Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies - Test2 - elhamkhani - TriplyDB

Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies

Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies

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Traumatic Axonal Injury: Mechanisms and Translational Opportunities Role of Glia in Memory Deficits Following Traumatic Brain Injury: Biomarkers of Glia Dysfunction Near-infrared photonic energy penetration: can infrared phototherapy effectively reach the human brain?Alkyne-azide "click" chemistry in designing nanocarriers for applications in biology Molecular mechanisms of cognitive dysfunction following traumatic brain injury Imaging mass spectrometry reveals loss of polyunsaturated cardiolipins in the cortical contusion, hippocampus, and thalamus after traumatic brain injury Succinate supplementation improves metabolic performance of mixed glial cell cultures with mitochondrial dysfunction.Treatment of mitochondrial disorders.The neuroprotective steroid progesterone promotes mitochondrial uncoupling, reduces cytosolic calcium and augments stress resistance in yeast cells DTI measures identify mild and moderate TBI cases among patients with complex health problems: A receiver operating characteristic analysis of U.S. veterans.Neuroinflammatory responses to traumatic brain injury: etiology, clinical consequences, and therapeutic opportunities.Methylene blue-induced neuronal protective mechanism against hypoxia-reoxygenation stress Systematic Review of Traumatic Brain Injury and the Impact of Antioxidant Therapy on Clinical Outcomes.Neurotrauma: The Crosstalk between Neurotrophins and Inflammation in the Acutely Injured Brain.Altered Mitochondrial Dynamics and TBI Pathophysiology.Rapamycin ameliorates cadmium-induced activation of MAPK pathway and neuronal apoptosis by preventing mitochondrial ROS inactivation of PP2A.Neuroprotective effect of tea polyphenols on oxyhemoglobin induced subarachnoid hemorrhage in mice.Crosstalk between Ca2+ signaling and mitochondrial H2O2 is required for rotenone inhibition of mTOR signaling pathway leading to neuronal apoptosis.Quercetin induces mitochondrial biogenesis in experimental traumatic brain injury via the PGC-1α signaling pathway.Potassium Aspartate Attenuates Brain Injury Induced by Controlled Cortical Impact in Rats Through Increasing Adenosine Triphosphate (ATP) Levels, Na+/K+-ATPase Activity and Reducing Brain Edema.Neuroinflammation in animal models of traumatic brain injury.Elevated levels of plasma mitochondrial DNA DAMPs are linked to clinical outcome in severely injured human subjects Antioxidant gene therapy against neuronal cell death.MSCs-Derived Exosomes and Neuroinflammation, Neurogenesis and Therapy of Traumatic Brain Injury.Mechanical stretch exacerbates the cell death in SH-SY5Y cells exposed to paraquat: mitochondrial dysfunction and oxidative stress Diet and cognition: interplay between cell metabolism and neuronal plasticity.Diets involved in PPAR and PI3K/AKT/PTEN pathway may contribute to neuroprotection in a traumatic brain injury.Chemical regulation of signaling pathways to programmed necrosis.Crosstalk Between Endoplasmic Reticulum Stress, Oxidative Stress, and Autophagy: Potential Therapeutic Targets for Acute CNS Injuries.Double-Injected Human Stem Cells Enhance Rehabilitation in TBI Mice Via Modulation of Survival and Inflammation.Pathogenesis of brain edema and investigation into anti-edema drugs.Role of Wnt Signaling in Central Nervous System Injury.Fusion or Fission: The Destiny of Mitochondria In Traumatic Brain Injury of Different Severities.Neuroprotective effects of hydrogen sulfide on sodium azide‑induced autophagic cell death in PC12 cells.Degradomics in Neurotrauma: Profiling Traumatic Brain Injury.The Pathophysiology of Concussion.Pathophysiology Associated with Traumatic Brain Injury: Current Treatments and Potential Novel Therapeutics.Kaempferol attenuates the glutamate-induced oxidative stress in mouse-derived hippocampal neuronal HT22 cells.Cytoprotective effect of eckol against oxidative stress-induced mitochondrial dysfunction: involvement of the FoxO3a/AMPK pathway.The use of antioxidants in the treatment of traumatic brain injury.

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Mitochondria in traumatic brain injury and mitochondrial-targeted multipotential therapeutic strategies

http://www.wikidata.org/entity/Q27024426

description

2012 nî lūn-bûn

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2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Mitochondria in traumatic brai ...... tential therapeutic strategies

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Mitochondria in traumatic brai ...... tential therapeutic strategies

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Mitochondria in traumatic brai ...... tential therapeutic strategies

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type

label

Mitochondria in traumatic brai ...... tential therapeutic strategies

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Mitochondria in traumatic brai ...... tential therapeutic strategies

@en

Mitochondria in traumatic brai ...... tential therapeutic strategies

@nl

prefLabel

Mitochondria in traumatic brai ...... tential therapeutic strategies

@ast

Mitochondria in traumatic brai ...... tential therapeutic strategies

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Mitochondria in traumatic brai ...... tential therapeutic strategies

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J.1476-5381.2012.02025.X

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British Journal of Pharmacology

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Mitochondria in traumatic brai ...... tential therapeutic strategies

@en

P2093

Gang Cheng

http://www.w3.org/2001/XMLSchema#string

Jian-ning Zhang

http://www.w3.org/2001/XMLSchema#string

Lei-ming Zhang

http://www.w3.org/2001/XMLSchema#string

Rong-hua Kong

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P2860

Molecular characterization of mitochondrial apoptosis-inducing factor

Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent manner

Bcl-2 family proteins regulate the release of apoptogenic cytochrome c by the mitochondrial channel VDAC

Identification of the cytolinker plectin as a major early in vivo substrate for caspase 8 during CD95- and tumor necrosis factor receptor-mediated apoptosis

The Fas death signaling pathway connecting reactive oxygen species generation and FLICE inhibitory protein down-regulation

Cytochrome c acts as a cardiolipin oxygenase required for release of proapoptotic factors

An endoplasmic reticulum stress-specific caspase cascade in apoptosis. Cytochrome c-independent activation of caspase-9 by caspase-12

Inhibition of ADP/ATP exchange in receptor-interacting protein-mediated necrosis.

Bid, a Bcl2 interacting protein, mediates cytochrome c release from mitochondria in response to activation of cell surface death receptors

Cleavage of BID by caspase 8 mediates the mitochondrial damage in the Fas pathway of apoptosis

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scholarly article

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10.1111/J.1476-5381.2012.02025.X

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4

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262847987

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23003569

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traumatic brain injury

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