Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease - Test2 - elhamkhani - TriplyDB

Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Morphological changes of glutamatergic synapses in animal models of Parkinson's disease Aldehyde Dehydrogenase 1 making molecular inroads into the differential vulnerability of nigrostriatal dopaminergic neuron subtypes in Parkinson's disease The benefits of humanized yeast models to study Parkinson's disease Upstream deregulation of calcium signaling in Parkinson's disease DNA variants in CACNA1C modify Parkinson disease risk only when vitamin D level is deficient.Physiological characterisation of human iPS-derived dopaminergic neurons Converging roles of ion channels, calcium, metabolic stress, and activity pattern of Substantia nigra dopaminergic neurons in health and Parkinson's disease GDNF-Ret signaling in midbrain dopaminergic neurons and its implication for Parkinson disease Quantification of mitochondrial morphology in neurites of dopaminergic neurons using multiple parameters Aldehyde dehydrogenase 1 defines and protects a nigrostriatal dopaminergic neuron subpopulation Ionic currents influencing spontaneous firing and pacemaker frequency in dopamine neurons of the ventrolateral periaqueductal gray and dorsal raphe nucleus (vlPAG/DRN): A voltage-clamp and computational modelling study.VPS35 regulates parkin substrate AIMP2 toxicity by facilitating lysosomal clearance of AIMP2.Defining a role for hemoglobin in Parkinson's disease Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunction.Nicotinamide nucleotide transhydrogenase (Nnt) links the substrate requirement in brain mitochondria for hydrogen peroxide removal to the thioredoxin/peroxiredoxin (Trx/Prx) system.Chronic behavioral and cognitive deficits in a rat survival model of paraoxon toxicity Differential neuroprotective and anti-inflammatory effects of L-type voltage dependent calcium channel and ryanodine receptor antagonists in the substantia nigra and locus coeruleus Parkin cooperates with GDNF/RET signaling to prevent dopaminergic neuron degeneration Involvement of Mu Opioid Receptor Signaling in the Protective Effect of Opioid against 6-Hydroxydopamine-Induced SH-SY5Y Human Neuroblastoma Cells Apoptosis.Understanding the susceptibility of dopamine neurons to mitochondrial stressors in Parkinson's disease.Similar molecular determinants on Rem mediate two distinct modes of inhibition of CaV1.2 channels.Circadian dysfunction in response to in vivo treatment with the mitochondrial toxin 3-nitropropionic acid.Polyphenols: multipotent therapeutic agents in neurodegenerative diseases.Neuronal calcium signaling: function and dysfunction.Calcium signaling in Parkinson's disease.Interactions between calcium and alpha-synuclein in neurodegeneration.Vitamin D: a custodian of cell signalling stability in health and disease.Alterations in late endocytic trafficking related to the pathobiology of LRRK2-linked Parkinson's disease.Clinical and scientific perspectives on movement disorders: Stanley Fahn's contributions.PKA Phosphorylation of NCLX Reverses Mitochondrial Calcium Overload and Depolarization, Promoting Survival of PINK1-Deficient Dopaminergic Neurons Parkinson's Disease and Neurodegeneration: GABA-Collapse Hypothesis Calcium and Parkinson's disease.Cav1.2 and Cav1.3 L-type calcium channels operate in a similar voltage range but show different coupling to Ca(2+)-dependent conductances in hippocampal neurons.The role of Ca2+ signaling in Parkinson's disease.The Hyperpolarization-Activated Current Determines Synaptic Excitability, Calcium Activity and Specific Viability of Substantia Nigra Dopaminergic Neurons.Live calcium and mitochondrial imaging in the enteric nervous system of Parkinson patients and controls.Human umbilical cord mesenchymal stem cells infected with adenovirus expressing HGF promote regeneration of damaged neuron cells in a Parkinson's disease model.Calcium currents regulate dopamine autoreceptors.Cav1.3 channels control D2-autoreceptor responses via NCS-1 in substantia nigra dopamine neurons PINK1-mediated phosphorylation of LETM1 regulates mitochondrial calcium transport and protects neurons against mitochondrial stress.

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P2860

Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

http://www.wikidata.org/entity/Q26853225

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2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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Calcium, bioenergetics, and neuronal vulnerability in Parkinson's disease

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D. J. Surmeier

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How mitochondria produce reactive oxygen species

Lysosomal Ca(2+) homeostasis: role in pathogenesis of lysosomal storage diseases

Ionic mechanisms underlying autonomous action potential generation in the somata and dendrites of GABAergic substantia nigra pars reticulata neurons in vitro

'Rejuvenation' protects neurons in mouse models of Parkinson's disease

Oxidant stress evoked by pacemaking in dopaminergic neurons is attenuated by DJ-1.

Mutations in SDHD, a mitochondrial complex II gene, in hereditary paraganglioma

Mitochondrial complex III is required for hypoxia-induced ROS production and cellular oxygen sensing

Stages in the development of Parkinson's disease-related pathology

Neuroinflammation in Parkinson's disease: a target for neuroprotection?

The cell-non-autonomous nature of electron transport chain-mediated longevity

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23086948

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Parkinson's disease

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