All three SOS-inducible DNA polymerases (Pol II, Pol IV and Pol V) are involved in induced mutagenesis
about
Inhibition of mutation and combating the evolution of antibiotic resistanceInteractions in the error-prone postreplication repair proteins hREV1, hREV3, and hREV7The processivity factor beta controls DNA polymerase IV traffic during spontaneous mutagenesis and translesion synthesis in vivoSOS-induced DNA polymerases enhance long-term survival and evolutionary fitnessReplication slippage involves DNA polymerase pausing and dissociation.Roles of DNA polymerases V and II in SOS-induced error-prone and error-free repair in Escherichia coliManaging DNA polymerases: coordinating DNA replication, DNA repair, and DNA recombinationAnswering the Call: Coping with DNA Damage at the Most Inopportune TimeAn SOS-regulated operon involved in damage-inducible mutagenesis in Caulobacter crescentusDispersal and regulation of an adaptive mutagenesis cassette in the bacteria domain.Quantitative proteomic view associated with resistance to clinically important antibiotics in Gram-positive bacteria: a systematic reviewPhysiology of the read-write genomeStructural basis for recruitment of translesion DNA polymerase Pol IV/DinB to the -clampStructural Insight into Translesion Synthesis by DNA Pol IIX-ray Crystal Structure of the Bacterial Conjugation Factor PsiB, a Negative Regulator of RecALesion bypass in yeast cells: Pol eta participates in a multi-DNA polymerase process.Mechanism of DNA polymerase II-mediated frameshift mutagenesisAntibiotic treatment enhances the genome-wide mutation rate of target cellsAn active site aromatic triad in Escherichia coli DNA Pol IV coordinates cell survival and mutagenesis in different DNA damaging agentsEvidence for a Rad18-independent frameshift mutagenesis pathway in human cell-free extractsA chemical genetics analysis of the roles of bypass polymerase DinB and DNA repair protein AlkB in processing N2-alkylguanine lesions in vivoTCA cycle-mediated generation of ROS is a key mediator for HeR-MRSA survival under β-lactam antibiotic exposureThe beta clamp targets DNA polymerase IV to DNA and strongly increases its processivityMutagenic nucleotide incorporation and hindered translocation by a food carcinogen C8-dG adduct in Sulfolobus solfataricus P2 DNA polymerase IV (Dpo4): modeling and dynamics studies.Metagenomic DNA fragments that affect Escherichia coli mutational pathways.Two-polymerase mechanisms dictate error-free and error-prone translesion DNA synthesis in mammalsThe molecular mechanisms and physiological consequences of oxidative stress: lessons from a model bacteriumRole of Escherichia coli DNA polymerase I in chromosomal DNA replication fidelity.Error-prone DNA polymerase IV is regulated by the heat shock chaperone GroE in Escherichia coliIn vivo evidence for a recA-independent recombination process in Escherichia coli that permits completion of replication of DNA containing UV damage in both strands.Genes regulated by the Escherichia coli SOS repressor LexA exhibit heterogeneous expressionCoordinating DNA polymerase traffic during high and low fidelity synthesis.Role of the DinB homologs Rv1537 and Rv3056 in Mycobacterium tuberculosisClusters of nucleotide substitutions and insertion/deletion mutations are associated with repeat sequencesSulfolobus solfataricus P2 DNA polymerase IV (Dpo4): an archaeal DinB-like DNA polymerase with lesion-bypass properties akin to eukaryotic poleta.Error-prone DNA polymerase IV is controlled by the stress-response sigma factor, RpoS, in Escherichia coli.Nucleotide excision repair or polymerase V-mediated lesion bypass can act to restore UV-arrested replication forks in Escherichia coli.Error-Prone Translesion DNA Synthesis by Escherichia coli DNA Polymerase IV (DinB) on Templates Containing 1,2-dihydro-2-oxoadenineUV hyper-resistance in Prochlorococcus MED4 results from a single base pair deletion just upstream of an operon encoding nudix hydrolase and photolyaseSyntheses and characterizations of the in vivo replicative bypass and mutagenic properties of the minor-groove O2-alkylthymidine lesions.
P2860
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P2860
All three SOS-inducible DNA polymerases (Pol II, Pol IV and Pol V) are involved in induced mutagenesis
description
2000 nî lūn-bûn
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2000 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
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2000 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2000年の論文
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All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@ast
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@en
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@nl
type
label
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@ast
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@en
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@nl
prefLabel
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@ast
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@en
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@nl
P2093
P2860
P3181
P356
P1433
P1476
All three SOS-inducible DNA po ...... nvolved in induced mutagenesis
@en
P2093
R Janel-Bintz
R Napolitano
P2860
P304
P3181
P356
10.1093/EMBOJ/19.22.6259
P407
P577
2000-11-15T00:00:00Z