Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine - Test2 - elhamkhani - TriplyDB

Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine

Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine

http://www.wikidata.org/entity/Q24604466

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Recent Insights Into the Genetics of Inflammatory Bowel Disease Autophagy in immunity and inflammation Autophagy protects against active tuberculosis by suppressing bacterial burden and inflammation Our microbial selves: what ecology can teach us Mucosal Interactions between Genetics, Diet, and Microbiome in Inflammatory Bowel Disease The Gut Microbiota in Immune-Mediated Inflammatory Diseases The Mucosal Immune System and Its Regulation by Autophagy Paneth cell α-defensins and enteric microbiota in health and disease Nonproliferative and Proliferative Lesions of the Gastrointestinal Tract, Pancreas and Salivary Glands of the Rat and Mouse Mechanisms of Microbe-Host Interaction in Crohn's Disease: Dysbiosis vs. Pathobiont Selection The Intestinal Microbiota in Inflammatory Bowel Disease Gut inflammation and immunity: what is the role of the human gut virome?From genetics of inflammatory bowel disease towards mechanistic insights The virome in host health and disease Endoplasmic reticulum stress and inflammation Role of endoplasmic reticulum stress and autophagy as interlinking pathways in the pathogenesis of inflammatory bowel disease Role of viruses and bacteria-virus interactions in autoimmunity From systems to structure: bridging networks and mechanism Defining dysbiosis and its influence on host immunity and disease Murine models of inflammatory bowel disease (IBD): challenges of modeling human disease Environmental risk factors for inflammatory bowel diseases: a review The genetics of complex cholestatic disorders Analysis of the human gut microbiome and association with disease PAMPs and DAMPs: signal 0s that spur autophagy and immunity Advances in norovirus biology Effector and memory T cell responses to commensal bacteria Immunopathology of inflammatory bowel disease Norovirus regulation of the innate immune response and apoptosis occurs via the product of the alternative open reading frame 4 Genetically determined epithelial dysfunction and its consequences for microflora-host interactions The multifaceted roles of autophagy in tumors-implications for breast cancer.Effect of barrier microbes on organ-based inflammation.Inflammatory Bowel Disease Drugs: A Focus on Autophagy The Multibiome: The Intestinal Ecosystem's Influence on Immune Homeostasis, Health, and Disease A Multihit Model: Colitis Lessons from the Interleukin-10-deficient Mouse Norovirus immunology: Of mice and mechanisms Autophagy proteins control goblet cell function by potentiating reactive oxygen species production An image-based genetic assay identifies genes in T1D susceptibility loci controlling cellular antiviral immunity in mouse Metagenomic analysis of microbiome in colon tissue from subjects with inflammatory bowel diseases reveals interplay of viruses and bacteria The virome in mammalian physiology and disease Genetics and pathogenesis of inflammatory bowel disease

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Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine

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2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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Virus-plus-susceptibility gene ...... tg16L1 phenotypes in intestine

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P2860

Microbial translocation is a cause of systemic immune activation in chronic HIV infection

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

A physical and regulatory map of host-influenza interactions reveals pathways in H1N1 infection

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

Paneth cells directly sense gut commensals and maintain homeostasis at the intestinal host-microbial interface

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Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1beta production

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