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Mechanisms of chronic central neuropathic pain after spinal cord injury

Mechanisms of chronic central neuropathic pain after spinal cord injury

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The search for novel analgesics: targets and mechanisms Poly(ADP-ribose) polymerase inhibition reveals a potential mechanism to promote neuroprotection and treat neuropathic pain Cannabinoid receptor-mediated antinociception with acetaminophen drug combinations in rats with neuropathic spinal cord injury pain Acrolein involvement in sensory and behavioral hypersensitivity following spinal cord injury in the rat Aberrant sensory responses are dependent on lesion severity after spinal cord contusion injury in mice.New insights on NOX enzymes in the central nervous system.Chronic post-traumatic headache: clinical findings and possible mechanisms.Spinal cord transection-induced allodynia in rats--behavioral, physiopathological and pharmacological characterization Aquaporins in spinal cord injury: the janus face of aquaporin 4 Isolated spinal cord contusion in rats induces chronic brain neuroinflammation, neurodegeneration, and cognitive impairment. Involvement of cell cycle activation Spinal cord injury causes brain inflammation associated with cognitive and affective changes: role of cell cycle pathways.Spinal cord injuries containing asymmetrical damage in the ventrolateral funiculus is associated with a higher incidence of at-level allodynia Chronic at-level thermal hyperalgesia following rat cervical contusion spinal cord injury is accompanied by neuronal and astrocyte activation and loss of the astrocyte glutamate transporter, GLT1, in superficial dorsal horn.Pathogenesis of spinal cord injury induced edema and neuropathic pain: expression of multiple isoforms of wnk1.Vascular endothelial growth factor and spinal cord injury pain.Persistent at-level thermal hyperalgesia and tactile allodynia accompany chronic neuronal and astrocyte activation in superficial dorsal horn following mouse cervical contusion spinal cord injury Neuroprotective role of hydralazine in rat spinal cord injury-attenuation of acrolein-mediated damage.Characterization of recovery, repair, and inflammatory processes following contusion spinal cord injury in old female rats: is age a limitation?Chronic spontaneous activity generated in the somata of primary nociceptors is associated with pain-related behavior after spinal cord injury.Gap junction proteins and their role in spinal cord injury Dysregulation of Kv3.4 channels in dorsal root ganglia following spinal cord injury Effect of the Combination of CI-988 and Morphine on Neuropathic Pain after Spinal Cord Injury in Rats.Conditioned place preference reveals tonic pain in an animal model of central pain.Somatosensory phenotype is associated with thalamic metabolites and pain intensity after spinal cord injury Fibronectin inhibits chronic pain development after spinal cord injury Randall-Selitto test: a new approach for the detection of neuropathic pain after spinal cord injury.Spinal cord injury triggers an intrinsic growth-promoting state in nociceptors Toll-like receptors in chronic pain.Maladaptive spinal plasticity opposes spinal learning and recovery in spinal cord injury.Neuropathic Pain and Spinal Cord Injury: Phenotypes and Pharmacological Management.Thalamocortical asynchrony in conditions of spinal cord injury pain in rats.GLT1 overexpression reverses established neuropathic pain-related behavior and attenuates chronic dorsal horn neuron activation following cervical spinal cord injury.Acrolein contributes to TRPA1 up-regulation in peripheral and central sensory hypersensitivity following spinal cord injury.Progressive inflammation-mediated neurodegeneration after traumatic brain or spinal cord injury.Chronic pain following spinal cord injury Licofelone modulates neuroinflammation and attenuates mechanical hypersensitivity in the chronic phase of spinal cord injury Astrocytic CX43 hemichannels and gap junctions play a crucial role in development of chronic neuropathic pain following spinal cord injury.Ablation of the transcription factors E2F1-2 limits neuroinflammation and associated neurological deficits after contusive spinal cord injury Dietary omega-3 polyunsaturated fatty acids improve the neurolipidome and restore the DHA status while promoting functional recovery after experimental spinal cord injury Metabolite concentrations in the anterior cingulate cortex predict high neuropathic pain impact after spinal cord injury.

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Mechanisms of chronic central neuropathic pain after spinal cord injury

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Mechanisms of chronic central neuropathic pain after spinal cord injury

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Mechanisms of chronic central neuropathic pain after spinal cord injury

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Mechanisms of chronic central neuropathic pain after spinal cord injury

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Mechanisms of chronic central neuropathic pain after spinal cord injury

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J.BRAINRESREV.2008.12.010

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Brain Research Reviews

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Mechanisms of chronic central neuropathic pain after spinal cord injury

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Bryan C Hains

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Claire E Hulsebosch

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Eric D Crown

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Infiltrative microgliosis: activation and long-distance migration of subependymal microglia following periventricular insults

International union of pharmacology. XXII. Nomenclature for chemokine receptors

Modulation of thalamic nociceptive processing after spinal cord injury through remote activation of thalamic microglia by cysteine cysteine chemokine ligand 21

Challenge with innate and protein antigens induces CCR7 expression by microglia in vitro and in vivo

Excitotoxic spinal cord injury: behavioral and morphological characteristics of a central pain model

Excitatory monocyte chemoattractant protein-1 signaling is up-regulated in sensory neurons after chronic compression of the dorsal root ganglion

Upregulation of sodium channel Nav1.3 and functional involvement in neuronal hyperexcitability associated with central neuropathic pain after spinal cord injury

Glutamate-induced losses of oligodendrocytes and neurons and activation of caspase-3 in the rat spinal cord

Microglia: active sensor and versatile effector cells in the normal and pathologic brain

Function and regulation of CREB family transcription factors in the nervous system

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neuropathic pain

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