Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice - Test2 - elhamkhani - TriplyDB

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

http://www.wikidata.org/entity/Q24623247

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Inhibition of microRNA function by antimiR oligonucleotides MicroRNAs in NF-kappaB signaling MicroRNAs in Valvular Heart Diseases: Potential Role as Markers and Actors of Valvular and Cardiac Remodeling Interplay between cardiac function and heart development Epigenetic associations in relation to cardiovascular prevention and therapeutics MicroRNAs and the resolution phase of inflammation in macrophages The function of miRNA in cardiac hypertrophy MicroRNAs in the Stressed Heart: Sorting the Signal from the Noise Non-Coding RNAs Including miRNAs and lncRNAs in Cardiovascular Biology and Disease Role of microRNAs in the reperfused myocardium towards post-infarct remodelling MicroRNAs as therapeutic targets and biomarkers of cardiovascular disease MicroRNAs in heart failure: Small molecules with major impact MicroRNAs as potential therapeutic targets in kidney disease MicroRNAs in stress signaling and human disease Differential expression of microRNAs in different disease states.Non-coding RNAs: the "dark matter" of cardiovascular pathophysiology.Using microRNA as an alternative treatment for hyperlipidemia and cardiovascular disease: cardio-miRs in the pipeline MicroRNAs in idiopathic pulmonary fibrosis: involvement in pathogenesis and potential use in diagnosis and therapeutics The fibrosis-cell death axis in heart failure Epigenetic mechanisms in heart development and disease Turning 21: Induction of miR-21 as a Key Switch in the Inflammatory Response MicroRNA-34a regulates cardiac fibrosis after myocardial infarction by targeting Smad4 Implication of microRNAs in atrial natriuretic peptide and nitric oxide signaling in vascular smooth muscle cells miR-21 promotes fibrogenic epithelial-to-mesenchymal transition of epicardial mesothelial cells involving Programmed Cell Death 4 and Sprouty-1 Induction of specific microRNAs inhibits cutaneous wound healing Mechanisms of fibrosis: therapeutic translation for fibrotic disease Haemodynamically dependent valvulogenesis of zebrafish heart is mediated by flow-dependent expression of miR-21 miR-21 is associated with fibrosis and right ventricular failure miR-24-mediated knockdown of H2AX damages mitochondria and the insulin signaling pathway.Emerging Roles of microRNAs in Ischemic Stroke: As Possible Therapeutic Agents Non-coding RNAs in cardiac hypertrophy.Induction of microRNA-21 with exogenous hydrogen sulfide attenuates myocardial ischemic and inflammatory injury in mice MicroRNAs are potential therapeutic targets in fibrosing kidney disease: lessons from animal models.Adult-specific functions of animal microRNAs.Down-regulation of miR-21 biogenesis by estrogen action contributes to osteoclastic apoptosis IL-10R blockade during chronic schistosomiasis mansoni results in the loss of B cells from the liver and the development of severe pulmonary disease.Deep sequencing reveals predominant expression of miR-21 amongst the small non-coding RNAs in retinal microvascular endothelial cells.MicroRNA 10a marks regulatory T cells.BAF60 A, B, and Cs of muscle determination and renewal.The myriad essential roles of microRNAs in cardiovascular homeostasis and disease.

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Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

http://www.wikidata.org/entity/Q24623247

description

2010 nî lūn-bûn

@nan

2010 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

@yue

2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@ast

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@en

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@nl

type

label

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@ast

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@en

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@nl

prefLabel

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@ast

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@en

Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

@nl

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Journal of Clinical Investigation

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Stress-dependent cardiac remodeling occurs in the absence of microRNA-21 in mice

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P2093

Eric N Olson

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Eva van Rooij

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Joseph A Hill

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Rusty L Montgomery

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Sakari Kauppinen

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Susanna Obad

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Xiaoxia Qi

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P2860

miR-21 Gene expression triggered by AP-1 is sustained through a double-negative feedback mechanism

MicroRNAs miR-143 and miR-145 modulate cytoskeletal dynamics and responsiveness of smooth muscle cells to injury

MicroRNA expression signature and the role of microRNA-21 in the early phase of acute myocardial infarction

A signature pattern of stress-responsive microRNAs that can evoke cardiac hypertrophy and heart failure

Expression of microRNAs is dynamically regulated during cardiomyocyte hypertrophy

A calcineurin-dependent transcriptional pathway for cardiac hypertrophy

MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

Control of stress-dependent cardiac growth and gene expression by a microRNA

Therapeutic silencing of microRNA-122 in primates with chronic hepatitis C virus infection

LNA-mediated microRNA silencing in non-human primates

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3912-6

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3963883

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10.1172/JCI43604

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11

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120

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David M Patrick

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2010-11-01T00:00:00Z

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20978354

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2964990

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