DISC1: a key lead in studying cortical development and associated brain disorders - Test2 - elhamkhani - TriplyDB

DISC1: a key lead in studying cortical development and associated brain disorders

DISC1: a key lead in studying cortical development and associated brain disorders

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Molecular underpinnings of prefrontal cortex development in rodents provide insights into the etiology of neurodevelopmental disorders Neuroanatomical and behavioral deficits in mice haploinsufficient for Pericentriolar material 1 (Pcm1).DISC1 knockdown impairs the tangential migration of cortical interneurons by affecting the actin cytoskeleton Neuropeptide precursor VGF is genetically associated with social anhedonia and underrepresented in the brain of major mental illness: its downregulation by DISC1.Drug Abuse and Psychosis: New Insights into Drug-induced Psychosis Perineuronal nets and schizophrenia: the importance of neuronal coatings.Half-life of DISC1 protein and its pathological significance under hypoxia stress Disordered ripples are a common feature of genetically distinct mouse models relevant to schizophrenia.Interneuronal DISC1 regulates NRG1-ErbB4 signalling and excitatory-inhibitory synapse formation in the mature cortex.Utility and validity of DISC1 mouse models in biological psychiatry.Aggregation of scaffolding protein DISC1 dysregulates phosphodiesterase 4 in Huntington's disease Role of DISC1 in Neuronal Trafficking and its Implication in Neuropsychiatric Manifestation and Neurotherapeutics.A null mutation in TNIK defines a novel locus for intellectual disability.DISC1 causes associative memory and neurodevelopmental defects in fruit flies.Genetic interaction of DISC1 and Neurexin in the development of fruit fly glutamatergic synapses Caveolin-1 regulation of disrupted-in-schizophrenia-1 as a potential therapeutic target for schizophrenia.Modeling Neurological Diseases With Human Brain Organoids.

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DISC1: a key lead in studying cortical development and associated brain disorders

http://www.wikidata.org/entity/Q26851318

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2013年の論文

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2013年論文

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DISC1: a key lead in studying cortical development and associated brain disorders

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DISC1: a key lead in studying cortical development and associated brain disorders

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DISC1: a key lead in studying cortical development and associated brain disorders

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DISC1: a key lead in studying cortical development and associated brain disorders

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DISC1: a key lead in studying cortical development and associated brain disorders

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The Neuroscientist

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A. Sawa

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K. Nakajima

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S. Narayan

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Disordered migration and loss of virus-infected neuronal cells in developing mouse brains infected with murine cytomegalovirus

A role for the lissencephaly gene LIS1 in mitosis and cytoplasmic dynein function

DISC1 (Disrupted-In-Schizophrenia 1) is a centrosome-associated protein that interacts with MAP1A, MIPT3, ATF4/5 and NUDEL: regulation and loss of interaction with mutation

DISC1-NDEL1/NUDEL protein interaction, an essential component for neurite outgrowth, is modulated by genetic variations of DISC1

Disrupted in Schizophrenia 1 Interactome: evidence for the close connectivity of risk genes and a potential synaptic basis for schizophrenia

Recruitment of PCM1 to the centrosome by the cooperative action of DISC1 and BBS4: a candidate for psychiatric illnesses

Association of GSK3beta polymorphisms with brain structural changes in major depressive disorder

Disrupted in schizophrenia 1 regulates neuronal progenitor proliferation via modulation of GSK3beta/beta-catenin signaling

Point mutations and an intragenic deletion in LIS1, the lissencephaly causative gene in isolated lissencephaly sequence and Miller-Dieker syndrome

The Bardet-Biedl protein BBS4 targets cargo to the pericentriolar region and is required for microtubule anchoring and cell cycle progression

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