5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing - Test2 - elhamkhani - TriplyDB

5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

http://www.wikidata.org/entity/Q27027416

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Selective serotonin 5-HT1A receptor biased agonists elicitdistinct brain activation patterns: a pharmacoMRI study RGS6 as a Novel Therapeutic Target in CNS Diseases and Cancer.Abrogated Freud-1/Cc2d1a Repression of 5-HT1A Autoreceptors Induces Fluoxetine-Resistant Anxiety/Depression-Like Behavior.Asenapine modulates mood-related behaviors and 5-HT1A/7 receptors-mediated neurotransmission.Positive regulation of raphe serotonin neurons by serotonin 2B receptors.Mechanisms of 5-HT1A receptor-mediated transmission in dorsal raphe serotonin neurons.

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

http://www.wikidata.org/entity/Q27027416

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2015 nî lūn-bûn

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2015 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի հուլիսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

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ACSCHEMNEURO.5B00034

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ACS Chemical Neuroscience

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5-HT1A Receptor-Mediated Autoinhibition and the Control of Serotonergic Cell Firing

@en

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Daniel Huereca

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Elaine M. Andrade

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Joseph G. Lyons

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Kelly M. McGregor

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Rodrigo Andrade

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P2860

Crystal Structure of the Mammalian GIRK2 K+ Channel and Gating Regulation by G Proteins, PIP2, and Sodium

EVIDENCE FOR THE EXISTENCE OF MONOAMINE-CONTAINING NEURONS IN THE CENTRAL NERVOUS SYSTEM. I. DEMONSTRATION OF MONOAMINES IN THE CELL BODIES OF BRAIN STEM NEURONS

WAY-100635, a potent and selective 5-hydroxytryptamine1A antagonist, increases serotonergic neuronal activity in behaving cats: comparison with (S)-WAY-100135

Electrophysiological, biochemical, neurohormonal and behavioural studies with WAY-100635, a potent, selective and silent 5-HT1A receptor antagonist

Receptor-mediated inhibition of G protein-coupled inwardly rectifying potassium channels involves G(alpha)q family subunits, phospholipase C, and a readily diffusible messenger.

Cellular and subcellular localization of 5-hydroxytryptamine1B receptors in the rat central nervous system: immunocytochemical, autoradiographic and lesion studies.

Quantitative analysis of mammalian GIRK2 channel regulation by G proteins, the signaling lipid PIP2 and Na+ in a reconstituted system.

WAY100135: a novel, selective antagonist at presynaptic and postsynaptic 5-HT1A receptors.

Serotonergic neurons signal reward and punishment on multiple timescales.

Projections and interconnections of genetically defined serotonin neurons in mice.

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