A common variant in the 3'UTR of the GRIK4 glutamate receptor gene affects transcript abundance and protects against bipolar disorder
about
The genetics of bipolar disorderGlutamate receptor ion channels: structure, regulation, and functionAnalysis of pharmacogenomic variants associated with population differentiationGenome-wide association study in bipolar patients stratified by co-morbidityThe GluK4 kainate receptor subunit regulates memory, mood, and excitotoxic neurodegeneration.Complete Disruption of the Kainate Receptor Gene Family Results in Corticostriatal Dysfunction in Mice.Brain glutamatergic characteristics of pediatric offspring of parents with bipolar disorder.Detecting local haplotype sharing and haplotype associationGenetic variants in human leukocyte antigen-DP influence both hepatitis C virus persistence and hepatitis C virus F protein generation in the Chinese Han population.Synaptic drive at developing synapses: transient upregulation of kainate receptors.Identifying and searching for conserved RNA localisation signals.A cytogenetic abnormality and rare coding variants identify ABCA13 as a candidate gene in schizophrenia, bipolar disorder, and depressionExpression profiling in neuropsychiatric disorders: emphasis on glutamate receptors in bipolar disorderGenetic ablation of the GluK4 kainate receptor subunit causes anxiolytic and antidepressant-like behavior in mice.Ionotropic GABA and Glutamate Receptor Mutations and Human Neurologic Diseases.Targeted massively parallel sequencing of autism spectrum disorder-associated genes in a case control cohort reveals rare loss-of-function risk variants.Glutamate receptor mutations in psychiatric and neurodevelopmental disorders.Emerging Synaptic Molecules as Candidates in the Etiology of Neurological Disorders.Progress in defining the biological causes of schizophrenia.Genetic Studies on the Tripartite Glutamate Synapse in the Pathophysiology and Therapeutics of Mood Disorders.GRIK4/KA1 protein expression in human brain and correlation with bipolar disorder risk variant status.Characterization of a Novel Mutation in SLC1A1 Associated with Schizophrenia.Molecular characterization of myostatin (MSTN) gene and association analysis with growth traits in the bighead carp (Aristichthys nobilis).Identification of common variants in BRCA2 and MAP2K4 for susceptibility to sporadic pancreatic cancer.A GRIK4 variant conferring protection against bipolar disorder modulates hippocampal function.Framework for reanalysis of publicly available Affymetrix® GeneChip® data sets based on functional regions of interest.A kainate receptor GluK4 deletion, protective against bipolar disorder, is associated with enhanced cognitive performance across diagnoses in the TwinsUK cohort.Kainate Receptors Inhibit Glutamate Release Via Mobilization of Endocannabinoids in Striatal Direct Pathway Spiny Projection Neurons.
P2860
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P2860
A common variant in the 3'UTR of the GRIK4 glutamate receptor gene affects transcript abundance and protects against bipolar disorder
description
2008 nî lūn-bûn
@nan
2008 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
A common variant in the 3'UTR ...... tects against bipolar disorder
@ast
A common variant in the 3'UTR ...... tects against bipolar disorder
@en
A common variant in the 3'UTR ...... tects against bipolar disorder
@nl
type
label
A common variant in the 3'UTR ...... tects against bipolar disorder
@ast
A common variant in the 3'UTR ...... tects against bipolar disorder
@en
A common variant in the 3'UTR ...... tects against bipolar disorder
@nl
prefLabel
A common variant in the 3'UTR ...... tects against bipolar disorder
@ast
A common variant in the 3'UTR ...... tects against bipolar disorder
@en
A common variant in the 3'UTR ...... tects against bipolar disorder
@nl
P2093
P2860
P356
P1476
A common variant in the 3'UTR ...... tects against bipolar disorder
@en
P2093
B S Pickard
D C Soares
D H R Blackwood
D J Porteous
D St Clair
H M Knight
J K F Boyd
P2860
P304
P356
10.1073/PNAS.0800643105
P407
P50
P577
2008-09-30T00:00:00Z