Interference by toxic metal ions with DNA repair processes and cell cycle control: molecular mechanisms - Test2 - elhamkhani - TriplyDB

Interference by toxic metal ions with DNA repair processes and cell cycle control: molecular mechanisms

Interference by toxic metal ions with DNA repair processes and cell cycle control: molecular mechanisms

http://www.wikidata.org/entity/Q24812521

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Cadmium inhibits mismatch repair by blocking the ATPase activity of the MSH2-MSH6 complex Arsenic-induced enhancement of ultraviolet radiation carcinogenesis in mouse skin: a dose-response study Inhibition of Ape1 nuclease activity by lead, iron, and cadmium.Ascorbate depletion: a critical step in nickel carcinogenesis?Advances in Understanding How Heavy Metal Pollution Triggers Gastric Cancer Low concentration of arsenite exacerbates UVR-induced DNA strand breaks by inhibiting PARP-1 activity Inhibition of poly(ADP-ribose) polymerase-1 by arsenite interferes with repair of oxidative DNA damage Causes of genome instability: the effect of low dose chemical exposures in modern society Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: the challenge ahead Nickel stimulates L1 retrotransposition by a post-transcriptional mechanism Nickel Compounds Cysteine and histidine shuffling: mixing and matching cysteine and histidine residues in zinc finger proteins to afford different folds and function.Membrane transporters and protein traffic networks differentially affecting metal tolerance: a genomic phenotyping study in yeast.A genome-wide deletion mutant screen identifies pathways affected by nickel sulfate in Saccharomyces cerevisiae Uptake of CdSe and CdSe/ZnS quantum dots into bacteria via purine-dependent mechanisms.Properties of experimental copper-aluminium-nickel alloys for dental post-and-core applications The effects of arsenic trioxide on DNA synthesis and genotoxicity in human colon cancer cells Distinct signaling pathways respond to arsenite and reactive oxygen species in Schizosaccharomyces pombe.Specific Inhibition of NEIL-initiated repair of oxidized base damage in human genome by copper and iron: potential etiological linkage to neurodegenerative diseases.Response to arsenate treatment in Schizosaccharomyces pombe and the role of its arsenate reductase activity.Environmental cadmium and breast cancer risk.Gene-environment interactions between JAZF1 and occupational and household lead exposure in prostate cancer among African American men.Cytotoxic Effect of Arsenic Trioxide in Adenocarcinoma Colorectal Cancer (HT-29) Cells.Exposures to airborne particulate matter and adverse perinatal outcomes: a biologically plausible mechanistic framework for exploring potential effect modification by nutrition Prevalence of at-risk genotypes for genotoxic effects decreases with age in a randomly selected population in Flanders: a cross sectional study.Metal binding mediated conformational change of XPA protein:a potential cytotoxic mechanism of nickel in the nucleotide excision repair A genome-wide screen of genes involved in cadmium tolerance in Schizosaccharomyces pombe.As(III) inhibits ultraviolet radiation-induced cyclobutane pyrimidine dimer repair via generation of nitric oxide in human keratinocytes.Characterization of the exposure-disease continuum in neonates of mothers exposed to carcinogens during pregnancy.Significance of differential metal loads in normal versus cancerous cadaver tissues - biomed 2010.Effects of corexit oil dispersants and the WAF of dispersed oil on DNA damage and repair in cultured human bronchial airway cells, BEAS-2B.Effects of nickel on cyclin expression, cell cycle progression and cell proliferation in human pulmonary cells.Genetic and epigenetic effects of environmental arsenicals.Genetic toxicology of thallium: a review.Metal-mediated DNA damage and cell death: mechanisms, detection methods, and cellular consequences.The comet assay in Environmental Risk Assessment of marine pollutants: applications, assets and handicaps of surveying genotoxicity in non-model organisms.In vitro genotoxicity of airborne Ni-NP in air-liquid interface.Nickel induces transcriptional down-regulation of DNA repair pathways in tumorigenic and non-tumorigenic lung cells.Genotoxicity and DNA repair processes of zinc oxide nanoparticles.Microbial strategy for potential lead remediation: a review study.

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Interference by toxic metal ions with DNA repair processes and cell cycle control: molecular mechanisms

http://www.wikidata.org/entity/Q24812521

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2002 nî lūn-bûn

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P2860

Instability and decay of the primary structure of DNA

The DNA damage response: putting checkpoints in perspective

Cadmium induces conformational modifications of wild-type p53 and suppresses p53 response to DNA damage in cultured cells

Differential effects of toxic metal compounds on the activities of Fpg and XPA, two zinc finger proteins involved in DNA repair.

Carcinogenicity of metal compounds: possible role of DNA repair inhibition.

p53 and human cancer: the first ten thousand mutations.

Nucleotide excision repair and human syndromes.

Arsenic disrupts cellular levels of p53 and mdm2: a potential mechanism of carcinogenesis.

Molecular models in nickel carcinogenesis.

Arsenite is a cocarcinogen with solar ultraviolet radiation for mouse skin: an animal model for arsenic carcinogenesis.

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110 Suppl 5

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