamnolipid biosurfactants as new players in animal and plant defense against microbes.Molecular approaches to pathogenesis study of Burkholderia cenocepacia, an important cystic fibrosis opportunistic bacterium.Rhamnolipids Enhance in Vivo Oral Bioavailability of Poorly Absorbed Molecules.On the Thermus thermophilus HB8 potential pathogenicity triggered from rhamnolipids secretion: morphological alterations and cytotoxicity induced on fibroblastic cell line.
P2860
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P2860
Rhamnolipids are virulence factors that promote early infiltration of primary human airway epithelia by Pseudomonas aeruginosa
description
2006 nî lūn-bûn
@nan
2006 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@ast
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@en
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@nl
type
label
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@ast
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@en
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@nl
prefLabel
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@ast
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@en
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@nl
P2093
P2860
P3181
P356
P1476
Rhamnolipids are virulence fac ...... elia by Pseudomonas aeruginosa
@en
P2093
Coralie Canard
Dorothée Caille
Jean-Silvain Lacroix
Laurence Zulianello
Paolo Meda
Thilo Köhler
P2860
P304
P3181
P356
10.1128/IAI.01772-05
P407
P577
2006-06-01T00:00:00Z