STAT6-dependent and -independent mechanisms in Th2 polarization
about
Inhibition of the Mechanistic Target of Rapamycin (mTOR)-Rapamycin and BeyondPARP9 and PARP14 cross-regulate macrophage activation via STAT1 ADP-ribosylationEosinophils and type 2 cytokine signaling in macrophages orchestrate development of functional beige fat.Key mediators in the immunopathogenesis of allergic asthmaPARP is activated in human asthma and its inhibition by olaparib blocks house dust mite-induced disease in mice.JAK3/STAT6 Stimulates Bone Marrow-Derived Fibroblast Activation in Renal Fibrosis.The lncRNAs involved in mouse airway allergic inflammation following induced pluripotent stem cell-mesenchymal stem cell treatment.STAT6 deficiency ameliorates Graves' disease severity by suppressing thyroid epithelial cell hyperplasiaSTAT6 and lung inflammation.Protective role of NKT cells and macrophage M2-driven phenotype in bleomycin-induced pulmonary fibrosis.Interleukin-17 inhibits development of malignant pleural effusion via interleukin-9-dependent mechanism.Anemoside A3 ameliorates experimental autoimmune encephalomyelitis by modulating T helper 17 cell response.Enforced Expression of Hoxa3 Inhibits Classical and Promotes Alternative Activation of Macrophages In Vitro and In Vivo.Aspirin induces IL-4 production: augmented IL-4 production in aspirin-exacerbated respiratory diseaseSodium Benzoate, a Food Additive and a Metabolite of Cinnamon, Enriches Regulatory T Cells via STAT6-Mediated Upregulation of TGF-βHuman ADMC-Derived Adipocyte Thermogenic Capacity Is Regulated by IL-4 Receptor.Unbiased Quantitative Proteomics Reveals a Crucial Role of the Allergen Context for the Activation of Human Dendritic Cells.Actin dynamics shape microglia effector functions.The role of macrophage phenotype in regulating the response to radiation therapy.Histone modifications of Notch1 promoter affect lung CD4+ T cell differentiation in asthmatic rats.Bcl11b is essential for licensing Th2 differentiation during helminth infection and allergic asthma.TH2 cell development and function.Invariant Natural Killer T Cell Subsets—More Than Just Developmental Intermediates.Parkinson's disease patients have a complex phenotypic and functional Th1 bias: cross-sectional studies of CD4+ Th1/Th2/T17 and Treg in drug-naïve and drug-treated patients.
P2860
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P2860
STAT6-dependent and -independent mechanisms in Th2 polarization
description
2012 nî lūn-bûn
@nan
2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
STAT6-dependent and -independent mechanisms in Th2 polarization
@ast
STAT6-dependent and -independent mechanisms in Th2 polarization
@en
STAT6-dependent and -independent mechanisms in Th2 polarization
@nl
type
label
STAT6-dependent and -independent mechanisms in Th2 polarization
@ast
STAT6-dependent and -independent mechanisms in Th2 polarization
@en
STAT6-dependent and -independent mechanisms in Th2 polarization
@nl
prefLabel
STAT6-dependent and -independent mechanisms in Th2 polarization
@ast
STAT6-dependent and -independent mechanisms in Th2 polarization
@en
STAT6-dependent and -independent mechanisms in Th2 polarization
@nl
P2860
P921
P3181
P356
P1476
STAT6-dependent and -independent mechanisms in Th2 polarization
@en
P2093
Elisabeth Maier
P2860
P304
P3181
P356
10.1002/EJI.201242433
P407
P577
2012-10-08T00:00:00Z