Allelic imbalances and microdeletions affecting the PTPRD gene in cutaneous squamous cell carcinomas detected using single nucleotide polymorphism microarray analysis - Test2 - elhamkhani - TriplyDB

Allelic imbalances and microdeletions affecting the PTPRD gene in cutaneous squamous cell carcinomas detected using single nucleotide polymorphism microarray analysis

Allelic imbalances and microdeletions affecting the PTPRD gene in cutaneous squamous cell carcinomas detected using single nucleotide polymorphism microarray analysis

http://www.wikidata.org/entity/Q24657188

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Loss-of-Function PTPRD Mutations Lead to Increased STAT3 Activation and Sensitivity to STAT3 Inhibition in Head and Neck Cancer The tyrosine phosphatase PTPRD is a tumor suppressor that is frequently inactivated and mutated in glioblastoma and other human cancers Genomic alterations in breast cancer patients in betel quid and non betel quid chewers Genome wide analysis of DNA copy number neutral loss of heterozygosity (CNNLOH) and its relation to gene expression in esophageal squamous cell carcinoma High-resolution genomic profiling of human papillomavirus-associated vulval neoplasia.Genomic profiling of adult acute lymphoblastic leukemia by single nucleotide polymorphism oligonucleotide microarray and comparison to pediatric acute lymphoblastic leukemia Loss of inositol polyphosphate 5-phosphatase is an early event in development of cutaneous squamous cell carcinoma.Reduced expression of PTPRD correlates with poor prognosis in gastric adenocarcinoma.Molecular profiling of cutaneous squamous cell carcinomas and actinic keratoses from organ transplant recipients.Key differences identified between actinic keratosis and cutaneous squamous cell carcinoma by transcriptome profiling.Stress alters rates and types of loss of heterozygosity in Candida albicans.Integrative mRNA profiling comparing cultured primary cells with clinical samples reveals PLK1 and C20orf20 as therapeutic targets in cutaneous squamous cell carcinoma From keratinocyte to cancer: the pathogenesis and modeling of cutaneous squamous cell carcinoma.Integrated genomic analysis of sézary syndrome Protein tyrosine phosphatase receptor delta acts as a neuroblastoma tumor suppressor by destabilizing the aurora kinase A oncogene.Integrating the multiple dimensions of genomic and epigenomic landscapes of cancer.Somatic uniparental isodisomy explains multifocality of glomuvenous malformations.SNP arrays in heterogeneous tissue: highly accurate collection of both germline and somatic genetic information from unpaired single tumor samples.Abundant copy-number loss of CYCLOPS and STOP genes in gastric adenocarcinoma.Detection of DNA copy number alterations in cancer by array comparative genomic hybridization.PTCH mutations in basal cell carcinomas from azathioprine-treated organ transplant recipients.Protein tyrosine phosphatases: functional inferences from mouse models and human diseases.Allele-specific imbalance mapping identifies HDAC9 as a candidate gene for cutaneous squamous cell carcinoma.Chromosomal aberrations in UVB-induced tumors of immunosuppressed mice.Lack of inherited mutations of PTPRD in familial melanoma and melanoma-astrocytoma syndrome.Mutational inactivation of PTPRD in glioblastoma multiforme and malignant melanoma.High throughput molecular diagnostics in bladder cancer - on the brink of clinical utility.Genetic alterations of protein tyrosine phosphatases in human cancers.Receptor-type protein tyrosine phosphatases in cancer.Phosphoinositide 3-Kinase-Dependent Signalling Pathways in Cutaneous Squamous Cell Carcinomas.miR-135a-5p-mediated downregulation of protein tyrosine phosphatase receptor delta is a candidate driver of HCV-associated hepatocarcinogenesis.The telomere profile distinguishes two classes of genetically distinct cutaneous squamous cell carcinomas.Molecular markers in cutaneous squamous cell carcinoma Single nucleotide polymorphism array analysis defines a specific genetic fingerprint for well-differentiated cutaneous SCCs.Examination of Smad2 and Smad4 copy-number variations in skin cancers.Metastatic cutaneous squamous cell carcinoma shows frequent deletion in the protein tyrosine phosphatase receptor Type D gene.D9S168 microsatellite alteration predicts a poor prognosis in patients with clear cell renal cell carcinoma and correlates with the down-regulation of protein tyrosine phosphatase receptor delta.Protein Tyrosine Phosphatases as Potential Regulators of STAT3 Signaling

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Allelic imbalances and microdeletions affecting the PTPRD gene in cutaneous squamous cell carcinomas detected using single nucleotide polymorphism microarray analysis

http://www.wikidata.org/entity/Q24657188

description

2007 nî lūn-bûn

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2007 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2007年の論文

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2007年論文

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Allelic imbalances and microde ...... lymorphism microarray analysis

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Allelic imbalances and microde ...... lymorphism microarray analysis

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Allelic imbalances and microde ...... lymorphism microarray analysis

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Allelic imbalances and microde ...... lymorphism microarray analysis

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Genes, Chromosomes and Cancer

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Allelic imbalances and microde ...... lymorphism microarray analysis

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Bryan D Young

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Charlotte M Proby

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Gael Molloy

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Irene M Leigh

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Karin J Purdie

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Sally R Lambert

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Tracy Chaplin

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P2860

Protein tyrosine phosphatase receptor-type O (PTPRO) exhibits characteristics of a candidate tumor suppressor in human lung cancer

Global variation in copy number in the human genome

Receptor tyrosine phosphatase-dependent cytoskeletal remodeling by the hedgehog-responsive gene MIM/BEG4

The consensus coding sequences of human breast and colorectal cancers

The epidemiology of UV induced skin cancer.

Identification of chromosome arm 9p as the most frequent target of homozygous deletions in lung cancer.

A role for sunlight in skin cancer: UV-induced p53 mutations in squamous cell carcinoma.

Are keratoacanthomas variants of squamous cell carcinomas? A comparison of chromosomal aberrations by comparative genomic hybridization.

High-resolution analysis of chromosomal breakpoints and genomic instability identifies PTPRD as a candidate tumor suppressor gene in neuroblastoma.

Homozygous deletions and chromosome amplifications in human lung carcinomas revealed by single nucleotide polymorphism array analysis.

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661-669

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10.1002/GCC.20447

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7

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46

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David P. Kelsell

Catherine Harwood

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2007-07-01T00:00:00Z

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6401684

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17420988

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single-nucleotide polymorphism

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2426828

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