Physiological consequences of complex II inhibition for aging, disease, and the mKATP channel
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ATP Sensitive Potassium Channels in the Skeletal Muscle Function: Involvement of the KCNJ11(Kir6.2) Gene in the Determination of Mechanical Warner Bratzer Shear ForceCardiovascular KATP channels and advanced agingMitochondrial mechanisms in cerebral vascular control: shared signaling pathways with preconditioning.Ischaemic accumulation of succinate controls reperfusion injury through mitochondrial ROS.High fat, high sucrose diet causes cardiac mitochondrial dysfunction due in part to oxidative post-translational modification of mitochondrial complex II.High molecular weight forms of mammalian respiratory chain complex IIMitochondrial Reactive Oxygen Species Mediate Cardiac Structural, Functional, and Mitochondrial Consequences of Diet-Induced Metabolic Heart DiseaseKir6.2 is not the mitochondrial KATP channel but is required for cardioprotection by ischemic preconditioningChromophore-Assisted Light Inactivation of Mitochondrial Electron Transport Chain Complex II in Caenorhabditis elegans.The environmental carcinogen benzo[a]pyrene induces a Warburg-like metabolic reprogramming dependent on NHE1 and associated with cell survival.Potentially diagnostic electron paramagnetic resonance spectra elucidate the underlying mechanism of mitochondrial dysfunction in the deoxyguanosine kinase deficient rat model of a genetic mitochondrial DNA depletion syndromeHigh fructose causes cardiac hypertrophy via mitochondrial signaling pathway.Oxidative stress--a key emerging impact factor in health, ageing, lifestyle and aesthetics.Moving Forwards by Blocking Back-Flow: The Yin and Yang of MI Therapy.The Slo(w) path to identifying the mitochondrial channels responsible for ischemic protection.Alteration of ROS homeostasis and decreased lifespan in S. cerevisiae elicited by deletion of the mitochondrial translocator FLX1.Molecular Bases of Brain PreconditioningMcl-1 regulates reactive oxygen species via NOX4 during chemotherapy-induced senescence.The reduced myofilament responsiveness to calcium contributes to the negative force-frequency relationship in rat cardiomyocytes: role of reactive oxygen species and p-38 map kinase.Role of Mitochondria in Cerebral Vascular Function: Energy Production, Cellular Protection, and Regulation of Vascular Tone.Use the Protonmotive Force: Mitochondrial Uncoupling and Reactive Oxygen Species.Overexpression of Nmnat3 efficiently increases NAD and NGD levels and ameliorates age-associated insulin resistance
P2860
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P2860
Physiological consequences of complex II inhibition for aging, disease, and the mKATP channel
description
2013 nî lūn-bûn
@nan
2013 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Physiological consequences of ...... disease, and the mKATP channel
@ast
Physiological consequences of ...... disease, and the mKATP channel
@en
Physiological consequences of ...... disease, and the mKATP channel
@nl
type
label
Physiological consequences of ...... disease, and the mKATP channel
@ast
Physiological consequences of ...... disease, and the mKATP channel
@en
Physiological consequences of ...... disease, and the mKATP channel
@nl
prefLabel
Physiological consequences of ...... disease, and the mKATP channel
@ast
Physiological consequences of ...... disease, and the mKATP channel
@en
Physiological consequences of ...... disease, and the mKATP channel
@nl
P2093
P2860
P3181
P1476
Physiological consequences of ...... disease, and the mKATP channel
@en
P2093
Andrew P. Wojtovich
C. Owen Smith
Cole M. Haynes
Keith W. Nehrke
Paul S. Brookes
P2860
P304
P3181
P356
10.1016/J.BBABIO.2012.12.007
P407
P577
2013-05-01T00:00:00Z