Lighting the fires within: the cell biology of autoinflammatory diseases - Test2 - elhamkhani - TriplyDB

Lighting the fires within: the cell biology of autoinflammatory diseases

Lighting the fires within: the cell biology of autoinflammatory diseases

http://www.wikidata.org/entity/Q26827163

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Familial Mediterranean fever: current perspectives Localization and functionality of the inflammasome in neutrophils Munchausen by proxy syndrome mimicking systemic autoinflammatory disease: case report and review of the literature.Identification of multifaceted binding modes for pyrin and ASC pyrin domains gives insights into pyrin inflammasome assembly.Genome-wide transcriptional analysis of T cell activation reveals differential gene expression associated with psoriasis.Pathogenesis of adult-onset Still's disease: new insights from the juvenile counterpart.NLRP7 inter-domain interactions: the NACHT-associated domain is the physical mediator for oligomeric assembly Analysis of oxidative stress in zebrafish embryos.When less is more: primary immunodeficiency with an autoinflammatory kick An inherited mutation in NLRC4 causes autoinflammation in human and mice.The structural basis for receptor recognition of human interleukin-18 Inhibition of inflammatory arthritis using fullerene nanomaterials.Poly(ADP-ribose) polymerase-13 and RNA regulation in immunity and cancer.Understanding autoimmunity in the eye: from animal models to novel therapies.Serum uric acid levels in patients with myasthenia gravis are inversely correlated with disability.Ribotoxic stress through p38 mitogen-activated protein kinase activates in vitro the human pyrin inflammasome.microRNA-mediated regulation of innate immune response in rheumatic diseases Chronic nonbacterial osteomyelitis in a child with previous juvenile dermatomyositis "Model t" cells: a time-tested vehicle for gene therapy Immunogenicity of self tumor associated proteins is enhanced through protein truncation.Chronic proliferative dermatitis in Sharpin null mice: development of an autoinflammatory disease in the absence of B and T lymphocytes and IL4/IL13 signaling.A Potential Link between Environmental Triggers and Autoimmunity.Familial Mediterranean Fever: Recent Developments in Pathogenesis and New Recommendations for Management Neutrophil extracellular chromatin traps connect innate immune response to autoimmunity.How Cytokine networks fuel inflammation: Interleukin-17 and a tale of two autoimmune diseases.Key facts and hot spots on tumor necrosis factor receptor-associated periodic syndrome.Proteomics focusing on immune markers in psoriatic arthritis.Competition between virus-derived and endogenous small RNAs regulates gene expression in Caenorhabditis elegans.Protein misfolding and dysregulated protein homeostasis in autoinflammatory diseases and beyond.Autoinflammatory Syndromes in Children.Causes and consequences of endoplasmic reticulum stress in rheumatic disease.Monogenic Periodic Fever Syndromes: Treatment Options for the Pediatric Patient.Autoinflammatory Diseases with Periodic Fevers.The multiple pathways to autoimmunity.Effects of HLA-DRB1 alleles on susceptibility and clinical manifestations in Japanese patients with adult onset Still's disease.Chronic Autoimmune Meningoencephalitis and Periodic Fever Syndrome Treated with Anakinra.Clinical update on inflammasomopathies.Diagnostic validity of colchicine in patients with Familial Mediterranean fever.Enhanced TLR-MYD88 signaling stimulates autoinflammation in SH3BP2 cherubism mice and defines the etiology of cherubism.Dysfunctional immunoproteasomes in autoinflammatory diseases.

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P2860

Lighting the fires within: the cell biology of autoinflammatory diseases

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2012 nî lūn-bûn

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Nature Reviews Immunology

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Lighting the fires within: the cell biology of autoinflammatory diseases

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Ariel Bulua Bourla

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Daniel L. Kastner

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Heiyoung Park

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Richard M. Siegel

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Robert A. Colbert

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A proposed classification of the immunological diseases

Coupling of stress in the ER to activation of JNK protein kinases by transmembrane protein kinase IRE1

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

Immunoproteasomes preserve protein homeostasis upon interferon-induced oxidative stress

Chronic infantile neurological cutaneous and articular syndrome is caused by mutations in CIAS1, a gene highly expressed in polymorphonuclear cells and chondrocytes

Endoplasmic reticulum stress activates cleavage of CREBH to induce a systemic inflammatory response

Pannexin-1 mediates large pore formation and interleukin-1beta release by the ATP-gated P2X7 receptor

SOD1 mutations disrupt redox-sensitive Rac regulation of NADPH oxidase in a familial ALS model

A mutation in the immunoproteasome subunit PSMB8 causes autoinflammation and lipodystrophy in humans

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

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