Role of glucocorticoids in mediating effects of fasting and diabetes on hypothalamic gene expression - Test2 - elhamkhani - TriplyDB

Role of glucocorticoids in mediating effects of fasting and diabetes on hypothalamic gene expression

Role of glucocorticoids in mediating effects of fasting and diabetes on hypothalamic gene expression

http://www.wikidata.org/entity/Q24793433

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Control of energy balance by hypothalamic gene circuitry involving two nuclear receptors, neuron-derived orphan receptor 1 and glucocorticoid receptor Mammalian comparative sequence analysis of the Agrp locus Regulation of Agouti-Related Protein and Pro-Opiomelanocortin Gene Expression in the Avian Arcuate Nucleus Functional role of c-Jun-N-terminal kinase in feeding regulation.Hypothalamic responses to fasting indicate metabolic reprogramming away from glycolysis toward lipid oxidation.5-hydroxy-L-tryptophan Suppressed Food Intake in Rats Despite an Increase in the Arcuate NPY Expression.Hypothalamic Fkbp51 is induced by fasting, and elevated hypothalamic expression promotes obese phenotypes.Glucocorticoids decrease body weight and food intake and inhibit appetite regulatory peptide expression in the hypothalamus of rats.Brain-specific homeobox factor as a target selector for glucocorticoid receptor in energy balance.Hypothalamic Agouti-Related Peptide mRNA is Elevated During Natural and Stress-Induced Anorexia Effects of BDNF, T3, and corticosterone on expression of the hypothalamic obesity gene network in vivo and in vitro Leptin metabolically licenses T cells for activation to link nutrition and immunity Leptin receptor gene expression and number in the brain are regulated by leptin level and nutritional status.Behavioral and Stereological Analysis of the Effects of Intermittent Feeding Diet on the Orally Administrated MDMA ("ecstasy") in Mice.Insulin determines leptin responses during a glucose challenge in fed and fasted rats.Stress symptoms induced by repeated morphine withdrawal in comparison to other chronic stress models in mice.Apolipoprotein A-IV constrains HPA and behavioral stress responsivity in a strain-dependent manner.Caloric restriction selectively reduces the GABAergic phenotype of mouse hypothalamic proopiomelanocortin neurons.Early life stress experience may blunt hypothalamic leptin signalling.Effects of maternal separation on the dietary preference and behavioral satiety sequence in rats.

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Role of glucocorticoids in mediating effects of fasting and diabetes on hypothalamic gene expression

http://www.wikidata.org/entity/Q24793433

description

2003 nî lūn-bûn

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2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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Role of glucocorticoids in med ...... n hypothalamic gene expression

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Role of glucocorticoids in med ...... n hypothalamic gene expression

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Role of glucocorticoids in med ...... n hypothalamic gene expression

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Role of glucocorticoids in med ...... n hypothalamic gene expression

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Charles V Mobbs

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Fumiko Isoda

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Hideo Makimura

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Jeffrey H Silverstein

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Joe Beasley

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Tooru M Mizuno

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P2860

Effects of streptozotocin-induced diabetes and insulin treatment on the hypothalamic melanocortin system and muscle uncoupling protein 3 expression in rats

Effects of leptin receptor mutation on Agrp gene expression in fed and fasted lean and obese (LA/N-faf) rats

Role of leptin in the neuroendocrine response to fasting

Insulin and leptin: dual adiposity signals to the brain for the regulation of food intake and body weight.

Two important systems in energy homeostasis: melanocortins and melanin-concentrating hormone.

Leptin regulation of proopiomelanocortin.

A transgenic model of visceral obesity and the metabolic syndrome.

Fasting regulates hypothalamic neuropeptide Y, agouti-related peptide, and proopiomelanocortin in diabetic mice independent of changes in leptin or insulin.

Obese gene expression: reduction by fasting and stimulation by insulin and glucose in lean mice, and persistent elevation in acquired (diet-induced) and genetic (yellow agouti) obesity.

Effects of leptin and corticosterone on the expression of corticotropin-releasing hormone, agouti-related protein, and proopiomelanocortin in the brain of ob/ob mouse.

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