Hematopoietic origin of pathological grooming in Hoxb8 mutant mice - Test2 - elhamkhani - TriplyDB

Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

http://www.wikidata.org/entity/Q24624921

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Neuroinflammation and psychiatric illness The role of immune dysfunction in the pathophysiology of autism Autonomic-Immune-Vascular Interaction: An Emerging Concept for Neurogenic Hypertension A New Outlook on Mental Illnesses: Glial Involvement Beyond the Glue The "quad-partite" synapse: microglia-synapse interactions in the developing and mature CNS The significance of meaning: why do over 90% of behavioral neuroscience results fail to translate to humans, and what can we do to fix it?Building blocks of the cerebral cortex: from development to the dish Bidirectional microglia-neuron communication in the healthy brain Converging Pathways in Autism Spectrum Disorders: Interplay between Synaptic Dysfunction and Immune Responses Microglia development and function Microglia contact induces synapse formation in developing somatosensory cortex.Neuron-Microglia Interactions in Mental Health Disorders: "For Better, and For Worse"Neuroimmune mechanisms of depression Microglia sculpt postnatal neural circuits in an activity and complement-dependent manner Animal models of neuropsychiatric disorders Function and Dysfunction of Microglia during Brain Development: Consequences for Synapses and Neural Circuits Interactions of innate and adaptive immunity in brain development and function.Using mice to model Obsessive Compulsive Disorder: From genes to circuits.Microglia: Dynamic Mediators of Synapse Development and Plasticity.Imaging activity in neurons and glia with a Polr2a-based and cre-dependent GCaMP5G-IRES-tdTomato reporter mouse.Wild-type microglia arrest pathology in a mouse model of Rett syndrome.Structured evaluation of rodent behavioral tests used in drug discovery research.Microglia - the brain's busy bees.Modeling an autism risk factor in mice leads to permanent immune dysregulation.The Long and the Short of it: Gene and Environment Interactions During Early Cortical Development and Consequences for Long-Term Neurological Disease.Annual Research Review: Transgenic mouse models of childhood-onset psychiatric disorders.The absence of P2X7 receptors (P2rx7) on non-haematopoietic cells leads to selective alteration in mood-related behaviour with dysregulated gene expression and stress reactivity in mice.Repeated cortico-striatal stimulation generates persistent OCD-like behavior.Deep-brain imaging via epi-fluorescence Computational Cannula Microscopy Microglia promote learning-dependent synapse formation through brain-derived neurotrophic factor.Microglial NFκB-TNFα hyperactivation induces obsessive-compulsive behavior in mouse models of progranulin-deficient frontotemporal dementia.Amplification and propagation of interleukin-1β signaling by murine brain endothelial and glial cells.Potential translational targets revealed by linking mouse grooming behavioral phenotypes to gene expression using public databases.Efficient reprogramming of adult neural stem cells to monocytes by ectopic expression of a single gene Microglial dysregulation in psychiatric disease Microglia: unique and common features with other tissue macrophages.Modeling complex neuropsychiatric disease with induced pluripotent stem cells Nicotinic receptor alpha7 expression identifies a novel hematopoietic progenitor lineage.Slitrks as emerging candidate genes involved in neuropsychiatric disorders In vivo imaging of translocator protein, a marker of activated microglia, in alcohol dependence

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Hematopoietic origin of pathological grooming in Hoxb8 mutant mice

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Erik Peden

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Gerald Spangrude

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Mario R Capecchi

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Sen Wu

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The immune response in autism: a new frontier for autism research

Common variants conferring risk of schizophrenia

Common variants on chromosome 6p22.1 are associated with schizophrenia

Cortico-striatal synaptic defects and OCD-like behaviours in Sapap3-mutant mice.

Generalized lacZ expression with the ROSA26 Cre reporter strain

Common polygenic variation contributes to risk of schizophrenia and bipolar disorder

Schizophrenia, autoimmunity and immune system dysregulation: a comprehensive model updated and revisited

MicroRNA-196 inhibits HOXB8 expression in myeloid differentiation of HL60 cells

Hoxb8 is required for normal grooming behavior in mice

Loss of Hoxb8 alters spinal dorsal laminae and sensory responses in mice

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