Interferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155* - Test2 - elhamkhani - TriplyDB

Interferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155*

Interferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155*

http://www.wikidata.org/entity/Q24627698

about

Aberrant expression of interleukin-1β and inflammasome activation in human malignant gliomas Estrogen replacement therapy-induced neuroprotection against brain ischemia-reperfusion injury involves the activation of astrocytes via estrogen receptor β.Decreased levels of miR-224 and the passenger strand of miR-221 increase MBD2, suppressing maspin and promoting colorectal tumor growth and metastasis in mice In vivo inhibition of miR-155 significantly alters post-stroke inflammatory response LPS and IL-1 differentially activate mouse and human astrocytes: role of CD14 Human metapneumovirus infection induces significant changes in small noncoding RNA expression in airway epithelial cells.Expression, regulation and function of microRNAs in multiple sclerosis A Refined Bead-Free Method to Identify Astrocytic Exosomes in Primary Glial Cultures and Blood Plasma Micro RNA detection in long-term fixed tissue of cortical glutamatergic pyramidal neurons after targeted laser-capture neuroanatomical microdissection miR-155 as a multiple sclerosis-relevant regulator of myeloid cell polarization.Heterogeneity of oligodendrocyte progenitor cells in adult human brain Differential cytokine expression by brain microglia/macrophages in primary culture after oxygen glucose deprivation and their protective effects on astrocytes during anoxia.Differential expression and clinical significance of three inflammation-related microRNAs in gangliogliomas MicroRNA Expression Patterns in Human Astrocytes in Relation to Anatomical Location and Age.Loss of interleukin receptor-associated kinase 4 signaling suppresses amyloid pathology and alters microglial phenotype in a mouse model of Alzheimer's disease.The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.MicroRNA dysregulation in multiple sclerosis.Antisense oligonucleotide treatment enhances the recovery of acute lung injury through IL-10-secreting M2-like macrophage-induced expansion of CD4+ regulatory T cells.Synergistic control of herpes simplex virus pathogenesis by IRF-3, and IRF-7 revealed through non-invasive bioluminescence imaging.miR-155 Deletion in Mice Overcomes Neuron-Intrinsic and Neuron-Extrinsic Barriers to Spinal Cord Repair.Pathways to neurodegeneration: mechanistic insights from GWAS in Alzheimer's disease, Parkinson's disease, and related disorders Epigenetics and the modulation of neuroinflammation.MicroRNA 155 regulates Japanese encephalitis virus-induced inflammatory response by targeting Src homology 2-containing inositol phosphatase 1.Regulation of microglial activation in stroke.Anti-viral and anti-inflammatory mechanisms of the innate immune transcription factor interferon regulatory factor 3: relevance to human CNS diseases.Modulation of macrophage activation and programming in immunity.MicroRNAs in the brain: it's regulatory role in neuroinflammation.The role of inflammatory and oxidative stress mechanisms in the pathogenesis of Parkinson's disease: focus on astrocytes.MicroRNAs as novel regulators of neuroinflammation.The use of microRNAs to modulate redox and immune response to stroke.Regulation of miRNA strand selection: follow the leader?MicroRNAs: Roles in Regulating Neuroinflammation.Roles of microglia in brain development, tissue maintenance and repair.Early miR-155 upregulation contributes to neuroinflammation in Alzheimer's disease triple transgenic mouse model.Interferon regulatory factor 3 alters glioma inflammatory and invasive properties.Interferon regulatory factor 3 plays an anti-inflammatory role in microglia by activating the PI3K/Akt pathway.Suppression of microRNA-155 attenuates neuropathic pain by regulating SOCS1 signalling pathway.MiRNAs in Astrocyte-Derived Exosomes as Possible Mediators of Neuronal Plasticity.Overexpressed miRNA-155 dysregulates intestinal epithelial apical junctional complex in severe acute pancreatitis.Effects of fumarates on inflammatory human astrocyte responses and oligodendrocyte differentiation.

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Interferon regulatory factor 3 inhibits astrocyte inflammatory gene expression through suppression of the proinflammatory miR-155 and miR-155*

http://www.wikidata.org/entity/Q24627698

description

2011 nî lūn-bûn

@nan

2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

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2011年論文

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2011年论文

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name

Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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type

label

Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

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Interferon regulatory factor 3 ...... lammatory miR-155 and miR-155*

@en

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Avital Bauman

http://www.w3.org/2001/XMLSchema#string

Bridget Shafit-Zagardo

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Hyeon-Sook Suh

http://www.w3.org/2001/XMLSchema#string

Leonid Tarassishin

http://www.w3.org/2001/XMLSchema#string

Olivier Loudig

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Sunhee C Lee

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P2860

The RIN: an RNA integrity number for assigning integrity values to RNA measurements

Virus-dependent phosphorylation of the IRF-3 transcription factor regulates nuclear translocation, transactivation potential, and proteasome-mediated degradation

Widespread regulatory activity of vertebrate microRNA* species

MicroRNA-155 promotes autoimmune inflammation by enhancing inflammatory T cell development

The kinase Akt1 controls macrophage response to lipopolysaccharide by regulating microRNAs

Inositol phosphatase SHIP1 is a primary target of miR-155

microRNAs and the immune response

NF-kappaB-dependent induction of microRNA miR-146, an inhibitor targeted to signaling proteins of innate immune responses

MicroRNA-155 is induced during the macrophage inflammatory response

Alternative activation of macrophages

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1911-22

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10.1002/GLIA.21233

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12

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22170100

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