Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury - Test2 - elhamkhani - TriplyDB

Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury

Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury

http://www.wikidata.org/entity/Q24632770

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Monoamine oxidase B prompts mitochondrial and cardiac dysfunction in pressure overloaded hearts The Role of Mitochondrial Reactive Oxygen Species in Cardiovascular Injury and Protective Strategies Why Does Exercise "Trigger" Adaptive Protective Responses in the Heart?Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioning NADPH oxidases in heart failure: poachers or gamekeepers?Key Targets for Multi-Target Ligands Designed to Combat Neurodegeneration Mitochondrial Metabolism in Aging Heart The Concise Guide to PHARMACOLOGY 2013/14: enzymes.Effects of hypertension and exercise on cardiac proteome remodelling Monoamine oxidases as sources of oxidants in the heart Differential metal content and gene expression in rat left ventricular hypertrophy due to hypertension and hyperactivity.Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Reactive oxygen species and redox compartmentalization Mitochondrial approaches to protect against cardiac ischemia and reperfusion injury.Structure-based discovery of prescription drugs that interact with the norepinephrine transporter, NET.Monoamine oxidase A (MAO-A): a signature marker of alternatively activated monocytes/macrophages Reactive oxygen species in organ-specific autoimmunity.Oxidative stress during mitochondrial biogenesis compromises mtDNA integrity in growing hearts and induces a global DNA repair response.Quantitative proteomic analysis reveals novel mitochondrial targets of estrogen deficiency in the aged female rat heart IL-4 and IL-13 employ discrete signaling pathways for target gene expression in alternatively activated monocytes/macrophages.Age-related differences in cardiac ischemia-reperfusion injury: effects of estrogen deficiency.Concepts of scientific integrative medicine applied to the physiology and pathophysiology of catecholamine systems.Monoamine Oxidase Is Overactivated in Left and Right Ventricles from Ischemic Hearts: An Intriguing Therapeutic Target.Patent-related survey on new monoamine oxidase inhibitors and their therapeutic potential.From depression to neurodegeneration and heart failure: re-examining the potential of MAO inhibitors.Novel aspects of ROS signalling in heart failure.Monoamine oxidase A and B substrates: probing the pathway for drug development.The "Goldilocks Zone" from a redox perspective-Adaptive vs. deleterious responses to oxidative stress in striated muscle.Imaging mitochondrial reactive oxygen species with fluorescent probes: current applications and challenges.Equilibrative Nucleoside Transporters 1 and 4: Which One Is a Better Target for Cardioprotection Against Ischemia-Reperfusion Injury?β-Adrenergic signaling, monoamine oxidase A and antioxidant defence in the myocardium of SHR and SHR-mtBN conplastic rat strains: the effect of chronic hypoxia.Moving Forwards by Blocking Back-Flow: The Yin and Yang of MI Therapy.Rasagiline and selegiline suppress calcium efflux from mitochondria by PK11195-induced opening of mitochondrial permeability transition pore: a novel anti-apoptotic function for neuroprotection.Ischemia/Reperfusion.Mitochondrial ROS control of cancer.Role of the immune system in cardiac tissue damage and repair following myocardial infarction.Escaping Death: Mitochondrial Redox Homeostasis in Cancer Cells Proteomic and transcriptomic analysis of heart failure due to volume overload in a rat aorto-caval fistula model provides support for new potential therapeutic targets - monoamine oxidase A and transglutaminase 2.Monoamine oxidase inhibition improves vascular function in mammary arteries from nondiabetic and diabetic patients with coronary heart disease.Impact of caloric restriction on myocardial ischaemia/reperfusion injury and new therapeutic options to mimic its effects

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P2860

Monoamine oxidases (MAO) in the pathogenesis of heart failure and ischemia/reperfusion injury

http://www.wikidata.org/entity/Q24632770

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年论文

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Biochimica et Biophysica Acta

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Monoamine oxidases (MAO) in th ...... nd ischemia/reperfusion injury

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Andrea Carpi

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Nina Kaludercic

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Roberta Menabò

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P2860

Abnormal behavior associated with a point mutation in the structural gene for monoamine oxidase A

Specific genetic deficiencies of the A and B isoenzymes of monoamine oxidase are characterized by distinct neurochemical and clinical phenotypes

Aggressive behavior and altered amounts of brain serotonin and norepinephrine in mice lacking MAOA

How mitochondria produce reactive oxygen species

cDNA cloning of human liver monoamine oxidase A and B: molecular basis of differences in enzymatic properties

Activation of aldehyde dehydrogenase-2 reduces ischemic damage to the heart

Free radicals in the physiological control of cell function

Cloning, after cloning, knock-out mice, and physiological functions of MAO A and B

The FAD binding sites of human monoamine oxidases A and B

A key amino acid responsible for substrate selectivity of monoamine oxidase A and B

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Nazareno Paolocci

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20869994

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reperfusion injury

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