Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response - Test2 - elhamkhani - TriplyDB

Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response

Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response

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Perinatal bisphenol A exposure promotes dose-dependent alterations of the mouse methylome LINE-1 hypomethylation in spermatozoa is associated with Bisphenol A exposure Disruption of adult expression of sexually selected traits by developmental exposure to bisphenol A.Biological Tools to Study the Effects of Environmental Contaminants at the Feto-Maternal Interface Preimplantation Exposure to Bisphenol A and Triclosan May Lead to Implantation Failure in Humans Bisphenol-A and Female Infertility: A Possible Role of Gene-Environment Interactions Epigenetic epidemiology: promises for public health research Early-life Exposure to Endocrine Disrupting Chemicals and Later-life Health Outcomes: An Epigenetic Bridge?Epigenetic effects of environmental chemicals bisphenol A and phthalates Bisphenol A and the female reproductive tract: an overview of recent laboratory evidence and epidemiological studies Developmental programming mediated by complementary roles of imprinted Grb10 in mother and pup Bisphenol A exposure alters developmental gene expression in the fetal rhesus macaque uterus Epigenetic Regulation of Non-Lymphoid Cells by Bisphenol A, a Model Endocrine Disrupter: Potential Implications for Immunoregulation EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals DNA methylation changes in whole blood is associated with exposure to the environmental contaminants, mercury, lead, cadmium and bisphenol A, in women undergoing ovarian stimulation for IVF Global DNA methylation loss associated with mercury contamination and aging in the American alligator (Alligator mississippiensis)Environmental epigenetics and its implication on disease risk and health outcomes Evidence for bisphenol A-induced female infertility: a review (2007-2016)Bisphenol a and reproductive health: update of experimental and human evidence, 2007-2013 Using mouse models of autism spectrum disorders to study the neurotoxicology of gene-environment interactions Chronic Exposure to Bisphenol A Affects Uterine Function During Early Pregnancy in Mice Environmental chemical exposures and human epigenetics Environmental factors, epigenetics, and developmental origin of reproductive disorders Bisphenol a exposure disrupts genomic imprinting in the mouse Neonatal exposure to estradiol/bisphenol A alters promoter methylation and expression of Nsbp1 and Hpcal1 genes and transcriptional programs of Dnmt3a/b and Mbd2/4 in the rat prostate gland throughout life Chronic exposure to bisphenol a impairs progesterone receptor-mediated signaling in the uterus during early pregnancy The role of Bisphenol A in shaping the brain, epigenome and behavior.High content imaging-based assay to classify estrogen receptor-α ligands based on defined mechanistic outcomes.Bisphenol-A and diethylstilbestrol exposure induces the expression of breast cancer associated long noncoding RNA HOTAIR in vitro and in vivo.Early Life Origins of Metabolic Syndrome: The Role of Environmental Toxicants Maternal Methyl Donor Supplementation during Gestation Counteracts the Bisphenol A-Induced Impairment of Intestinal Morphology, Disaccharidase Activity, and Nutrient Transporters Gene Expression in Newborn and Weaning Pigs.Prenatal exposure to BPA alters the epigenome of the rat mammary gland and increases the propensity to neoplastic development Epigenomic disruption: the effects of early developmental exposures.Environmental toxicants and the developing immune system: a missing link in the global battle against infectious disease?Bisphenol A (BPA) and its potential role in the pathogenesis of the polycystic ovary syndrome (PCOS).Environmental induction of the fetal epigenome.In utero bisphenol A concentration, metabolism, and global DNA methylation across matched placenta, kidney, and liver in the human fetus Cloning of multiple ERα mRNA variants in killifish (Fundulus heteroclitus), and differential expression by tissue type, stage of reproduction, and estrogen exposure in fish from polluted and unpolluted environments.In utero exposure to diethylstilbestrol (DES) or bisphenol-A (BPA) increases EZH2 expression in the mammary gland: an epigenetic mechanism linking endocrine disruptors to breast cancer Bisphenol-A exposure alters endometrial progesterone receptor expression in the nonhuman primate.

P2860

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P2860

Bisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen response

http://www.wikidata.org/entity/Q24632509

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Bisphenol-A exposure in utero ...... g of uterine estrogen response

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Hugh S Taylor

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Jason G Bromer

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Leo Doherty

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Melissa B Taylor

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Yuping Zhou

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P2860

Urinary concentrations of bisphenol A and 4-nonylphenol in a human reference population

Developmental effects of endocrine-disrupting chemicals in wildlife and humans

HOXA10 is expressed in response to sex steroids at the time of implantation in the human endometrium

Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development

Disposition of orally administered 2,2-Bis(4-hydroxyphenyl)propane (Bisphenol A) in pregnant rats and the placental transfer to fetuses

The power and the promise of DNA methylation markers

Implantation in the human: the role of HOX genes

The role of HOX genes in human implantation

Exposure to bisphenol A advances puberty

Estrogenicity of resin-based composites and sealants used in dentistry

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