Prenatal exposure to the environmental obesogen tributyltin predisposes multipotent stem cells to become adipocytes - Test2 - elhamkhani - TriplyDB

Prenatal exposure to the environmental obesogen tributyltin predisposes multipotent stem cells to become adipocytes

Prenatal exposure to the environmental obesogen tributyltin predisposes multipotent stem cells to become adipocytes

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The retinoid X receptors and their ligands Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses The Role of Epigenetics in the Latent Effects of Early Life Exposure to Obesogenic Endocrine Disrupting Chemicals The epigenetic lorax: gene-environment interactions in human health Developmental origins of obesity and type 2 diabetes: molecular aspects and role of chemicals Do interactions between gut ecology and environmental chemicals contribute to obesity and diabetes?Predicting later-life outcomes of early-life exposures Endocrine-disrupting Chemicals: Review of Toxicological Mechanisms Using Molecular Pathway Analysis Role of environmental chemicals in diabetes and obesity: a National Toxicology Program workshop review Endocrine disrupting chemicals and the developmental programming of adipogenesis and obesity EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Adverse effects in adulthood resulting from low-level dioxin exposure in juvenile zebrafish Transgenerational effects of obesogens and the obesity epidemic The paradox of progress: environmental disruption of metabolism and the diabetes epidemic Effects of long-term endocrine disrupting compound exposure on Macaca mulatta embryonic stem cells Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: focus on the cancer hallmark of tumor angiogenesis The Mammalian "Obesogen" Tributyltin Targets Hepatic Triglyceride Accumulation and the Transcriptional Regulation of Lipid Metabolism in the Liver and Brain of Zebrafish Editor's Highlight: Screening ToxCast Prioritized Chemicals for PPARG Function in a Human Adipose-Derived Stem Cell Model of Adipogenesis Prioritizing Environmental Chemicals for Obesity and Diabetes Outcomes Research: A Screening Approach Using ToxCast™ High-Throughput Data Metabolism Disrupting Chemicals and Metabolic Disorders Associating cellular epigenetic models with human phenotypes Effects of the environmental contaminants DEHP and TCDD on estradiol synthesis and aryl hydrocarbon receptor and peroxisome proliferator-activated receptor signalling in the human granulosa cell line KGN.Obesity and diabetes: from genetics to epigenetics.Effects of Crude Oil/Dispersant Mixture and Dispersant Components on PPARγ Activity in Vitro and in Vivo: Identification of Dioctyl Sodium Sulfosuccinate (DOSS; CAS #577-11-7) as a Probable Obesogen.Quantification of total and visceral adipose tissue in fructose-fed rats using water-fat separated single echo MRI.Chlorinated persistent organic pollutants, obesity, and type 2 diabetes Altered Adipogenesis in Zebrafish Larvae Following High Fat Diet and Chemical Exposure Is Visualised by Stimulated Raman Scattering Microscopy Halogenated bisphenol-A analogs act as obesogens in zebrafish larvae (Danio rerio).Association of placenta organotin concentrations with growth and ponderal index in 110 newborn boys from Finland during the first 18 months of life: a cohort study What Are We Putting in Our Food That Is Making Us Fat? Food Additives, Contaminants, and Other Putative Contributors to Obesity.Maternal obesity and fetal metabolic programming: a fertile epigenetic soil.Transgenerational inheritance of prenatal obesogen exposure.Childhood obesity and environmental chemicals.Molecular analysis of endocrine disruption in hornyhead turbot at wastewater outfalls in southern california using a second generation multi-species microarray Minireview: PPARγ as the target of obesogens.Prenatal stress and developmental programming of human health and disease risk: concepts and integration of empirical findings Zebrafish obesogenic test: a tool for screening molecules that target adiposity Organotins are potent activators of PPARγ and adipocyte differentiation in bone marrow multipotent mesenchymal stromal cells Current methods of adipogenic differentiation of mesenchymal stem cells Structurally-diverse, PPARγ-activating environmental toxicants induce adipogenesis and suppress osteogenesis in bone marrow mesenchymal stromal cells.

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Prenatal exposure to the environmental obesogen tributyltin predisposes multipotent stem cells to become adipocytes

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2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Prenatal exposure to the envir ...... tem cells to become adipocytes

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Molecular Endocrinology

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Connie Chow

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Stephanie Casey

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Séverine Kirchner

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Tiffany Kieu

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A new mathematical model for relative quantification in real-time RT-PCR

Perturbed nuclear receptor signaling by environmental obesogens as emerging factors in the obesity crisis

Environmental obesogens: organotins and endocrine disruption via nuclear receptor signaling

Daily intakes of tributyltin and triphenyltin compounds from meals

Organotin compounds promote adipocyte differentiation as agonists of the peroxisome proliferator-activated receptor gamma/retinoid X receptor pathway

Endocrine-disrupting organotin compounds are potent inducers of adipogenesis in vertebrates

Dissecting direct reprogramming through integrative genomic analysis

Adipocyte differentiation from the inside out

Concise review: mesenchymal stem cells: their phenotype, differentiation capacity, immunological features, and potential for homing.

Subcutaneous fat in normal and diseased states 3. Adipogenesis: from stem cell to fat cell.

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