Cell atrophy and loss in depression: reversal by antidepressant treatment - Test2 - elhamkhani - TriplyDB

Cell atrophy and loss in depression: reversal by antidepressant treatment

Cell atrophy and loss in depression: reversal by antidepressant treatment

http://www.wikidata.org/entity/Q27013609

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Neuroplasticity and the next wave of antidepressant strategies Comorbidity Factors and Brain Mechanisms Linking Chronic Stress and Systemic Illness Dendritic Spines in Depression: What We Learned from Animal Models Glial abnormalities in mood disorders Sustained remission from depressive-like behavior depends on hippocampal neurogenesis.Brain morphological changes in adolescent and adult patients with anorexia nervosa.GABAergic neurons in nucleus accumbens are correlated to resilience and vulnerability to chronic stress for major depression.Gene expression analysis of novel genes in the prefrontal cortex of major depressive disorder subjects.The impacts of swimming exercise on hippocampal expression of neurotrophic factors in rats exposed to chronic unpredictable mild stress Pathophysiology of depression and innovative treatments: remodeling glutamatergic synaptic connections.Lurasidone exerts antidepressant properties in the chronic mild stress model through the regulation of synaptic and neuroplastic mechanisms in the rat prefrontal cortex.Molecular Mechanism for Stress-Induced Depression Assessed by Sequencing miRNA and mRNA in Medial Prefrontal Cortex.Glucocorticoid Induces Incoordination between Glutamatergic and GABAergic Neurons in the Amygdala.Antidepressant-like effects of curcumin in WKY rat model of depression is associated with an increase in hippocampal BDNF Rapid-acting glutamatergic antidepressants: the path to ketamine and beyond.Incoordination among Subcellular Compartments Is Associated with Depression-Like Behavior Induced by Chronic Mild Stress Glial cell morphological and density changes through the lifespan of rhesus macaques Inverse changes in L1 retrotransposons between blood and brain in major depressive disorder.Depression and treatment response: dynamic interplay of signaling pathways and altered neural processes.Impaired GABA synthesis, uptake and release are associated with depression-like behaviors induced by chronic mild stress.Neuroimmunomodulation in unipolar depression: a focus on chronobiology and chronotherapeutics.Neurobiology of stress, depression, and rapid acting antidepressants: remodeling synaptic connections.Hypothalamic-pituitary-adrenocortical axis: neuropsychiatric aspects.The antidepressant-like effect of Ocimum basilicum in an animal model of depression.New advances on glial activation in health and disease.The antidepressant effect of musk in an animal model of depression: a histopathological study.Chronic stress-induced disruption of the astrocyte network is driven by structural atrophy and not loss of astrocytes.Cocaine Self-Administration and Extinction Leads to Reduced Glial Fibrillary Acidic Protein Expression and Morphometric Features of Astrocytes in the Nucleus Accumbens Core Rose oil (from Rosa × damascena Mill.) vapor attenuates depression-induced oxidative toxicity in rat brain.Dysregulation of vitamin D metabolism in the brain and myocardium of rats following prolonged exposure to dexamethasone.Duloxetine Protects Human Neuroblastoma Cells from Oxidative Stress-Induced Cell Death Through Akt/Nrf-2/HO-1 Pathway.Anti-depressant effect of hesperidin in diabetic rats.Depression and Cognitive Function in Mild Cognitive Impairment: A 1-Year Follow-Up Study.Reduced astrocyte density underlying brain volume reduction in activity-based anorexia rats.Evaluation of the antidepressant-like effect of musk in an animal model of depression: how it works.Antidepressant Flavonoids and Their Relationship with Oxidative Stress.Roles of Hippocampal Somatostatin Receptor Subtypes in Stress Response and Emotionality.Hippocampal N-acetylaspartate and morning cortisol levels in drug-naive, first-episode patients with major depressive disorder: effects of treatment.Clinical Factors Associated with Brain Volume Reduction in Systemic Lupus Erythematosus Patients without Major Neuropsychiatric Manifestations.Peripheral Etanercept Administration Normalizes Behavior, Hippocampal Neurogenesis, and Hippocampal Reelin and GABAA Receptor Expression in a Preclinical Model of Depression.

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Cell atrophy and loss in depression: reversal by antidepressant treatment

http://www.wikidata.org/entity/Q27013609

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2011 nî lūn-bûn

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2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Current Opinion in Cell Biology

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Cell atrophy and loss in depression: reversal by antidepressant treatment

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Jason M Dwyer

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Mounira Banasr

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Ronald S Duman

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P2860

A randomized add-on trial of an N-methyl-D-aspartate antagonist in treatment-resistant bipolar depression

mTOR signaling: at the crossroads of plasticity, memory and disease

Positive AMPA receptor modulation rapidly stimulates BDNF release and increases dendritic mRNA translation

The molecular neurobiology of depression

Stress blunts serotonin- and hypocretin-evoked EPSCs in prefrontal cortex: role of corticosterone-mediated apical dendritic atrophy

Genetic variant BDNF (Val66Met) polymorphism alters anxiety-related behavior

Physiology and neurobiology of stress and adaptation: central role of the brain

VEGF is an essential mediator of the neurogenic and behavioral actions of antidepressants

Repeated unpredictable stress and antidepressants differentially regulate expression of the bcl-2 family of apoptotic genes in rat cortical, hippocampal, and limbic brain structures

The epidemiology of major depressive disorder: results from the National Comorbidity Survey Replication (NCS-R)

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