The expression of advanced glycation endproduct receptors in rpe cells associated with basal deposits in human maculas
about
Glycation-altered proteolysis as a pathobiologic mechanism that links dietary glycemic index, aging, and age-related disease (in nondiabetics)Intraocular expression and release of high-mobility group box 1 protein in retinal detachmentMethylglyoxal induces cell death through endoplasmic reticulum stress-associated ROS production and mitochondrial dysfunction.CTGF is increased in basal deposits and regulates matrix production through the ERK (p42/p44mapk) MAPK and the p38 MAPK signaling pathways.Quantitative proteomics: comparison of the macular Bruch membrane/choroid complex from age-related macular degeneration and normal eyesSerum carboxymethyllysine, an advanced glycation end product, and age-related macular degeneration: the Age, Gene/Environment Susceptibility-Reykjavik StudyMultiplex analysis of age-related protein and lipid modifications in human Bruch's membraneRAGE regulates immune cell infiltration and angiogenesis in choroidal neovascularization.Lipids, lipoproteins, and age-related macular degenerationAdvanced glycation endproduct changes to Bruch's membrane promotes lipoprotein retention by lipoprotein lipaseProgress and perspectives on the role of RPE cell inflammatory responses in the development of age-related macular degeneration.A local complement response by RPE causes early-stage macular degeneration.How does the macula protect itself from oxidative stress?Characterization of the gelatinase system of the laminar human optic nerve, and surrounding annulus of Bruch's membrane, choroid, and sclera.Lipoprotein(A) with An Intact Lysine Binding Site Protects the Retina From an Age-Related Macular Degeneration Phenotype in Mice (An American Ophthalmological Society Thesis).Correlations between Photodegradation of Bisretinoid Constituents of Retina and Dicarbonyl Adduct DepositionComplement dysregulation in AMD: RPE-Bruch's membrane-choroidC-type natriuretic peptide protects the retinal pigment epithelium against advanced glycation end product-induced barrier dysfunction.RAGE mediated intracellular Aβ uptake contributes to the breakdown of tight junction in retinal pigment epithelium.Age-related changes in human macular Bruch's membrane as seen by quick-freeze/deep-etch.Relationship between RPE and choriocapillaris in age-related macular degenerationVascular endothelial growth factor in eye disease.Plasma protein pentosidine and carboxymethyllysine, biomarkers for age-related macular degenerationAging is not a disease: distinguishing age-related macular degeneration from aging.Hyperoside downregulates the receptor for advanced glycation end products (RAGE) and promotes proliferation in ECV304 cells via the c-Jun N-terminal kinases (JNK) pathway following stimulation by advanced glycation end-products in vitro.Parainflammation associated with advanced glycation endproduct stimulation of RPE in vitro: implications for age-related degenerative diseases of the eye.The pivotal role of the complement system in aging and age-related macular degeneration: hypothesis re-visited.Dietary hyperglycemia, glycemic index and metabolic retinal diseases.The pathogenic role of Maillard reaction in the aging eye.Lipofuscins prepared by modification of photoreceptor cells via glycation or lipid peroxidation show the similar phototoxicity.The influence of sublethal blue light exposure on human RPE cells.Oxidized low density lipoproteins induce a pathologic response by retinal pigmented epithelial cells.Receptor mediated disruption of retinal pigment epithelium function in acute glycated-albumin exposure.Proteomic profiling of human retinal pigment epithelium exposed to an advanced glycation-modified substrate.Nrf2 is a critical modulator of the innate immune response in a model of uveitis.The Association Between Variants of Receptor for Advanced Glycation End Products (RAGE) Gene Polymorphisms and Age-Related Macular Degeneration.Retinal Pigment Epithelial Cell Apoptosis is Influenced by a Combination of Macrophages and Soluble Mediators Present in Age-Related Macular Degeneration.The glycation of fibronectin by glycolaldehyde and methylglyoxal as a model for aging in Bruch's membrane.Perspective on AMD Pathobiology: A Bioenergetic Crisis in the RPE.
P2860
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P2860
The expression of advanced glycation endproduct receptors in rpe cells associated with basal deposits in human maculas
description
2006 nî lūn-bûn
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2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
The expression of advanced gly ...... asal deposits in human maculas
@ast
The expression of advanced gly ...... asal deposits in human maculas
@en
The expression of advanced gly ...... asal deposits in human maculas
@nl
type
label
The expression of advanced gly ...... asal deposits in human maculas
@ast
The expression of advanced gly ...... asal deposits in human maculas
@en
The expression of advanced gly ...... asal deposits in human maculas
@nl
prefLabel
The expression of advanced gly ...... asal deposits in human maculas
@ast
The expression of advanced gly ...... asal deposits in human maculas
@en
The expression of advanced gly ...... asal deposits in human maculas
@nl
P2093
P2860
P3181
P1476
The expression of advanced gly ...... asal deposits in human maculas
@en
P2093
Gerard A Lutty
Imran A Bhutto
James T Handa
Kazuki Ishibashi
Kazuko Ishibashi
Yuko Yamada
P2860
P3181
P356
10.1016/J.EXER.2005.10.005
P407
P577
2006-05-01T00:00:00Z