Parainflammation, chronic inflammation, and age-related macular degeneration - Test2 - elhamkhani - TriplyDB

Parainflammation, chronic inflammation, and age-related macular degeneration

Parainflammation, chronic inflammation, and age-related macular degeneration

http://www.wikidata.org/entity/Q26796268

about

Epidemiology of age-related macular degeneration (AMD): associations with cardiovascular disease phenotypes and lipid factors.Combination of ginsenoside Rb1 and Rd protects the retina against bright light-induced degeneration Expression pattern of Ccr2 and Cx3cr1 in inherited retinal degeneration Gut microbiota influences pathological angiogenesis in obesity-driven choroidal neovascularization.Targeting translocator protein (18 kDa) (TSPO) dampens pro-inflammatory microglia reactivity in the retina and protects from degeneration.A novel bispecific molecule delivered by recombinant AAV2 suppresses ocular inflammation and choroidal neovascularization Higher plasma levels of complement C3a, C4a and C5a increase the risk of subretinal fibrosis in neovascular age-related macular degeneration: Complement activation in AMD.Celastrol protects mouse retinas from bright light-induced degeneration through inhibition of oxidative stress and inflammation.Targeting the complement system for the management of retinal inflammatory and degenerative diseases.Flow cytometric analysis of inflammatory and resident myeloid populations in mouse ocular inflammatory models Regulation of Stem Cell Properties of Müller Glia by JAK/STAT and MAPK Signaling in the Mammalian Retina.Peripheral blood mononuclear cells from neovascular age-related macular degeneration patients produce higher levels of chemokines CCL2 (MCP-1) and CXCL8 (IL-8).The impact of environmental enrichment on the murine inflammatory immune response.Age-Related Macular Degeneration in Patients With Chronic Myeloproliferative Neoplasms.Monomeric C-reactive protein and inflammation in age-related macular degeneration.Depletion of the Third Complement Component Ameliorates Age-Dependent Oxidative Stress and Positively Modulates Autophagic Activity in Aged Retinas in a Mouse Model Conditional, Genetically Encoded, Small Molecule-Regulated Inhibition of NFκB Signaling in RPE Cells.Correlation of neutrophil/lymphocyte and platelet/lymphocyte ratio with visual acuity and macular thickness in age-related macular degeneration.Age-Related Macular Degeneration: A Connection between Human Herpes Virus-6A-Induced CD46 Downregulation and Complement Activation?CB2 receptor activation causes an ERK1/2-dependent inflammatory response in human RPE cells.T-cell differentiation and CD56+ levels in polypoidal choroidal vasculopathy and neovascular age-related macular degeneration.Blockade of microglial adenosine A2A receptor impacts inflammatory mechanisms, reduces ARPE-19 cell dysfunction and prevents photoreceptor loss in vitro.Intraocular inflammatory cytokines in patients with neovascular age-related macular degeneration before and after initiation of intravitreal injection of anti-VEGF inhibitor.Microglia in the primate macula: specializations in microglial distribution and morphology with retinal position and with aging.The NLRP3 Inflammasome May Contribute to Pathologic Neovascularization in the Advanced Stages of Diabetic Retinopathy.A model to study complement involvement in experimental retinal degeneration.Age-related pro-inflammatory and pro-angiogenic changes in human aqueous humor.Oxidative damage induces MCP-1 secretion and macrophage aggregation in age-related macular degeneration (AMD).Systemic inflammation induced by lipopolysaccharide aggravates inherited retinal dystrophy.The Continuum of Aging and Age-Related Diseases: Common Mechanisms but Different Rates.Microglia Activation and Immunomodulatory Therapies for Retinal Degenerations.Modulation of three key innate immune pathways for the most common retinal degenerative diseases New insight into the role of the complement in the most common types of retinopathy-current literature review

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P2860

Parainflammation, chronic inflammation, and age-related macular degeneration

http://www.wikidata.org/entity/Q26796268

description

2015 nî lūn-bûn

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2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2015 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Parainflammation, chronic inflammation, and age-related macular degeneration

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Parainflammation, chronic inflammation, and age-related macular degeneration

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Parainflammation, chronic inflammation, and age-related macular degeneration

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Parainflammation, chronic inflammation, and age-related macular degeneration

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Parainflammation, chronic inflammation, and age-related macular degeneration

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Journal of Leukocyte Biology

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Parainflammation, chronic inflammation, and age-related macular degeneration

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Mei Chen

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P2860

An animal model of age-related macular degeneration in senescent Ccl-2- or Ccr-2-deficient mice

Induction of immunological tolerance by apoptotic cells requires caspase-dependent oxidation of high-mobility group box-1 protein

Autophagy in immunity and inflammation

DICER1 loss and Alu RNA induce age-related macular degeneration via the NLRP3 inflammasome and MyD88

Ranibizumab and bevacizumab for treatment of neovascular age-related macular degeneration: two-year results

An immunologically privileged retinal antigen elicits tolerance: major role for central selection mechanisms

Genetics of immunological and inflammatory components in age-related macular degeneration

An integrated hypothesis that considers drusen as biomarkers of immune-mediated processes at the RPE-Bruch's membrane interface in aging and age-related macular degeneration

The danger model: a renewed sense of self

High mobility group box 1 protein interacts with multiple Toll-like receptors

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