Mechanisms of neovascularization and resistance to anti-angiogenic therapies in glioblastoma multiforme - Test2 - elhamkhani - TriplyDB

Mechanisms of neovascularization and resistance to anti-angiogenic therapies in glioblastoma multiforme

Mechanisms of neovascularization and resistance to anti-angiogenic therapies in glioblastoma multiforme

http://www.wikidata.org/entity/Q26824557

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Evading anti-angiogenic therapy: resistance to anti-angiogenic therapy in solid tumors Circulating glioma biomarkers Revisiting tumor angiogenesis: vessel co-option, vessel remodeling, and cancer cell-derived vasculature formation Adaptation to antiangiogenic therapy in neurological tumors Induction of vascular endothelial growth factor receptor expression in human umbilical vein endothelial cells after repeated bevacizumab treatment in vitro.In vitro modeling of the neurovascular environment by coculturing adult human brain endothelial cells with human neural stem cells Systemic miRNA-7 delivery inhibits tumor angiogenesis and growth in murine xenograft glioblastoma.Therapeutic efficacy of aldoxorubicin in an intracranial xenograft mouse model of human glioblastoma.Failure of anti tumor-derived endothelial cell immunotherapy depends on augmentation of tumor hypoxia.Bevacizumab treatment induces metabolic adaptation toward anaerobic metabolism in glioblastomas Phase 1 trial of tivantinib in combination with sorafenib in adult patients with advanced solid tumors Immunotherapy for malignant glioma All-trans retinoic acid impairs the vasculogenic mimicry formation ability of U87 stem-like cells through promoting differentiation.Glioblastoma vasculogenic mimicry: signaling pathways progression and potential anti-angiogenesis targets.Wnt3a Promotes the Vasculogenic Mimicry Formation of Colon Cancer via Wnt/β-Catenin Signaling VB-111: a novel anti-vascular therapeutic for glioblastoma multiforme Talin1 targeting potentiates anti-angiogenic therapy by attenuating invasion and stem-like features of glioblastoma multiforme.Improved Treatment Efficacy of Antiangiogenic Therapy when Combined with Picornavirus Vaccination in the GL261 Glioma Model.Acquisition of tumorigenic potential and enhancement of angiogenesis in pulmonary stem/progenitor cells through Oct-4 hyperexpression.Human neural stem cell-induced endothelial morphogenesis requires autocrine/paracrine and juxtacrine signaling.Mechanistic patient-specific predictive correlation of tumor drug response with microenvironment and perfusion measurements Emerging biomarkers in glioblastoma Antiangiogenic cancer treatment: The great discovery and greater complexity (Review).Periostin (POSTN) Regulates Tumor Resistance to Antiangiogenic Therapy in Glioma Models.Intravenous Formulation of HET0016 Decreased Human Glioblastoma Growth and Implicated Survival Benefit in Rat Xenograft Models Resistance to antiangiogenic therapy.Recent advances in vascular disrupting agents in cancer therapy.Apatinib for the treatment of gastric cancer.Epithelioid glioblastoma presenting as massive intracerebral hemorrhage: Case report and review of the literature.Recent advance in molecular angiogenesis in glioblastoma: the challenge and hope for anti-angiogenic therapy.Tumor cell vascular mimicry: Novel targeting opportunity in melanoma.Evaluation of neovascularization patterns in an orthotopic rat glioma model with dynamic contrast-enhanced MRI.Prognostic value of preoperative von Willebrand factor plasma levels in patients with Glioblastoma Glucose-6-phosphatase is a key metabolic regulator of glioblastoma invasion.Vascular Mimicry: The Next Big Glioblastoma Target.Ephrin-B2/Fc promotes proliferation and migration, and suppresses apoptosis in human umbilical vein endothelial cells.High-resolution blood-pool-contrast-enhanced MR angiography in glioblastoma: tumor-associated neovascularization as a biomarker for patient survival. A preliminary study.VEGFR2 regulates endothelial differentiation of colon cancer cells.Deficiency in matrix metalloproteinase-2 results in long-term vascular instability and regression in the injured mouse spinal cord.Autophagy-induced KDR/VEGFR-2 activation promotes the formation of vasculogenic mimicry by glioma stem cells.

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Mechanisms of neovascularization and resistance to anti-angiogenic therapies in glioblastoma multiforme

http://www.wikidata.org/entity/Q26824557

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Mechanisms of neovascularizati ...... ies in glioblastoma multiforme

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Amy Rommel

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Chad Myskiw

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Inder M. Verma

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Yasushi Soda

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VEGF inhibits tumor cell invasion and mesenchymal transition through a MET/VEGFR2 complex

Activation of vascular endothelial growth factor gene transcription by hypoxia-inducible factor 1

Modes of resistance to anti-angiogenic therapy

Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis

Gelatinase-A (MMP-2), gelatinase-B (MMP-9) and membrane type matrix metalloproteinase-1 (MT1-MMP) are involved in different aspects of the pathophysiology of malignant gliomas

Induction of cell migration by matrix metalloprotease-2 cleavage of laminin-5

Bone marrow-derived circulating endothelial precursors do not contribute to vascular endothelium and are not needed for tumor growth

A perivascular niche for brain tumor stem cells

Angiogenesis in life, disease and medicine

Dll4 signalling through Notch1 regulates formation of tip cells during angiogenesis

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neovascularization

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