Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells - Test2 - elhamkhani - TriplyDB

Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells

Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells

http://www.wikidata.org/entity/Q24651034

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Gemcitabine sensitivity can be induced in pancreatic cancer cells through modulation of miR-200 and miR-21 expression by curcumin or its analogue CDF The ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?Cancer Stem Cells and Epithelial-to-Mesenchymal Transition (EMT)-Phenotypic Cells: Are They Cousins or Twins?Cellular and Molecular Mechanisms of 3,3'-Diindolylmethane in Gastrointestinal Cancer The Role of microRNAs in the Diagnosis and Treatment of Pancreatic Adenocarcinoma The miRacle in Pancreatic Cancer by miRNAs: Tiny Angels or Devils in Disease Progression Roles of Dietary Phytoestrogens on the Regulation of Epithelial-Mesenchymal Transition in Diverse Cancer Metastasis MicroRNA Targeted Therapeutic Approach for Pancreatic Cancer Hedgehog Signaling in Pancreatic Fibrosis and Cancer miRNAs As Diagnostic and Prognostic Biomarkers in Pancreatic Ductal Adenocarcinoma and Its Precursor Lesions: A Review Noncoding RNAs and pancreatic cancer Targeting colorectal cancer stem cells using curcumin and curcumin analogues: insights into the mechanism of the therapeutic efficacy Targeting CSCs in tumor microenvironment: the potential role of ROS-associated miRNAs in tumor aggressiveness Natural compounds as anticancer agents: Experimental evidence Epigenomics of leukemia: from mechanisms to therapeutic applications Antioxidant Mechanisms and ROS-Related MicroRNAs in Cancer Stem Cells Emerging roles of PDGF-D in EMT progression during tumorigenesis Pancreatic cancer stem cells: emerging target for designing novel therapy Targeting CSC-related miRNAs for cancer therapy by natural agents MicroRNA, nutrition, and cancer prevention The miR-200 and miR-221/222 microRNA families: opposing effects on epithelial identity MicroRNAs as targets for dietary and pharmacological inhibitors of mutagenesis and carcinogenesis Epigenetic diet: impact on the epigenome and cancer Regulating miRNA by natural agents as a new strategy for cancer treatment Noncoding RNAs in Tumor Epithelial-to-Mesenchymal Transition Concise Review: Stem Cells in Pancreatic Cancer: From Concept to Translation Let-7a inhibits migration, invasion and epithelial-mesenchymal transition by targeting HMGA2 in nasopharyngeal carcinoma miR-146a suppresses invasion of pancreatic cancer cells The Regulatory Role of MicroRNAs in EMT and Cancer A multi-targeted approach to suppress tumor-promoting inflammation EGFR-TKI resistance in NSCLC patients: mechanisms and strategies Sustained proliferation in cancer: Mechanisms and novel therapeutic targets Identification of microRNA-21 as a biomarker for chemoresistance and clinical outcome following adjuvant therapy in resectable pancreatic cancer Anti-tumor activity of a novel compound-CDF is mediated by regulating miR-21, miR-200, and PTEN in pancreatic cancer Activated K-ras and INK4a/Arf deficiency cooperate during the development of pancreatic cancer by activation of Notch and NF-κB signaling pathways 3, 3'-Diindolylmethane enhances the effectiveness of herceptin against HER-2/neu-expressing breast cancer cells Epimorphin-induced MET sensitizes ovarian cancer cells to platinum Let-7 Sensitizes KRAS Mutant Tumor Cells to Chemotherapy EMT, cancer stem cells and drug resistance: an emerging axis of evil in the war on cancer Evidence for mesenchymal-like sub-populations within squamous cell carcinomas possessing chemoresistance and phenotypic plasticity.

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Up-regulation of miR-200 and let-7 by natural agents leads to the reversal of epithelial-to-mesenchymal transition in gemcitabine-resistant pancreatic cancer cells

http://www.wikidata.org/entity/Q24651034

description

2009 nî lūn-bûn

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2009年の論文

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Up-regulation of miR-200 and l ...... istant pancreatic cancer cells

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Up-regulation of miR-200 and l ...... istant pancreatic cancer cells

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Up-regulation of miR-200 and l ...... istant pancreatic cancer cells

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Dejuan Kong

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Fazlul H Sarkar

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Philip A Philip

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Shadan Ali

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Timothy G VandenBoom

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Yiwei Li

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Zhiwei Wang

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Twist transcriptionally up-regulates AKT2 in breast cancer cells leading to increased migration, invasion, and resistance to paclitaxel

Direct regulation of TWIST by HIF-1alpha promotes metastasis

The miR-200 family inhibits epithelial-mesenchymal transition and cancer cell migration by direct targeting of E-cadherin transcriptional repressors ZEB1 and ZEB2

The miR-200 family determines the epithelial phenotype of cancer cells by targeting the E-cadherin repressors ZEB1 and ZEB2

Tumor protein 53-induced nuclear protein 1 expression is repressed by miR-155, and its restoration inhibits pancreatic tumor development

MicroRNA-dependent localization of targeted mRNAs to mammalian P-bodies

The tumor suppressor microRNA let-7 represses the HMGA2 oncogene.

Epithelial-mesenchymal transitions in tumour progression

Cancer statistics, 2008

MicroRNA gene expression deregulation in human breast cancer

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