Retinoic acid and arsenic for treating acute promyelocytic leukemia - Test2 - elhamkhani - TriplyDB

Retinoic acid and arsenic for treating acute promyelocytic leukemia

Retinoic acid and arsenic for treating acute promyelocytic leukemia

http://www.wikidata.org/entity/Q24809165

about

From an old remedy to a magic bullet: molecular mechanisms underlying the therapeutic effects of arsenic in fighting leukemia Arsenic exposure and the induction of human cancers Decomposition of gene expression state space trajectories The NRF2-mediated oxidative stress response pathway is associated with tumor cell resistance to arsenic trioxide across the NCI-60 panel.An emerging role for epigenetic dysregulation in arsenic toxicity and carcinogenesis.Acute promyelocytic leukemia: a model of molecular target based therapy.Dissection of mechanisms of Chinese medicinal formula Realgar-Indigo naturalis as an effective treatment for promyelocytic leukemia.Treatment of acute promyelocytic leukaemia with all-trans retinoic acid and arsenic trioxide: a paradigm of synergistic molecular targeting therapy.Biological screening of natural products and drug innovation in China.Targeted therapy: The new lease on life for acute promyelocytic leukemia, and beyond.Epigenetic alterations in acute myeloid leukemias.Synergistic targeted therapy for acute promyelocytic leukaemia: a model of translational research in human cancer.Combined chemotherapy for acute promyelocytic leukemia: a meta-analysis.

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Retinoic acid and arsenic for treating acute promyelocytic leukemia

http://www.wikidata.org/entity/Q24809165

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2005 nî lūn-bûn

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2005 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2005年の論文

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2005年論文

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2005年論文

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Retinoic acid and arsenic for treating acute promyelocytic leukemia

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Sai-Juan Chen

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Wei-Li Zhao

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Zhen-Yi Wang

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Zhu Chen

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Creative Commons Attribution 4.0 International

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Conjugation with the ubiquitin-related modifier SUMO-1 regulates the partitioning of PML within the nucleus.

Differential ligand-dependent interactions between the AF-2 activating domain of nuclear receptors and the putative transcriptional intermediary factors mSUG1 and TIF1

Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RAR alpha with a novel putative transcription factor, PML

Deconstructing a disease: RARalpha, its fusion partners, and their roles in the pathogenesis of acute promyelocytic leukemia

Ham-Wasserman lecture: treatment of acute leukemia by inducing differentiation and apoptosis

The t(15;17) translocation of acute promyelocytic leukaemia fuses the retinoic acid receptor alpha gene to a novel transcribed locus

Use of all-trans retinoic acid in the treatment of acute promyelocytic leukemia

Retinoic acid in the treatment of acute promyelocytic leukemia: inefficacy of the 13-cis isomer and induction of complete remission by the all-trans isomer complicated by thromboembolic events

Role of promyelocytic leukemia (PML) sumolation in nuclear body formation, 11S proteasome recruitment, and As2O3-induced PML or PML/retinoic acid receptor alpha degradation

Tissue factors on acute promyelocytic leukemia and endothelial cells are differently regulated by retinoic acid, arsenic trioxide and chemotherapeutic agents

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