C-Reactive protein binds to apoptotic cells, protects the cells from assembly of the terminal complement components, and sustains an antiinflammatory innate immune response: implications for systemic autoimmunity

C-Reactive protein binds to apoptotic cells, protects the cells from assembly of the terminal complement components, and sustains an antiinflammatory innate immune response: implications for systemic autoimmunity

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http://www.wikidata.org/entity/Q24675774

Q24675774

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C1q and mannose binding lectin engagement of cell surface calreticulin and CD91 initiates macropinocytosis and uptake of apoptotic cells C-reactive protein: a critical update I-PLA(2) activation during apoptosis promotes the exposure of membrane lysophosphatidylcholine leading to binding by natural immunoglobulin M antibodies and complement activation Serum levels of autoantibodies against monomeric C-reactive protein are correlated with disease activity in systemic lupus erythematosus Autoantibody profile in rheumatoid arthritis during long-term infliximab treatment C-reactive protein does not opsonize early apoptotic human neutrophils, but binds only membrane-permeable late apoptotic cells and has no effect on their phagocytosis by macrophages SLE-associated risk factors affect DC function Structure shows that a glycosaminoglycan and protein recognition site in factor H is perturbed by age-related macular degeneration-linked single nucleotide polymorphism Pentraxins (CRP, SAP) in the process of complement activation and clearance of apoptotic bodies through Fcγ receptors.The Complement System and Antibody-Mediated Transplant Rejection The innate immune response to products of phospholipid peroxidation Generation and biological activities of oxidized phospholipids Rat C-reactive protein activates the autologous complement system Pentraxins: structure, function, and role in inflammation Complement: a key system for immune surveillance and homeostasis Membrane curvature recognition by C-reactive protein using lipoprotein mimics.FcγRIIB and autoimmunity.Complement factor H: using atomic resolution structure to illuminate disease mechanisms.The era of molecular and other non-culture-based methods in diagnosis of sepsis The pentraxins PTX3 and SAP in innate immunity, regulation of inflammation and tissue remodelling Milk fat globule epidermal growth factor 8 attenuates acute lung injury in mice after intestinal ischemia and reperfusion Hepatic complications induced by immunosuppressants and biologics in inflammatory bowel disease.Targeted and restricted complement activation on acrosome-reacted spermatozoa.Host biomarkers of invasive pulmonary aspergillosis to monitor therapeutic response Neointimal formation is reduced after arterial injury in human crp transgenic mice Mutations of C-reactive protein (CRP) -286 SNP, APC and p53 in colorectal cancer: implication for a CRP-Wnt crosstalk.Human pentraxin 3 binds to the complement regulator c4b-binding protein.Complement-dependent modulation of antitumor immunity following radiation therapy.Complement control protein factor H: the good, the bad, and the inadequate.Milk fat globule EGF factor 8 attenuates sepsis-induced apoptosis and organ injury in alcohol-intoxicated rats.PTX3 in small-vessel vasculitides: an independent indicator of disease activity produced at sites of inflammation.C-reactive protein binds to both oxidized LDL and apoptotic cells through recognition of a common ligand: Phosphorylcholine of oxidized phospholipids NETs: the missing link between cell death and systemic autoimmune diseases?The macrophage and the apoptotic cell: an innate immune interaction viewed simplistically?A prospective evaluation of C-reactive protein levels and colorectal adenoma development.Phagocytosis of opsonized apoptotic cells: roles for 'old-fashioned' receptors for antibody and complement.Pathogen recognition by the long pentraxin PTX3.Protective molecules and their cognate antibodies: new players in autoimmunity Serum amyloid P component levels are not decreased in patients with systemic lupus erythematosus and do not rise during an acute phase reaction.C-reactive protein and cardiovascular disease: new insights from an old molecule.

P2860

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P2860

C-Reactive protein binds to apoptotic cells, protects the cells from assembly of the terminal complement components, and sustains an antiinflammatory innate immune response: implications for systemic autoimmunity

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http://www.wikidata.org/entity/Q24675774

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2000 nî lūn-bûn

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2000 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2000 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2000年の論文

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2000年論文

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2000年論文

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2000年論文

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2000年論文

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