Decreased glutathione accelerates neurological deficit and mitochondrial pathology in familial ALS-linked hSOD1(G93A) mice model
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Oxidative Stress in Neurodegenerative DiseasesDysregulation of glutathione homeostasis in neurodegenerative diseasesMitochondrial dysfunction in amyotrophic lateral sclerosis - a valid pharmacological target?Oxidative stress and mitochondrial damage: importance in non-SOD1 ALSThe omega-3 fatty acid eicosapentaenoic acid accelerates disease progression in a model of amyotrophic lateral sclerosisNew Therapeutics to Modulate Mitochondrial Function in Neurodegenerative Disorders.Hepatocyte circadian clock controls acetaminophen bioactivation through NADPH-cytochrome P450 oxidoreductase.Mitochondria-targeted catalase reverts the neurotoxicity of hSOD1G⁹³A astrocytes without extending the survival of ALS-linked mutant hSOD1 miceA Cystine-Rich Whey Supplement (Immunocal(®)) Delays Disease Onset and Prevents Spinal Cord Glutathione Depletion in the hSOD1(G93A) Mouse Model of Amyotrophic Lateral SclerosisGlutathione supplementation attenuates lipopolysaccharide-induced mitochondrial dysfunction and apoptosis in a mouse model of acute lung injury.Recent advances in the treatment of neurodegenerative diseases based on GSH delivery systems.Beneficial effects of Nrf2 overexpression in a mouse model of Alexander disease.Changes in Protein Expression and Lysine Acetylation Induced by Decreased Glutathione Levels in AstrocytesMitochondrial glutathione transport is a key determinant of neuronal susceptibility to oxidative and nitrosative stress.Astroglia acquires a toxic neuroinflammatory role in response to the cerebrospinal fluid from amyotrophic lateral sclerosis patientsAntioxidant gene therapy against neuronal cell death.Blood-spinal cord barrier disruption contributes to early motor-neuron degeneration in ALS-model mice.The modest impact of transcription factor Nrf2 on the course of disease in an ALS animal model.Role and Therapeutic Potential of Astrocytes in Amyotrophic Lateral Sclerosis.Decreased glutathione levels cause overt motor neuron degeneration in hSOD1WT over-expressing mice.
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P2860
Decreased glutathione accelerates neurological deficit and mitochondrial pathology in familial ALS-linked hSOD1(G93A) mice model
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2011 nî lūn-bûn
@nan
2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
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2011年論文
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2011年論文
@zh-hant
2011年論文
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2011年論文
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2011年論文
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2011年论文
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name
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@ast
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@en
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
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type
label
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@ast
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@en
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@nl
prefLabel
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@ast
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@en
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@nl
P2093
P2860
P3181
P1476
Decreased glutathione accelera ...... -linked hSOD1(G93A) mice model
@en
P2093
Delinda A Johnson
Jeffrey A Johnson
Marcelo R Vargas
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P304
P3181
P356
10.1016/J.NBD.2011.04.025
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P577
2011-09-01T00:00:00Z