Cellular mechanisms by which proinsulin C-peptide prevents insulin-induced neointima formation in human saphenous vein
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Molecular Pathways Regulating Macrovascular Pathology and Vascular Smooth Muscle Cells Phenotype in Type 2 DiabetesPhysiological effects and therapeutic potential of proinsulin C-peptideAberrant phenotype in human endothelial cells of diabetic origin: implications for saphenous vein graft failure?FSP-1 Impairs the Function of Endothelium Leading to Failure of Arteriovenous Grafts in Diabetic MiceThe clinical potential of C-peptide replacement in type 1 diabetes.Factors associated with diabetic retinopathy in chinese patients with type 2 diabetes mellitus.C-Peptide and vascular complications in type 2 diabetic subjects.C-peptide prevents hyperglycemia-induced endothelial apoptosis through inhibition of reactive oxygen species-mediated transglutaminase 2 activationC-peptide levels are associated with mortality and cardiovascular mortality in patients undergoing angiography: the LURIC study.C-peptide activates AMPKα and prevents ROS-mediated mitochondrial fission and endothelial apoptosis in diabetes.C-peptide promotes lesion development in a mouse model of arteriosclerosisWhat's new in diabetes: incretins and C-peptide.C-peptide: a new mediator of atherosclerosis in diabetesProinflammatory effects of C-Peptide in different tissues.Can C-peptide mediated anti-inflammatory effects retard the development of microvascular complications of type 1 diabetes?The vascular smooth muscle cell: a therapeutic target in Type 2 diabetes?C-Peptide and its career from innocent bystander to active player in diabetic atherogenesis.Vascular Smooth Muscle as a Target for Novel Therapeutics.C-peptide antioxidant adaptive pathways in β cells and diabetes.Autocrine C-peptide mechanism underlying INS1 beta cell adaptation to oxidative stress.Association between serum C-peptide levels and chronic microvascular complications in Korean type 2 diabetic patients.Proinsulin C-peptide activates α-enolase: implications for C-peptide--cell membrane interaction.Prevention of VEGF-mediated microvascular permeability by C-peptide in diabetic mice.C-peptide reduces high-glucose-induced apoptosis of endothelial cells and decreases NAD(P)H-oxidase reactive oxygen species generation in human aortic endothelial cells.
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P2860
Cellular mechanisms by which proinsulin C-peptide prevents insulin-induced neointima formation in human saphenous vein
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2010 nî lūn-bûn
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2010 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2010年の論文
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2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
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name
Cellular mechanisms by which p ...... mation in human saphenous vein
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Cellular mechanisms by which p ...... mation in human saphenous vein
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Cellular mechanisms by which p ...... mation in human saphenous vein
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Cellular mechanisms by which p ...... mation in human saphenous vein
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Cellular mechanisms by which p ...... mation in human saphenous vein
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Cellular mechanisms by which p ...... mation in human saphenous vein
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Cellular mechanisms by which p ...... mation in human saphenous vein
@en-gb
Cellular mechanisms by which p ...... mation in human saphenous vein
@nl
prefLabel
Cellular mechanisms by which p ...... mation in human saphenous vein
@ast
Cellular mechanisms by which p ...... mation in human saphenous vein
@en
Cellular mechanisms by which p ...... mation in human saphenous vein
@en-gb
Cellular mechanisms by which p ...... mation in human saphenous vein
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P2860
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2-s2.0-77955518344
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Cellular mechanisms by which p ...... mation in human saphenous vein
@en
P2093
P2860
P2888
P304
P356
10.1007/S00125-010-1736-6
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P5530
P577
2010-05-12T00:00:00Z
P5875
P6179
1043957943