A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4 - Test2 - elhamkhani - TriplyDB

A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4

A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4

http://www.wikidata.org/entity/Q24683143

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Transcriptional regulation of long-term memory in the marine snail Aplysia The problem of pseudoreplication in neuroscientific studies: is it affecting your analysis?Rolipram: a specific phosphodiesterase 4 inhibitor with potential antipsychotic activity.Epigenetic mechanisms underlying extinction of memory and drug-seeking behavior Epigenetics in the mature mammalian brain: effects on behavior and synaptic transmission Global and local missions of cAMP signaling in neural plasticity, learning, and memory The molecular basis of cognitive deficits in pervasive developmental disorders PDE4 as a target for cognition enhancement Genes and signaling pathways involved in memory enhancement in mutant mice From Learning to Memory: What Flies Can Tell Us about Intellectual Disability Treatment Potential for treatment of severe autism in tuberous sclerosis complex GABA(A) receptor-mediated acceleration of aging-associated memory decline in APP/PS1 mice and its pharmacological treatment by picrotoxin Mutation of the CH1 Domain in the Histone Acetyltransferase CREBBP Results in Autism-Relevant Behaviors in Mice Psychiatric aspects of phosphodiesterases: An overview.Common molecular mechanisms in explicit and implicit memory Phosphodiesterases as therapeutic targets for Alzheimer's disease.Epigenetics in the nervous system A new model of Pde4d deficiency: genetic knock-down of PDE4D enzyme in rats produces an antidepressant phenotype without spatial cognitive effects.A novel neurotrophic drug for cognitive enhancement and Alzheimer's disease A biphasic and brain-region selective down-regulation of cyclic adenosine monophosphate concentrations supports object recognition in the rat Spatial memory consolidation is associated with induction of several lysine-acetyltransferase (histone acetyltransferase) expression levels and H2B/H4 acetylation-dependent transcriptional events in the rat hippocampus.GEBR-7b, a novel PDE4D selective inhibitor that improves memory in rodents at non-emetic doses Enhanced expression of ADCY1 underlies aberrant neuronal signalling and behaviour in a syndromic autism model.Cdk5 is required for memory function and hippocampal plasticity via the cAMP signaling pathway.Developmental etiology for neuroanatomical and cognitive deficits in mice overexpressing Galphas, a G-protein subunit genetically linked to schizophrenia.Constitutive activation of Galphas within forebrain neurons causes deficits in sensorimotor gating because of PKA-dependent decreases in cAMP.CREB binding protein is required for both short-term and long-term memory formation.Using the MATRICS to guide development of a preclinical cognitive test battery for research in schizophrenia.Memory acquisition and retrieval impact different epigenetic processes that regulate gene expression.The cyclic AMP cascade is altered in the fragile X nervous system Transgenic mice expressing a truncated form of CREB-binding protein (CBP) exhibit deficits in hippocampal synaptic plasticity and memory storage.Phosphodiesterase type 1 inhibition improves learning in rats exposed to alcohol during the third trimester equivalent of human gestation Phosphodiesterase type 4 inhibition does not restore ocular dominance plasticity in a ferret model of fetal alcohol spectrum disorders.Membrane-associated glucocorticoid activity is necessary for modulation of long-term memory via chromatin modification.Flavonoid fisetin promotes ERK-dependent long-term potentiation and enhances memory.Brain region-specific decrease in the activity and expression of protein kinase A in the frontal cortex of regressive autism.A novel conditional genetic system reveals that increasing neuronal cAMP enhances memory and retrieval.CBP is required for environmental enrichment-induced neurogenesis and cognitive enhancement.Regulation of hippocampus-dependent memory by cyclic AMP-dependent protein kinase Histone deacetylase inhibitors enhance memory and synaptic plasticity via CREB:CBP-dependent transcriptional activation.

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A mouse model of Rubinstein-Taybi syndrome: defective long-term memory is ameliorated by inhibitors of phosphodiesterase 4

http://www.wikidata.org/entity/Q24683143

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2003 nî lūn-bûn

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Proceedings of the National Academy of Sciences of the United States of America

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A mouse model of Rubinstein-Ta ...... ibitors of phosphodiesterase 4

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Darlene Romashko

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Jennifer Stanley

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Ray Catapano

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Regina Lidge

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Rod Scott

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Rusiko Bourtchouladze

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Scott Gossweiler

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Tim Tully

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P2860

Rolipram, a type IV-specific phosphodiesterase inhibitor, facilitates the establishment of long-lasting long-term potentiation and improves memory

A CBP integrator complex mediates transcriptional activation and AP-1 inhibition by nuclear receptors

Transcription factor-specific requirements for coactivators and their acetyltransferase functions

Rubinstein-Taybi syndrome caused by mutations in the transcriptional co-activator CBP

Impairment of cerebral cAMP-mediated signal transduction system and of spatial memory function after microsphere embolism in rats

The transcriptional co-activator p/CIP binds CBP and mediates nuclear-receptor function

Acetylation: a regulatory modification to rival phosphorylation?

Function and regulation of CREB family transcription factors in the nervous system

Rubinstein-Taybi syndrome. Review of 732 cases and analysis of the typical traits.

Role of Ca2+-stimulated adenylyl cyclases in LTP and memory formation.

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