NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease - Test2 - elhamkhani - TriplyDB

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

http://www.wikidata.org/entity/Q24608662

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Interactions between Autophagy and Inhibitory Cytokines Uptake and persistence of Mycobacterium avium subsp. paratuberculosis in human monocytes.Genetic and functional profiling of Crohn's disease: autophagy mechanism and susceptibility to infectious diseases Role of the gut microbiota in inflammatory bowel disease pathogenesis: what have we learnt in the past 10 years?Role of autophagy genetic variants for the risk of Candida infections.ATG16L1 and NOD2 polymorphisms enhance phagocytosis in monocytes of Crohn's disease patients.The Interplay between NLRs and Autophagy in Immunity and Inflammation.The role of autophagy in Crohn's disease.

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P2860

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

http://www.wikidata.org/entity/Q24608662

description

2011 nî lūn-bûn

@nan

2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

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2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

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NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

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NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

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type

label

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

@ast

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

@en

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

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prefLabel

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

@ast

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

@en

NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

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WJG.V17.I23.2829

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World Journal of Gastroenterology

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NOD2 and ATG16L1 polymorphisms affect monocyte responses in Crohn's disease

@en

P2093

Jacqui I Keenan

http://www.w3.org/2001/XMLSchema#string

Judy McKenzie

http://www.w3.org/2001/XMLSchema#string

Rebecca L Roberts

http://www.w3.org/2001/XMLSchema#string

Robert W Bentley

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P2860

High prevalence of adherent-invasive Escherichia coli associated with ileal mucosa in Crohn’s disease

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis

Human IRGM induces autophagy to eliminate intracellular mycobacteria

A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1

Phenotypic and functional features of human Th17 cells

Impaired autophagy of an intracellular pathogen induced by a Crohn's disease associated ATG16L1 variant.

Concordance of inflammatory bowel disease among Danish twins. Results of a nationwide study.

Clinical patterns of familial inflammatory bowel disease

Increased production of tumour necrosis factor-alpha interleukin-1 beta, and interleukin-6 by morphologically normal intestinal biopsies from patients with Crohn's disease.

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2829-37

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scholarly article

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983230

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10.3748/WJG.V17.I23.2829

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23

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17

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John F. Pearson

Richard B. Gearry

Murray L. Barclay

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2011-06-21T00:00:00Z

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51472688

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21734790

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P921

Crohn's disease

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3120942

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