From the neurobiology of extinction to improved clinical treatments.
about
A systematic review of the neural bases of psychotherapy for anxiety and related disordersEyeblink classical conditioning and post-traumatic stress disorder - a model systems approach.Emerging drugs for the treatment of anxietyThe role of stress and fear in the development of mental disorders.Less fear, more diversityXenon impairs reconsolidation of fear memories in a rat model of post-traumatic stress disorder (PTSD)Translational Approaches Targeting Reconsolidation.Activity-dependent signaling: influence on plasticity in circuits controlling fear-related behaviorIncreasing brain angiotensin converting enzyme 2 activity decreases anxiety-like behavior in male mice by activating central Mas receptors.Serotonin regulates brain-derived neurotrophic factor expression in select brain regions during acute psychological stress.At the crossroads: the intersection of substance use disorders, anxiety disorders, and posttraumatic stress disorder.Threat processing: models and mechanisms.Examining the relationship between blast-induced mild traumatic brain injury and posttraumatic stress-related traits.Genetic approaches for the study of PTSD: Advances and challenges.Novel pharmacological treatment strategies for posttraumatic stress disorder.Extinction after fear memory reactivation fails to eliminate renewal in rats.Trauma Cognitions Are Related to Symptoms up to 10 Years After Cognitive Behavioral Treatment for Posttraumatic Stress Disorder.Reconsolidation and extinction: Using epigenetic signatures to challenge conventional wisdom.Angiotensin type 1a receptors in the paraventricular nucleus of the hypothalamus control cardiovascular reactivity and anxiety-like behavior in male mice.Reversible Inactivation of the Bed Nucleus of the Stria Terminalis Prevents Reinstatement But Not Renewal of Extinguished Fear(1,2,3)Environmental enrichment facilitates cocaine-cue extinction, deters reacquisition of cocaine self-administration and alters AMPAR GluA1 expression and phosphorylation.The Role of Serine Racemase in the Pathophysiology of Brain Disorders.Dynamic expression of FKBP5 in the medial prefrontal cortex regulates resiliency to conditioned fear.Fructus and Its Active Ingredients as Promising Resources for the Treatment of Neurological DiseasesPosttraumatic Stress Disorder: An Integrated Overview of the Neurobiological Rationale for Pharmacology
P2860
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P2860
From the neurobiology of extinction to improved clinical treatments.
description
2013 nî lūn-bûn
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2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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name
From the neurobiology of extinction to improved clinical treatments
@nl
From the neurobiology of extinction to improved clinical treatments.
@ast
From the neurobiology of extinction to improved clinical treatments.
@en
type
label
From the neurobiology of extinction to improved clinical treatments
@nl
From the neurobiology of extinction to improved clinical treatments.
@ast
From the neurobiology of extinction to improved clinical treatments.
@en
prefLabel
From the neurobiology of extinction to improved clinical treatments
@nl
From the neurobiology of extinction to improved clinical treatments.
@ast
From the neurobiology of extinction to improved clinical treatments.
@en
P2860
P3181
P356
P1476
From the neurobiology of extinction to improved clinical treatments.
@en
P2093
Filomene G Morrison
P2860
P304
P3181
P356
10.1002/DA.22214
P407
P577
2013-11-20T00:00:00Z